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Heart pt.2
A&P II Chapter 20
Question | Answer |
---|---|
Systole | Depolarization, squeeze, positive, Higher BP number (120mmHg is Ideal) contraction |
Diastole | Repolarization, relax, resting, lower BP |
membrane potential | polarity (1/1000 of a volt) |
Cardiac Cycle | Relies on Electricity, measured on ECG/EKG Excitation = contraction of monocytes Higher BP causes Valves to open and close |
Heartbeat | single cardiac contraction all heart chambers contract in series 1st atria 2nd ventricles |
route of electrical conduction through | myocardium |
Rhythmicity | regular heartbeat |
Automaticity | heart makes its own electricity through ATP Spontaneously pump via pacemaker cells (generators) |
Cardiac Myocytes | connected by intercalated discs (only in cardiac) and gap junctions small size, will sync with each other, 1 central nucleus branching interconnections between cells |
Intercalated Discs | connect cardiac contractive discs secured by desmosomes (spot welds) linked by gap junctions for ion movement convey force of contraction make ATP (electricity) |
Two types of Cardiac Myocytes | 1. Conductive Cells 2. Contractile Cells |
Conductive Cells | nodes and electrical branches of autorhythmric cells Modified cardiac myocytes distribute action potentials (electric messages) |
Contractile Cells | systole cardiac muscle cells, produce contractions calcium dependent |
Cardiac Action Potential Phases | Phase 0. Depolarization Phase 1. Brief (early) Repolarization Phase 2. Plateau Phase 3. Repolarization Phase 4. Diastole/Rest Absolute Refractory Period Relative Refractory Period |
Conduction Pathway of Electricity | Sinoatrial node, internodal tracts & Brachman's Bundle, Atrioventricular Node, Atrioventricular bundle of His, Left and Right Bundle Brackets, Purkinje Fibers |
Sinoatrial Node | always firing an action potential highest rate of spontaneous depolarization and sets the pace in post wall of R. atrium near entrance of superior Vena Cava |
Sinus Rhythm | normal electrical pattern established by SA node |
Pacemaker prepotential/ SA node | nodes are always in state of slow depolarization SA & AV nodes dont have a stable resting membrane potential Action potential results to spontaneous depolarization of SA node |
Approaching threshold in nodes | Constant and slow influx of Na, causes nodes to try and approach threshold Ca2+ channels open (Na+ Close) is leaving the cell |
"funny currents" | control beginning of depolarization of SA node |
AV node | delays 100ms, due to small diameter of AV cells allows atria to contract before ventricle contract -spreads to ventricular myocardium so ventricles can begin contraction MAX AP limit = 230/min, higher= damage |
AV Bundle of His | connects atria to the ventricles electrically |
Left and Right Bundle Branches | extend towards apex & fan out to reach Purkinje Fibers, send impulse through moderator band to papillary muscles of R Ventricle |
Purkinje Fibers | fast conductors that reach all ventricular myocytes |
Cardiac Cycle | start of one heartbeat to the start of another one all chambers in the heart experience alternate periods of contraction and relaxation |
5 phases of Systole and Diastole | 1. Atrial Systole (Atrial depol. & contract) 2. Early Ventricular Systole (Vent. depol,) 3. Late Ventricular systole (ventricular ejection) 4. Early ventricular diastole( vent. repol) 5. Late Ventricular diastole (passive vent. filling) |
Early Ventricular Systole | Ventricular depolarization, Iso volumetric ventricular contraction, S1-lub |
Early Ventricular Diastole | Ventricular repolarization, Iso volumetric ventricular S2- dub |
Blood Pressure and Flow | BP in each chamber: rises during systole falls during diastole blood flows high to low pressure -controlled by timing of contractions directed by one- way valves |
Cardiac Output | amount of blood the heart pumps out in 1 min indicates blood flow through peripheral tissues =health of ventricles HRxSV=CO |
Stroke Volume | amount of blood pumped out of ventricle during each contraction SV= EDV-ESV |
End diastolic Volume (EDV) | amount of blood in each ventricle @ end of vent diastole |
End Sytolic Volume (ESV0 | amount of blood in each ventricle @ end of vent systole |
Ejection Fraction | percentage of EDV during contraction (60-80% usually) |
Factors effecting stroke volume | 1.Preload (tension/stretch) due to filling of blood in vent= more volume=more preload 2. Contractility- force of ventricular contraction 3. Afterload- obstacles like pressure that vent must overcome to force semilunar valves open & eject blood |
High BP | bad bc resistance |
Normal HR | young adult females- 72-80bpm young adult male- 64-72bpm |
Ecoptic Pacemaker | abnormal cells generate high rate of APs bypasses conducting system disrupts timing of ventricular contractions |
Neurotransmitters change cardiac output | Norepinephrine- used by SNS -speeds up HR (more O2) Acetylcholine- used by PNS -slows HR via Vagus nerve |
Intropic agents | affect contractility of the heart + factors strengthen heart contraction (more Ca2+) - factors weaken heart contraction (less Ca2+) |
Chrontropic agents | influence HR + increase HR - decrease HR affect electrical conduction system of the heart by affecting nodes |
Right Sided Heart Failure | R. Ventricular Fail -vena cava backup and swelling Systemic edema (esp; legs and feet), enlarged liver and spleen, JVD, ascites |
Left Sided Heart Failure | L. ventricular fail pulmonary backup and drown SOB, pulmonary edema, coughing |