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Pulmonology

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Question
Answer
Rapid, deep labored breathing   Kussmaul breathing – DKA, Metabolic acidosis  
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Deep breathing alternating w/ apnea   Cheyne-Stokes breathing – heart failure, brain damage  
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Cavitations, patchy infiltrates on CXR   Infections – lung abscess, TB (Gohn focus)  
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Pleural thickening on CXR   Mesothelioma  
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Hilar mass on CXR   Lung Ca  
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Eggshell pattern on CXR   Silicosis (Sandblasters)  
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Ground glass appearance on CXR   Asbestosis (shipbuilders, building demolition)  
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CXR: reticular-nodular pattern, diffuse rounded opacities; Upper lung zones are primarily affected   Coal Miner’s lung  
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Patchy fibrosis on CXR   Farmers lung  
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Pediatric wheezing   lower respiratory FB, asthma  
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Thumbprint sign   Epiglottitis  
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Steeple sign   FB, viral croup (laryngotracheobronchitis)  
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Inspiratory stridor   FB, viral croup (laryngotracheobronchitis)  
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Premature infant with respiratory distress   Hyaline Mb Disease  
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CXR of premature infant w/ hypoexpansion (ATX), air bronchograms:   Hyaline Mb Disease  
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Smoker, chronic productive cough. NO hemoptysis, weight loss =   Bronchitis (COPD)  
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Smoker, DOE, cough =   COPD  
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Hyperinflation on CXR, tear drop heart   Emphysema  
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Wheezing, prolonged expiration   Asthma  
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Airway edema with eosinophils, neutrophils, lymphocytes   Asthma  
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Pediatric with Hx of recurrent lung infections, pancreatitis, reproductive problems, FTT =   Cystic fibrosis (Staph & Pseudomonal infections usually cause of death)  
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< 2 days post-op with fever =   Atelectasis  
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Stab wound, hyperresonance to percussion, decreased breath sounds, tympany =   Pneumothorax  
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Stab wound, dullness to percussion, decreased breath sounds =   Hemothorax  
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Tall, skinny, male, band student, acute onset one-sided chest pain, dyspnea =   Spontaneous PTX  
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Stab wound to chest. Hypotension, tracheal shift =   Tension PTX  
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Poor sleeping, obese, daytime fatigue & drowsy, snoring, HTN, PM wakening   Obstructive sleep apnea  
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s/p thoracic trauma. Multiple rib fractures. Chest wall moves in with inspiration, out with expiration.   Flail chest (pain control, incentive spirometry, pulmonary toilet, intubation)  
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Irreversible dilation of bronchi resulting from damage of airway wall:   bronchiectasis; usuallu 2/2 repeat infxn; dz of airways (not parenchyma)  
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ARDS on CXR =   bilateral widespread pulmonary infiltrates  
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CXR: infiltrates in mid or lower lung fields, hilar adenopathy, cavitation =   tuberculosis  
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CXR with upper lobe infiltrates, esp apical / posterior segments, cavitation common =   reactivated TB  
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Saddle Nose deformity =   Wegener’s Granulomatosis  
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A change in structure and function of the right ventricle of the heart as a result of a respiratory disorder =   cor pulmonale  
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In Pulmonary HTN, mean pulmonary pressure is __ at rest   >25 mmHg  
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Patients with pulmonary hypertension also have:   low cardiac output  
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The most common secondary cause of pulmonary hypertension =   connective tissue disease (scleroderma)  
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This should be performed in all patients suspected of pulmonary hypertension:   right ventricular catheterization  
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Median survival after diagnosis of pulmonary hypertension =   3 years  
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ARDS on CXR =   bilateral widespread pulmonary infiltrates  
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On CXR: infiltrates in mid or lower lung fields, hilar adenopathy, cavitation =   tuberculosis  
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CXR with upper lobe infiltrates (especially apical & posterior segments); cavitation common =   reactivated TB  
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Viral pneumonia on CXR =   diffuse interstitial infiltrates & hyperinflation  
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Mycoplasma pneumoniae on CXR =   interstitial or bronchopneumonic infiltrates, frequently in the middle or lower lobes  
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Hampton's hump on CXR =   shallow wedge-shaped opacity in lung periphery with its base against pleural surface  
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Hampton's hump on CXR: Dx =   PE  
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Westermark sx on CXR =   oligemia (vasoconstriction) distal to a PE  
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Deer antler on CXR (large branching opaque mass, usually in L upper abdomen) =   PNA 2/2 proteus  
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PNA on CXR =   segmental infiltrates, atelectasis, pleural effusions; possible empyema  
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Pleura anatomy   Visceral (no sensory nerves) covers lungs, parietal (+sensory nerves) outside that & covering inside of chest wall  
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Decreased breath sounds & tactile fremitus, decreased diaphragmatic excursion on affected side, DTP, egophony =   pleural effusion  
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CXR in chronic bronchitis =   increased AP diameter, thickened bronchial markings, large right side of heart  
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CXR in emphysema =   low flat diaphragm, increased retrosternal space, pruning of vascular markings, long narrow heart, bullae  
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Most sensitive CXR view for smaller pleural effusion:   Lateral. PA may miss up to 300mL  
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ILD on CXR   Early: ground-glass appearance; later: nodular, linear, and/or honeycombed appearance  
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Respiratory failure (hypoxia) on CXR =   small "white" lungs, patchy, diffuse infiltrates, signs of consolidation, and/or lobar atelectasis  
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Respiratory failure (hypercapnia) on CXR =   hyperinflation, large "black" lungs, bullae, increased vascular markings (COPD); in NM dz / drug toxicity, may see small black lungs & hypoinflation  
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Volume to which lungs can be expanded with greatest inspiratory effort   TLC (total lung capacity)  
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volume of air inhaled and exhaled in a minute   MV (minute volume)  
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Part of TV that does not participate in alveolar gas exhange   Dead space  
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