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Cards HTN Tx Fill In The Blanks

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Question: Firstline tx in HTN: MOA (eg, , BB)Answer: decrease volume, decrease afterload and systolic BP
Question: Most type of diuretics & MOA = Answer: loop (furosemide): inhibit Cl reabsorption in TAL -> Na Cl and H2) follows Na&Cl. Results in high K+ losses.
Question: diuretics MOA =Answer: HCTZ & : inhibit K+ & Na+ reabsorption in TAL. Decrease plasma volume, increase Na excretion
Question: Thiazide AEs =Answer: Volume depletion, low K+ & Na, / insulin resistance; hyperuricemia/gout, photosensitivity, decreased placental flow. May increase serum cholesterol & digoxin
Question: Hypertensive : txAnswer: Controlled, gradual lowering of BP; 10% decrease in first hour, then 15% over next 3–12 hrs to BP of no less than 160/110; rapid correction of BP to normal levels puts pt at high risk for worsening cerebral, renal or ischemia
Question: Syndrome: tx for HTN Answer: HTN is reversible if cause is eliminated (ie, adenoma, corticosteroid Rx)
Question: Beta : MOAAnswer: Blockade of parts of the NS (reduce PVR); Lowers HR; Initially lowers cardiac output; Reduces circulating renin
Question: ACEI AEs Answer: Cough; Angioedema; Hyperkalemia & hyponatremia (2/2 low aldosterone); Rash. CI in , and use cautiously in renal artery stenosis (RAS)
Question: ARBs: Answer: Act to block Angiotensin II from binding points; same effect as ACEI, without some of the
Question: ARBs AEs Answer: Hyperkalemia; Angioedema (rare, 10% cross-over); CI in PG; Cautious use in
Question: CCBs: MOA Answer: Inhibit Ca+ -> block vascular smooth mx contractility -> vasodilatation & afterload reduction (aoso preload). Also coronary vasodilatation: CCBs are used in coronary artery spasm (Prinzmetal, Raynaud)
Question: CCBs: Dihydropyridines (DHPs) vs Answer: DHPs more vascular selective (fewer cardiac conduction fx): tx HTN/angina. more cardio-selective with more inhibitory fx on SA/AV node (CAUTION in CHF)
Question: DHP CCBs (amlodipine, ): AEs Answer: Ankle ; Flushing; HA; Increased HR
Question: Non-DHP CCBs (diltiazem, ): AEs Answer: Bradycardia, constipation, hotn, edema, CHF, AV node
Question: HTN lifestyle Answer: Wt reduction (BMI 18.5 - 25) (biggest fx on bp); ETOH; activity 30 min; Na+ to 2.4 mg/day; K+; DASH diet
Question: Most single HTN meds BP:Answer: at most 20/10 mm Hg (so most pts on more than 1 drug)
Question: Beta-1 stimulation causes (a), and beta-2 causes (b)Answer: (a) tachycardia; (b)
Question: BBs (2) used for migraine, SVT, V-tach, , EtOH W/D =Answer: Inderal (propranolol) and
Question: BB used to reduce IOP in = Answer: nadolol
Question: BBs are in:Answer: any resp dz (asthma, COPD, DF); type 1 DM. Also pt may have W/D and increase in CP (2/2 CAD) bc of increased HR & thus O2 demand
Question: CCBs are in:Answer: CHF, LV dysfunction, AV block, sick syndrome
Question: Line Tx for HTN Answer: *Thiazide*; beta; ACEI; ARB; diuretics; CCB
Question: ACEI MOA:Answer: Inhibit ACE in lung -> block formation of angiotensin II -> vasodilatation and Na+ loss
Question: ACEI work less well in AA bc of:Answer: blood renin levels
Question: Rx of for DM nephropathyAnswer: ACEI (increase blood flow to kidneys -> promote renal ); act synergistically with a diuretic
Question: Presynaptic adrenergic release inhibitors: 2 Answer: central and
Question: Central presynaptic release inhibitors (anti-adrenergic meds) MOAAnswer: prevent adrenergic outflow from the brain, by stimulating alpha-2 receptors (eg, clonidine)
Question: presynaptic adrenergic release inhibitors (anti-adrenergic meds) MOA Answer: norepinephrine release from peripheral nerve terminals (eg, in heart) -> block alpha-1 receptors (eg, prazosin)
Question: Sudden DC of anti-adrenergic meds can :Answer: HTN
Question: Firstline & 2ndline HTN tx in patient =Answer: 1st: methyldopa (Aldomet). 2nd:
Question: indications of anti-adrenergic meds Answer: Central: nicotine/heroin W/D; Ppx for , glaucoma, DM-assoc diarrhea. Peripheral: BPH; asthma (relax smooth mx); meds lower LDL & inc HDL
Question: Nitrates Answer: 1) dilate large myocardial arteries -> increase blood flow to heart; 2) reduce venous tone -> blood pooling in periphery -> reduces preload -> reduces cardiac work (so: increase O2 & decrease O2 demand)
Question: AEsAnswer: hotn, rebound tachycardia, flushing, HA
Question: JNC8: tx of uncomplicated HTN for most (non-AA) Answer: Firstline: ACEI, ARB, CCB, diuretics
Question: JNC 8: HTN tx for AA patientsAnswer: CCBs and diuretics
Question: For pts with DM regardless of race), HTN tx include:Answer: ACEI or
Question: Hypertensive txAnswer: nitroprusside (CI in PG) or labetolol; to 110 over hours
Question: HTN Compelling Indications: Answer: 1st: ACE plus diuretic (HCTZ / Lasix); 2) Beta (atenolol); 3) clonidine. Also ARB
Question: HTN Compelling Indications: High Dz Risk Answer: Beta; ACEI; CCB;
Question: HTN Indications: Post-MI Answer: 1 Beta; 2 ACEI; CCB
Question: HTN Compelling Indications: Answer: 1 ACEI; 2 Beta (for DM2); 3 Verapamil or
Question: HTN Indications: Recurrent Stroke Prevention Answer: ACEI;
Question: JNC 8: in >60, start tx at:Answer: SBP >150 or DBP >90
Question: JNC 8: in <60, start tx at:Answer: 140/90 (same used in pts >18 yo with CKD or DM)
Question: (&2nd & 3rd) tx for uncomplicated isolated systolic HTN (elderly)Answer: 1) HCTZ; 2) ; 3) captopril or verapamil
Question: HTN Indications: CKDAnswer: 1 ACE; 2 BB (atenolol) or or HCTZ; 3 clonidine
Question: Tx for Answer: Prazosin +/-
Question: Tx for (w/HTN meds):Answer: 1 atenolol; 2
Question: Tx (w/HTN med):Answer:
Question: Tx for pre-op Answer:
Question: 1stline HTN tx in Answer: 1 ; 2 hydralazine
Question: Pt just started on ACEI develops weakness. What test (lab, UA, CT, US) is most to dx pt?Answer: Renal
Question: HTN Tx in Answer:
Question: HTN Tx in Answer:
Question: Alternate to ACEI if CHF or other Answer: ARB
Question: HTN tx if BP >20/10 over goal:Answer: 2
 
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