Pharm Y2B3
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each of the black spaces below before clicking
on it to display the answer.
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| thiazide | MOA: early distal tubule - inc flow of Na and H2O downstream segments of DT
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| thiazide | PT: Cl/Na channel - dec in Na reabsorption
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| thiazide | ADR: impotence, hyponatremia, hemolytic anemia
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| thiazide | hydrocholorothiazide
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| thiazide | chlorthalidone
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| thiazide | indapamide
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| loop diuretic | MOA: ascending loop - inhibit Na/K/Cl cotransporter; DT and CD will NOT reabsorb extra Na and Cl
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| loop diuretic | MOA: second site - inhibit Na reabsorb into interstitial fluid
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| loop diuretic | MOA: third site - weak inhibition of CA
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| loop diuretic | ADR: ototoxicity/nephrotoxicity; severe dehydration
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| loop diuretic | furosemide
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| loop diuretic | bumetanide
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| loop diuretic | torsemide
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| loop diuretic | ethacrynic acid
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| K sparring agent | MOA: block Na channel in lumen, therefore less K diffusion in lumen
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| K sparring agent | amiloride
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| K sparring agent | triamterene
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| aldosterone receptor blocker | MOA: CT - inhibit Na/H2O reabsorb. only takes several days
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| aldosterone receptor blocker | ADR: gynecomastia
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| aldosterone receptor blocker | spironolactone
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| CA inhibitor | acetazolamide
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| CA inhibitor | dorzolamide
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| osmotic diuretics | 4 MOA sites: pretty much prevents osmosis, even reduces efficiency of Na/K/Cl cotransporter
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| osmotic diuretics | ADR: worsening of CHF/pulmonary edema, reversible ARF
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| osmotic diuretic | mannitol
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| combo therapy | K-sparring + thiazide
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| K sparring agent | ADR: reduce GLU tolerance, photosensitivity
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