Anatomy- Blood
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red blood cells | transport(O2) from lungs to tissues &(CO2)from tissue to lungs; 1/3 hemoglobin, 2/3 lipids, ATP, carbonic anhydrase
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white blood cells | cells that protect the body from foreign microbes and toxins
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Granulocytes | contain numerous granules in the cytoplasm and have a nucleus that is irregularly shaped with lobes (WBC) Neutro, Eosino, Baso
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Neutrophils | the first WBCs to arrive at a site of infection, responding (by chemotaxis) to chemicals released by damaged cells.engulf bacteria, which are then destroyed by the various antibiotic proteins contained within the granules (granulocyte); 60-70% of WBC
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Eosinophils | enter tissue during inflammatory response; allergic reactions; destroyhistamines
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Basophils | inflammation and allergic response (granulocyte) least common
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Agranulocytes | (WBC) do not have visible granules in the cytoplasm and the nucleus is not lobed (lympho & mono)
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Lymphocytes | antibody production
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Platelets | Cell fragments that form platelet plugs and release chems for blood clotting
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Plasma | liquid portion of the blood; contains water, proteins, waste products, nutrients
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hematoiesis | the process that produces the formed elements of the blood; takes place in the red bone marrow
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proethryoblasts | stem cells develop into RBC
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myeloblasts | stem cells that develop into granulocytes
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albumin | protein in plasma; viscocity, osmotic pressue
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globulins | protein in plasma; transport lipids, carbs, etc
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fibriongen | protein in plasma; blood clotting
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lymphoblats | stem cells develop into lymphocytes
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monoblasts | stem cells develop into monocytes
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megakaryoblasts | stem cells develop into platelets
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iron | required for oxygen transport
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erythropotein | hormone stimulates RBC production; produced by kidneys in response to low blood O2 levels
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chemotaxis | atraction and movement of WBC towards foreign materials or damaged cells; dead WBC + bacteria= pus
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diapedesis | WBC cells become thin, elongate and move between or thru endothial cells of capillaries
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monocytes | break down antigens and present them to lymphocytes for recognition
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hemostasis | arrest of bleeding: vascular spasm, platelets plug, blood clotting
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platelet plug formation | 1. Von Willebrand factor causes adhesion of collagen and platelets2. ADP, thromboxones, etc cause cascade effectfribrogen forms bridge: platelet plug
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coagulation | prothrombinase activate-> converted to thrombin-> fibronigen convert to fibrin. clot= fibrin+trapped blood cells, platelets and fluids
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extrinsic clotting pathway | begins with chems outside blood; damaged tissues release tissue factor; when CA2 is present activates factor X, prothrombinase formed->thrombin->fibrogin to fibrin; thrombin activates factor XII to stabalize clot
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intrinsic clotting pathway | begins with chems part of blood;
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fibronylisis | clot retraction
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blood grouping | determined by antigens on surface of RBCs
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hemolytic disease of newborn | rh+ fetus, rh- mom; in second pregnancy antibodies caused by 1st pregnancy may attack fetal RBCs
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