click below
click below
Normal Size Small Size show me how
Path Male Genitals
| Question | Answer |
|---|---|
| cryptorchidism | 4% of newborns no descent of the testes ( increased risk of germ cell tumor and infertility) |
| Testicular torsion | testis twist on their cord resulting in hrmorrhagic infarct and venous occlusion violence |
| Paradoxical torsion | during sleep |
| Varicocele | venous plexus enlarges resulting in increased blood flow and infertility? |
| Hydrocele | intestines in the testis |
| all germ cell testiculular tumors have the word? | seminoma |
| Seminoma | most common yellow tan with no necrosis INCREASED HCG |
| Anaplsatic seminoma | more pleomorphic with more metosis form ALL are radiation sensitive Increased HCG |
| Spermatocytic Seminoma | more differenticated NEVER MIXED with no counterpart and almost NEVER mets |
| ovarian counterpart to testicular seminoma | Dysgerminoma |
| Embryonal cell carcinoma | pleomorphic hemorrhagic and necrotic |
| Polyembryona cell carcinoma | embryonal cell carinoma with many embryo like structures that is VERY METASTATIC via lymphatics and hamatagenous routes |
| Yolk sac Tumor | like ovarian named via Schiller duval bodies and Secretes Alpha feto protein common in young boys as pure and older men as mixed |
| schiller duval bodies | resemble rat embryos found in yolk sac tumors of the ovary and testes |
| yolk sac tumors are mostly mixed with? | embryonal cell tumors |
| Teratoma | if prepuberty benign if post think malignant |
| Teratocarcinoma | a mix of embryonal and teratoma tumors |
| choriocarcinoma | PURE RARE FATAL- need cytotrophoblast and syncytiotrophoblast with hemorrhage and necrosis in young with HEMOPTYSIS and HCG producted |
| Mixed Choriocarcinoma | exception to the rule any mixing makes this better . reactive to treatment but very destructive so that it is still possible to cause mortality |
| Mixed Germ Cell Carcinoma | 1/2 will be mixed very common take prognosis of worse except with choriocarcinoma |
| Stromal Tumors | Leydig and Sertoli cells may be hormonally active often benign |
| Congenital Penile diseases | hypospadius (ventr) epispadius (dorsal) phimosis(small prepuce) Paraphimosis (forced over prepuce) |
| Infections of the penis | balanoposthitis, peyronies, priapism |
| Condylomas of the penis | condyloma lata and giant condyloma of Buschke lowenstein |
| Carcinoma in situ of the penis | aka erythroplasia of queyrat or Bowens pink red velvety plaques that are hyperchromatic and have atypical mitotic figures NO METS POTENTIAL |
| Bowenoid papulosis | in young boys does not seem to progress |
| concyloma lata | secondary syphilis (NOT VIRAL) |
| Giant condyloma of buschke lowenstein | well differentiated verrucous SCC |
| Balanoposthitis | non specific inf of staph or strep |
| Peyronie | proliferation of fibrotic tissue results in curvature w/erection |
| Priapism | persistant erection |
| Congenital problems with the prostate | Stenotic urethral valve resulting in hydronephrosis and failure |
| Nodular hypeplasia | dilation of the prostate common with advancing age 90% in men over 70 AFFECTS THE CENTRAL PORTION |
| how do you treat Nodular and etiology | from imbalance of testosterone resulting in hyperplasia treat with 5 alpha reductase to block testoster conversion to dihydrotestosteron |
| Adenocarcinoma | most common carcinoma in men rare before age 50 affects the periphery of the prostate which is why a rectal exam is so important can be treated with follow up alone |