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HEENT Ears

HEENT Examination

QuestionAnswer
movable cartilage covered with skin auricle/pinna
separated from the mouth by a fold of tissue on each side called anterior tonsillar pillars Oropharynx
lymphoid tissue behind tonsillar pillars Tonsils
movable cartilage covered with skin auricle/pinna
Mastoid process= important Landmark auricle/pinna
S-shaped pathway leading to the Middle ear External Auditory Canal
2.5 to 3 cm. long in adult External Auditory Canal
This canal lining is protected and lubricated with cerumen External Auditory Canal
Lymphatic drainage of the external ear parotid , mastoid, superficial cervical nodes
air filled cavity in the temporal bone Middle ear
It contains the ossicles ( malleus, incus,stapes) that transmit sound from the TM to the oval window of the inner ear middle ear
Conducts sound vibration from outer ear to inner ear Protects the inner ear by reducing the amplitude of loud sounds Eustachian tube allows equalization of air pressure on each side of the ear drum to avoid rupture ( high altitudes) Functions of Middle ear
Contains the Bony Labyrinth which holds the sensory organs for hearing and equilibrium Inner ear
normal pathway of hearing, the most efficient Air Conduction
bones of the skull vibrate and transmit vibrations to the inner ear and acoustic nerve Bone Conduction
begins when the patient responds to your questions and directions. The patient responds without excessive requests for repetition Rough quantitative test for hearing loss
Speech with a monotonous tone and erratic volume may indicate hearing loss Rough quantitative test for hearing loss
Begins with the history:Conversational tone Whisper test
may indicate the presence of hearing loss but not the degree Whisper test
Sounds equal on both ears with normal neurosensory hearing and no conductive loss Positive Weber Test
lateralization of the sound to one ear indicates a conductive loss on the same side or a perceptive loss/sensorineural loss on the other side Negative Weber test
Rinne test= AC>BC both ears Positive Rinne Test
a.Outer ear- cerumen, foreign body,polyp in the external auditory canal Tinnitus
Middle ear – inflammation ,otosclerosis Tinnitus
Internal ear- fever, suppuration of the labyrinth, SY, acoustic nerve tumor fracture at the base of the skull, meniere syndrome Tinnitus
quinine, salicylates, aminoglycosides, gentamicin Tinnitus
pain may arise from inflammation of structure in the ear or be referred from other pharyngeal sites including the thyroid Otalgia
trauma,hematoma,frostbite,burn,eczema, lnsect bites, impetigo, herpes zoster Otalgia (auricle)
otitis externa ,carbuncle, eczema, hard cerumen, FB, herpes zoster Otalgia (middle ear)
unerrupted lower third molar, carious teeth, tonsillitis, carcinoma of pharynx, trigeminal neuralgia , subacute thyroiditis Otalgia (referred)
- described as being unsteady, weak, light headed or having the feeling of turning Dizziness
Idiopathic: multisystem atrophy Infectious: tabes dorsalis, meningitis, encephalitis, brain abscess Metabolic/ nutritional : pellagra, Vit.B1 def.,fluid & electrolyte imbalance Dizziness
Endocrine: hypothyroidism, pregnancy Neurologic :migraine, peripheral neuropathy Vascular: hypertension, orthostatic hypotension Dizziness
persistent stimulation of the semicircular canals or vestibular nucleus when the head is at rest Vertigo
It gives a hallucination of motion When the eyes open, the patients surrounding seems to be whirling or spinning - When the eyes closed, the patient continues to feel in motion Vertigo
Causes: Peripheral labyrinthine System: otitis media with effusion, otosclerosis,temporal bone fracture Central labyrinthine system: migraine,intracranial abscess Vertigo
Cranial VIII infections: Acute meningitis, tumors tuberculous meningitis, Brainstem nuclei: encephalitis, brain abscess,multiple sclerosis, hemorrhage Vertigo
Hearing loss seen in people with external or middle ear problems Conduction deafness
Causes include: obstruction of external auditory canal (FB, impacted cerumen) Disorder of the eardrum & middle ear ( perforated TM, pus/blood in the ME ) Overgrowth of bone with fixation of the stapes ((Otosclerosis) Conduction Deafness
Hearing loss involving the internal ear Sensorineural deafness
Causes: - disorders of the cochlea or the acoustic nerve (CN 8) Aging ( Presbycusis ) due to nerve degeneration Trauma Drug toxicity Tumors infections Heredity/congenital deafness Sensorineural deafness
Microtia small ear
Macrotia Large ear
Satyr ear pointed pinna
aztec or cagot ear lobe development failure
Right angle protrusion of the Pinna Lop or bat ear
untreated hematomas heal as nodular and bulbous irregularities of the helix and antihelix -result of blunt trauma and necrosis of the underlying cartilage Cauliflower ear
harmless developmental eminence in the upper 3rd of the posterior helix Darwin's Tubercle
whitish uric acid crystals covering parts of the ear gouty tophus
due to excessive production of wax or a narrowed meatus leads to partial or complete obstruction of the canal - complete obstruction leads to partial deafness acc. by tinnitus or dizziness cerumen impaction
Seen in Serous Otitis media - more concave TM - accentuated bony landmarks - distorted light reflex Retracted Tympanic Membrane
seen in Acute suppurative otitis media more conical loss of bony landmarks distorted light reflex Bulging tympanic membrane
Yellow otorrhea discharge melted cerumen
serous discharge: eczema in the meatal wall,early ruptured acute OM Otorrhea
Bloody otorrhea temporal bone fracture
chronic external otitis media chronic suppurative OM,cholesteatoma, TB, polyps otorrhea, purulent
Chronic suppurative otitis media ass. with permanent perforation of the eardrum -hearing is always impaired - painless aural discharge - pain and vertigo indicates development of complications like brain abscess
collection of desquamated epithelial cells in the middle ear Cholesteatoma
foul smelling discharge, marginal perforation,hearing loss, pearly gray mass Cholesteatoma
most frequent cause of vertigo Acute Labyrinthitis
patient gradually develop a sense of whirling that reaches a climax in 24-48 hrs. disappear gradually in 3-6 wks Acute Labyrinthitis
Benign Paroxysmal positional Vertigo (BPPV Calcium deposits in the labyrinth ( otoliths) are dislodged and move in response to gravity eliciting a feeling of motion
Benign Paroxysmal positional Vertigo (BPPV More common in older individuals Sudden onset, often when rolling over in bed or arising in the morning No headaches/fever but with nausea and inability to stand
Created by: betahelix
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