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BBC pharm

chapter 36: Hyperlipidemias

this ocurs in less than one percent of inherited hyuperlipidemia cases na doccurs either by way of lipoprotein lipase deficiency or abnormal lipoprotein lipase, or apoCII defects. type I (familial lipoprotein lipase deficiency)
end result of type I familial lipoprotein lipase deficiency? triglycerides and fatty acids cannot enter adiopse tissue form blood, causing hypertriglyceridemia with elevated chylomicrons and VLDLs.
this represents ten percent of all inerited hyperlipidemias. this is a defect of the LDL receptor, causing a decreased hepatic uptake of LDL. accordingly, LDL levels are elevated. type IIA, familial hypercholesterolemia.
manigests as another form of familial hypercholesterolemia, ad represents forty percent of all inherited hyperlipidemia cases. due to mutant apoprotein B100, resuting in a decreased affinity of LDL for LDL receptor. type IIB
rare, representing less than one precent of all inherited hyperlipidemias. eiology is a mutant apoE, resulting in increased chylomicrons, VLDLs, IDLs and decreased HDLs. type III familial dysbetalipoproteinemia
most common inhertie dhyperlipidemia? type IV (familial hyperlipidemia), due to overproduction of VLDL in association with glucose intolerance and hyperinsulinemia.
anotther type of hypertriglyceridemia and reprsents five percent of inherited hyperlipidemias. exact cause unknown, but hte disorder results in elevated chylomicrons and VLDLs. there is high cholesterol with low LDL and HDL. mildly atherogenic. familial hyperlipoproteineia
overall most common type of hypercholesterolemia. due to combination of genetic and environmental influes and ones ability to metabolize cholesterol, age, and physical activity. polygenic hypercholesterolemia.
Created by: aferdo01