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Pancreas & Diabetes Mellitus

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Question
Answer
combination of 2carbon acetate fragments w/complex organic compound coenzyme A   acetyl coenzyme A  
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glucagon-secreting cells of pancreatic islets   alpha cells  
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metabolic disease characterized by hyperglycemia & caused by insufficient insulin secretion/utilization of insulin   diabetes mellitus  
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disturbance of body's acid-base balance   acidosis  
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acidosis caused by inability to utilize glucose, requiring body's use of fat as energy source   diabetic ketosis  
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fat metabolism generates excessive amounts of acid ketone bodies which disrupts the normal   alkalinity of body fluids  
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long, straight-chain carbon compound containing terminal carboxyl group, which enters into formation of triglyceride   fatty acid  
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during pregnancy elevated hormones result from elevated blood glucose caused by insulin resistance   gestational diabetes  
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postpartum blood glucose returns to normal but women with gestational diabetes have an increased risk of   diabetes later in life  
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storage form of glucose present chiefly in liver & muscle   glycogen  
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hemoglobin with glucose molecules permanently attached   glycosylated hemoglobin  
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glycosylated hemoglobin concentration is related to concentration of   glucose in the blood  
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excessively high blood glucose concentration   hyperglycemia  
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coma resulting from neurologic dysfunction due to severe hypoglycemia causes hyperosmolarity of body fluids   hyperosmolar coma  
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lower than normal blood glucose concentration   hypoglycemia  
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cluster of endocrine cells in pancreas   islets of Langerhans  
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various derivatives of acetyl-CoA resulting from excessive mobilization of fat as an energy source   ketone bodies  
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acetoacetic acid, beta-hydroxybutyric acid, & acetone   acetyl-CoA derivatives in ketone bodies  
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excess of ketone bodies in blood resulting from utilization of fat as primary source of energy   ketosis  
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pancreas excretes digestive enzymes through the   pancreatic duct  
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hemorrhaging, elevated blood levels of pancreatic enzymes, & abdominal pain are all symptoms of   acute pancreatitis  
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diabetes, digestive difficulties, & scarring of the pancreas are all possible outcomes of   recurrent mild acute pancreatitis  
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cystic fibrosis affects   respiratory mucous, digestive enzymes, & sweat  
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hereditary disease where gene mutation causes defective transport across cell membranes of chloride, sodium, & water molecules in which they are dissolved   cystic fibrosis  
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Hyperglycemia results from elevated blood concentrations of   glucose  
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characterized by an inadequate response to insulin as opposed to reduced insulin secretion   maturity-onset diabetes  
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Risk factor predisposing a patient to Cystic Fibrosis and Type 1 & 2 Diabetes is   genetic predisposition  
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glucose that has accumulated in the blood of a diabetic will be excreted through the   urine  
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possible outcomes are diabetic acidosis, coma, & diabetic acidosis for   ketosis  
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urine tests & blood glucose measurements must be done frequently to monitor and control   diabetes  
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in addition to ketoacidosis & diabetic coma diabetes can also lead to   arteriosclerosis & susceptibility to infections  
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insulin-dependent, autoimmune disease, can lead to blindness, ketoacidosis, kidney damage & higher than normal glycosylated hemoglobin test   type 1 diabetes  
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responds to oral drugs to lower blood glucose, diet & exercise controlled, & can lead to blindness, hyperosmolar hyperglycemic nonketotic coma, & damage to kidneys, & higher than normal glycosylated hemoglobin test   type 2 diabetes  
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gallstone & excessive consumption of alcohol can   predispose to pancreatitis  
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