M6 13-005 Word Scramble
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Term | Definition |
Normal Systolic Blood Pressure | <120 mm Hg |
Normal Diastolic Blood Pressure | <80 mm Hg |
Prehypertension | 120-139/80-89 |
High Blood Pressure Systolic | arterial blood pressure 140 mmHg or higher |
High Blood Pressure Diastolic | arterial blood pressure 90 mmHg or greater |
Dx of HTN | 2 or more separate readings on 2 or more separate occasions |
Systolic BP | Greatest force caused by CONTRACTION of the LEFT VENTRICLE |
Diastolic Blood Pressure | Occurs during the RELAXATION phase between hearbeats |
Arterial Blood Pressure | Pressure exerted by blood on the vessel walls |
Vasoconstriciton & Vasodialtion | Controlled by sympathetic nervous system & Renin-angiotensin System |
3 Different Types of HTN | Primary (Essential), Secondary, Malignant |
Primary (Essential) HTN | Cause Unknown, Constitutes 90% to 95% of all cases of HTN |
Non-Modifiable Risk Factors (Essential) | Age, Race, Gender Family Hx |
Modifiable Risk Factors (Essential) | Smoking, Obesity, High Na+ Diet, Elevated Serum Cholesterol, Oral Contraceptive /Estrogen Therapy, Alcohol, Emotional Stress, Sedentary Lifestyle |
Untreated Primary HTN | Fibrous Tissue develop in the arterioles, Decreased Tissue perfusion |
Play an important role in regulating BP | Blood Flow & Peripheral Vascular Resistance |
Renin is released from the | Kidneys |
Angiotensin-Converting Enzyme is released from | Lungs |
Secondary HTN | Identifiable Medical Dx. |
Conditions associated with Secondary HTN | Renal Vascular Disease, Adrenal Cortex Disease, Coarctation of the Aorta, Head Trauma, Cranial Tumor, Poregnancy-induced HTN |
Adrenal Cortex Disease | Primary Aldosteronism, Cushing's Syndrome, Pheochromocytoma |
Malignant HTN | Severe Rapid progressive elevation in BP. Diastolic >120 mmHg |
Malignant HTN Causes | Damage in the small arterioles in major organs, heart, kidneys, brain eyes. |
Renin-Angiotensin System | Hormone system that regulates blood pressure and water (fluid) balance. |
Most distinguishable feature of Malignant HTN | Inflammation to arterioles of eyes (arteriolitis) |
Malignant HTN most common in | Black males under 40 years old |
Most common causes of death w/ Malignant HTN | MI, Heart failure, stroke, renal failure |
Manifestation of HTN | Asymptomatic until VASCULAR CHANGES occur. |
S/S Advanced HTN | Awaken w/ Headache, blurred vision, Spontaneous Epistaxis |
Persistent untreated HTN May result in | Target organ (Heart, Kidney, Brain) damage. |
Assessment of HTN | BP in both arms in SUPINE and SITTING positions. |
HTN Dx Tests (What do they do) | Evaluate baseline of brain, heart & kidneys |
HTN Lab Tests | CBC, Electolytes (Sodium, Potassium, Calcium), Lipid Profile, Fasting Blood Glucose, Creatinine, BUN and Urinalysis |
HTN Dx Tests | Chest Radiograph, ECG and Possible Echo, Intravenous Pyelograph (IVP) |
Why does HTN often go untreated? | Asymptomatic until target organ damage begins |
Goal of HTN Management | Keep BP below 140/90 (older adults), 131/85 (younger adults w/ mild HTN) |
Rx to decrease BP | Anti-hypertensives |
RX to treat uncomplicated HTN | Diuretics, Beat Blockers, Abgiotensin-Converting Enzyme (ACE) Inhibitors, Angiotensin II Receptor Blockers, Calcium Channel Blockers, Alpha-Agonists |
Special Rx Considerations for Diabetes Mellitus | ACE inhibitor helps protect the renal function |
Special Rx Considerations for Heart Failure | ACE Inhibitors, diruetics |
Special Rx Considerations for MI | Beta Blockers, ACE inhibitors |
Special Rx Considerations for African-Americans | Calcium Channel Blockers, Diuretics |
Non-Pharmacological Management | Weight Loss, Reduce Saturated Fats, Limit Alcohol Intake, Exercise, Reduce Sodium Intake, Exercise, Smoking Cessation, Relaxation Techniques |
Anti-hypertensive Agents Use | Tx of HTN |
Anti-hypertensive Agent Actions | Lower BP to a normal level (<90 mmHg Diastolic) or lowest level tolerated |
Anti-hypertensive Therapeutic Goal | Prevention of end-organ damage |
Anti-hypertensive Side Effects | CNS: Headache -- CV: hypotension, bradycardia, tachycardia -- GI: Nausea, Vomiting |
Adrenergic Action | Potent vasodilator, improves myocardial contraction, reduces pulmonary congestion, works on the nervous system |
ACE Inhibitor Action | Blocks the conversion of Angiotension 1 to Angiotension 2 (Prils) |
Angiotensin II Recetptor Antagonist Action | Block the Angiotension II receptors (Sartans) |
Beat Blockers Actions | Lols |
Calcium Channel Blocker Actions | Inhibits the transport of calcium resulting in relaxation of the smooth muscle |
Common side Effects | Hypotension, Bradycardia, Tachycardia, headache, nausea, vomiting, cough or SOB, Angioedema, ED |
Created by:
jtzuetrong
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