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cats
infectious disease
Question | Answer |
---|---|
cowpox disease in cats | domestic cat: fever cheetah: fatal |
cowpox disease in cats | bank voles bites |
pseudorabies in cats | acquired by eating aborted pig fetus or dead piglet infected with virus. signs are pruritus and death |
feline viral rhinotracheitis (FVR) | rhinitis and pharyngitis (8-14wks). sneezing, nasal discharge, lingual ulcers. dendritic corneal ulcers. latent or active carriers. glucocorticoids induce viral shedding |
FVR vaccine | modified live. possible to become infected after receiving vaccine but severity is less. |
Feline panleucopnia clinical signs | lethargy, anorexia, vomiting of yellow fluid. act thirsty, usually won't drink. high fever! Severe dehydration develops. diarrhea. GI palpably swollen and filled with gas and liquid. Ataxia b/c damage to cerebellum. |
Feline panleucopnia is what type of virus | parvovirus |
Feline panleucopnia prevention | Vaccination, shelter management |
Properties of retroviruses | chronic disease characterized by prolonged nature and immune compromise. Persistent infection. Transmission by direct contact or iatrogenic. Ability to acquire and alter-host derived genetic sequences, integrate into host genome. |
example of retrovirus in cats | FeLV |
FeLV transmission | vertical. horizontal among cats that live together. |
FeLV viremia | shed in saliva, nasal secretions, feces, milk, urine |
FeLV pathogenesis | lymphoid organs. Immune response unable to clear infection |
FeLV asymptomatic phase can turn into what two phases | 1. Progressive infection 2. regressive infection |
FeLV progressive infection | infection not contained. virus replicated in LN and bone marrow. eventually develop FeLV- associated diseases |
FeLV regressive infection | cat remains infected but reverts to as aviremic state. no antigen or culturable virus in circulation |
FeLV clinical disease | lymphoma, blood disorders, immune deficiency, loss appetite, poor coat, persistent fever, pale gums, gingivitis, stomatitis, seizures, eye conditions |
FeLV prevention and control | vaccine is only ok. best way to prevent is to ID and segregate infected cats. |
FIV transmission | bite wounds |
FIV viremia | High concentration in saliva |
FIV pathogenesis | drop in lymphocyte count. inversion of T lymphocyte ratio. immune response unable to clear infection. |
FIV clinical signs | persistent infection. long time for clinical disease to appear. Stomatitis |
FIV prevention | vaccine is only ok. isolate infected cats. may not spread in household with no fighting |
Rabies in cats clinical signs | loss of appetite, anxiety, infection of limbic system. |
FIP wet form clinical signs | effusion into the thorax and abdomen. fibrinous plaques on the serosal surface of abdominal organs. |
FIP general clinical signs | dyspnea, mild pyrexia, muffled heart sounds. uveitiss, keratic precipitations, changes in iris coloration. Fatality high |
FIP non effusive form clinical signs | lesions in eyes and CNS. granulomas in peritoneal cavity. |
FIP transmission | oronasal, replication in enterocytes, highly infectious, persists 3-7wks in env. |
FIP treatment | supportive only. anti-inflammatory immunosuppressive. Vaccine works |
Respiratory type calicivirus transmission | aerosol, oral |
Respiratory type calicivirus clinical signs | dyspena lasting up to a week. palatine ulcers predominate. |
Lymphoreticular type calicivirus transmission | aerosol, oral |
Lymphoreticular type calicivirus clinical signs | kittens 4-10wks. limping, stiffness, soreness, fever |
Calicivirus | respiratory disease progresses to submandibular and limb edema, high temp, alopecia, ulcerative dermatitis. 60% adults die |
calicivirus prevention/control | many cats asymptomatic from multiple cat env. hard to eliminate. |
calicivirus vaccine | do not protect against infection or establishment of a carrier state. does mitagate severity of disease. Modified live, killed subQ, modified live intranasal |