Fundamentals- Cardio
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each of the black spaces below before clicking
on it to display the answer.
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| show | Arterioles
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| What effect does Alpha receptor stimulation have on arterioles? | show 🗑
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| show | False. Modified by sympathetic activity and vasoactive substances
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| show | Depends on the receptor. Alpha would cause vascular constriction and arterial shift. Beta-2 would dilate the veins causing venous shift.
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| Pt has large amount of blood loss. What can their body (autonomic nervous system) do to compliance of the veins to ensure adequate circulating blood flow? | show 🗑
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| Pt has pulmonary edema resulting from CHF. How can you treat by changing venous compliance? | show 🗑
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| show | Radius
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| show | Dec'd resistance and BP.
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| show | blood flowing over obstruction
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| Pulse pressure | show 🗑
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| show | diastolic pressure + 1/3 pulse pressure
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| show | volume of blood ejected from one ventricle in a single cycle.
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| In what tissue of the heart are conducting cells found? | show 🗑
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| An increase in conduction velocity of the AV node will do what to stroke volume/ CO? | show 🗑
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| show | It is relatively stabilized during phase 2.
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| If SA node is blocked/damaged which latent pacemaker takes over and roughly what will HR be? | show 🗑
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| show | NO PHASE 1 or 2. NO PLATEAU. UNSTABLE MEMBRANE POTENTIAL. "funny" inward Na current causes slow depolarization of phase 4 --> next AP.
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| Which phase of SA node AP accounts for its automaticity? | show 🗑
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| Parasympatomimetic will affect what region of the heart? | show 🗑
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| show | Dec'd contractility, parasympatomimetic.
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| An increase in HR has what effect on contractility? | show 🗑
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| show | Positive ionotropic
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| show | Shift to the right. Greater volume at same phases/pressure.
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| Which is more costly for the heart (in terms of inc'd myocardial O2 consumption) : Pressure work (from afterload) or volume work? | show 🗑
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| Common pathology r/t chronic excessive workload? | show 🗑
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| How does ventricular hypertrophy effect CO? | show 🗑
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