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Summer Sono 2011

Quiz yourself by thinking what should be in each of the black spaces below before clicking on it to display the answer.
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Question
Answer
CLEFT LIP   THE MOST COMMON FACIAL ANOMALY  
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CYSTIC HYGROMA   MOST COMMON NECK ANOMALY  
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CRANIOSYNOSTOSIS   PREMATURE OSSIFICATION OF SUTURES OF THE HEAD  
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HEMIFACIAL MICROSOMIA   ONE SIDE OF FACE IS SMALL AND EAR SET LOW  
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MANDIBLE   LARGE U SHAPE BONE CONSTITUTING THE LOWER JAW, CONTAINS LOWER TEETH  
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MECKEL GRUBER SYNDROME   LETHAL CONDITION ASSO W ENCEPHALOCELE  
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MEDIAN CLEFT FACE   WIDE SEPARATION OF NASAL ELEMENTS, POSSIBLE CLEFTING  
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MICROGNATHIA   SMALL CHIN / MANDIBLE  
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OTOCEPHALY   ABSENT MANDIBLE EARS CLOSE TOGETHER ANT TOWARD NECK  
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5 WEEKS   THE CNS BEGINS TO FORM  
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6 WEEKS   THE SUPERIOR PORTION OF THE FETUS ELONGATES TO FORM THE BRAIN.  
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10-11 WEEKS   MINERALIZATION OF THE BONES  
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18 WEEKS   CEREBELLAR VERMIS DEVELOPS  
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18-20 WEEKS   CORPUS CALLOSUM DEVELOPS  
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7 WK   LIMB BUDS DEVELOP  
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8 WK   OSTEOGENESIS BEGINS IN CLAVICLE & MANDIBLE  
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11-12 WKS   PRIMARY OSSIFICATION CENTERS OF LONG BONES ARE IDENTIFIED  
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4TH MTH   ISCHIUM, METACARPALS OSSIFY  
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5TH-6TH MTH   PUBIS, CALCANEUS OSSIFY  
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CAUDAL REGRESSION   PARTIAL TO COMPLETE SACRAL AGENESIS AND ABNORMALITIES OF THE LUMBAR SPINE, PELVIS, AND LOWER LIMBS  
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THANATOPHORIC DYSPLASIA TYPE I   TYPICAL “TELEPHONE RECEIVER” SHAPE OF THE EXTREMITIES; FRONTAL BOSSING; CRANIOSYNOSTOSIS; PLATYSPONDYLY  
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ACHONDROGENESIS   2ND MOST COMMON LETHAL SKELETAL DYSPLASIA  
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ACHONDROGENESIS TYPE I   20%; SHORT, CUBOID BONES; METAPHYSEAL SCALLOPING; AUTOSOMAL RECESSIVE; THICK, REDUNDANT SKIN FOLDS  
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OSTEOGENESIS IMPERFECTA TYPE II   THE NEONATAL LETHAL FORM OF SKELETAL DYSPLASIA  
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HYPOPHOSPHATASIA CONGENITAL   LETHAL; 1:100,000; THIN APPEARANCE OF BONES  
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CAMPTOMELIC DYSPLASIA   SHORT AND VENTRALLY BOWED TIBIA AND FEMUR; ABSENT OR HYPOPLASTIC FIBULA  
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SHORT-RIB POLYDACTYLY SYNDROME   DECREASED THORACIC CIRCUMFERENCE; POLYDACTYLY; GENITOURINARY ABNORMALITIES  
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HETEROZYGOUS ACHONDROPLASIA   BRACHYDACTYLY, WITH A TRIDENT CONFIGURATION OF THE HAND  
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ASPHYXIATING THORACIC DYSPLASIA   INVERTED “HANDLEBAR” APPEARANCE OF THE CLAVICLES, POLYDACTYLY  
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ELLIS-VAN CREVELD SYNDROME   HIGH PREVALENCE AMONG INBRED POPULATIONS  
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OSTEOGENESIS IMPERFECT TYPE I   BLUE SCLERAE; MOST FRACTURES OCCUR FROM CHILDHOOD TO PUBERTY; PROGRESSIVE HEARING LOSS  
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OSTEOGENESIS IMPERFECT TYPE III   NONLETHAL; PROGRESSIVELY DEFORMING VARIETY THAT OFTEN SPARES THE HUMERI, VERTEBRAE, AND PELVIS  
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PROXIMAL FOCAL FEMORAL DEFICIENCY   ASYMMETRICAL DEGREE OF ABSENCE OF SUBTROCHANTERIC FEMUR; IPSILATERAL FIBULAR HEMIMELIA  
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RADIAL RAY DEFECTS   ABSENCE OF A VISUALIZED FOREARM BONE, ASSOCIATED WITH TALIPOMANUS (CLUB HAND)  
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AASE SYNDROME   HYPOPLASTIC DISTAL RADIUS WITH RADIAL CLUBHAND, AND A TRIPHALANGEAL THUMB; CARDIAC DEFECTS  
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ARTHROGRYPOSIS MULTIPLEX CONGENITAL   MULTIPLE JOINT CONTRACTURES; LIMB PTERYGIUM; “BUDDHA POSITION” OF THE FETUS  
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DECIDUA BASALIS   THICK DECIDUA AT IMPLANTATION SITE  
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DECIDUA CAPSULARIS   THIN, OVERLIES GS FACING UTERINE CAVITY  
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CHORION FRONDOSUM   PORTION OF CHORION AT IMPLANTATION SITE  
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CHORIONIC VILLI   FUNCTIONING UNITS OF PLACENTA  
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COTYLEDONS   COMPOSED OF SEVERAL VILLI AND THEIR BRANCHES  
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RETROPLACENTAL COMPLEX   COMPOSED OF MYOMETRIUM, PLACENTA, AND VESSELS  
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FETAL SURFACE OF PLACENTA   CHORIONIC PLATE  
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BASAL PLATE   DIVIDED INTO COTYLEDONS(COMPOSED OF SEVERAL VILLI & THEIR BRANCHES)  
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