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liver path

liver pathology

hepatocellular carcinoma associated with pre-existing cirrhosis especially HBV, aflotoxin, increased alpha feto protein
Cholangiocarcinoma not associated with HBV, origionates from intrahepatic biliary epithelium
hemangiosarcoma associated with toxic exposure to polyvinyl chloirde, thorotrast and arsnic
hemiangioma most common benign tumor
adenoma asoociated with oral contraceptives, may rupture and cause intraperitoneal bleeding
pre hepatic HTN caused by portal and splenic vein obstruction
Intrahepatic HTN caused by cirrhosis/tumors
posthepatic HTN congestion in distal hepatic veins, CHF, constrictive pericarditis
inborn errors of metabolism that cause liver disease apha-1 antitrypsin deficiency, galactosemia, and glycogen storage diseases
Wilson diesease characterized by decreased serum ceruloplasmin, manifests as liver disease and invovles basal ganglia, marked by Kayser-Fleisher ring
hereditary hemochromatosis defect in iron avsorption resulting cirrhosis, diabetes, and increased pigmentation. Marked increased serum iron and decreased TIBC
secondary hemochromatosis caused by multiple transfusions and inefective erythropoiesis
Primary biliary cirrhosis most likely autoimmune, anti-mitochonddial antibodies, characterized by severe obstructive jaundice, itching and hpercholesterolemia
secondary biliary cirrhosis cuased by extrahepatic obstruction
clinical manifestations on alcoholic cirrhosis jaundice, hypoalbumineamia, coagulation factor deficienceis, hyperestrinism
consequense of increased portal venous pressure esophageal varices, rectal hemorrhoids, periumbilical venous collaterals, splenomegaly
Reye syndrome acute disorder of young children characterized by encephalopathy, coma, and microvesicular fatty liver, associated with aspirin administration with acute viral infections
Chronic hepatits may result from any viral hepatide except A&E, msy be autoimmune marked by anti smooth muscle antibodies
Hepatitis E enterically transmitted water bourn. not associated with chronic hepatitis
HDV replicatively defective, requires simultaneous infection with HBV, transmission sexual or parenteral
HCV transmitted parenterally, often leads to carrier state and chronic hepatitis, frequectly associated with carcinoma
HBV transmitted via parenteral,sexual and vertical routes, associated with hepatocellular carcinoma, can result in carrier or chronic hep.
HAV fecal oral transmission, does not cause carrier state or chronic hepatitis
Dubin-Johnson syndrome conjugated hyperbilirubinemia, discoloration of liver, caused by defective bilirubin transport
Crigler-Najjar syndrome unconjugated hyperbilirubinemia caused by a deficiency of glucuronlyl transferase
Crigler-Najjar syndrome leads to kernicterus but may respond to phenobarbital therapy
Gilbert Syndrome common, elevated nconjugated bilirubin, no clinical consequences
Created by: swohlers