Clinical Medicine-II
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| show | R: renal collecting ducts, D: arise anywhere in the nephron
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| show | Autosomal recessive PKD
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| Where is the mutation for recessive | show 🗑
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| What is a common feature of recessive PKD | show 🗑
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| show | PKD causes progressive kidney enlargement
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| Is this 100% genetic | show 🗑
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| show | PKD 2 and females
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| Why do PKD pts do so well on dialysis alone | show 🗑
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| Clinical features of PKD | show 🗑
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| show | can get infected, must be drained
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| How do we dx PKD | show 🗑
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| Tx for PKD | show 🗑
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| What is the worst thing a pt could do to worsen ANY kidney dz | show 🗑
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| What is goal tx of BP with PKD | show 🗑
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| show | liver cysts, RF: females exposed to estrogen: preggo, and oral contraceptives
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| Besides the liver cysts, what else can PKD precipitate | show 🗑
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| What is rhabdomyolysis | show 🗑
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| Where do we see rhabdo | show 🗑
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| show | physical trauma, ↑physical activity-szs, movement d/o’s, compromised flow to muscle→necrosis leads to release of CK, Drugs, Toxins, ↑↓temps, infectious causes and bites
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| What drugs may cause Rhabdo | show 🗑
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| Toxins that may cause rhabdo | show 🗑
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| show | pink urine, ↑CK: >10-20,000, evidence of renal failure,
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| Tx of rhabdo | show 🗑
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| What is mannitol | show 🗑
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| show | rhabdomyolysis
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| show | it is red, but has no blood cells, red d/t myoglobin
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| show | 40-90%!
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| show | pre-renal, intra-renal, post-renal (need to be able to tell the diff)
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| show | anything that may reduce renal perfusion: vol depletion, hypotension, CHF, arrhythmias,, intrarenal vasoconstriction? hypercalcemia, hepatorenal syndrome
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| What are some drugs that may ↑intrarenal vasoconstriction | show 🗑
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| What are intrarenal failure causes | show 🗑
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| Is cortical and tubular necrosis reversible | show 🗑
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| What is acute interstitial nephritis | show 🗑
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| Post renal causes | show 🗑
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| Any female that has bilateral obstruction of the kidneys has what | show 🗑
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| show | Oliguric, <400ml/24hrs
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| Complete obstruction of urine, major vascular catastrophy, commonly severe ATN | show 🗑
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| When is oliguria seen | show 🗑
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| show | intrarenal causes, nephrotoxic ATN, acute GN, AIN
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| show | altered taste sensation
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| Hiccups | show 🗑
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| show | pre-exisiting illness, uremic sx, small echogenic kidneys by US
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| show | FENA and BUN
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| show | prerenal or obstruction
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| show | ↑ calcium absorption: calcitriol, Stimulates RBC production, Regulates BP and electrolytes: renin
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| show | sudden, rapid ↓ urine output, usually reversible, tubular cell death with regeneration
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| show | progressive, not reversible, nephron loss, can lose ~75% renal fxn prior to having sxs
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| show | uraemia
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| show | Stage 1-5
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| Kidney damage w/ nl or ↑ GFR | show 🗑
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| show | stage 4 15-29
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| Kidney failure | show 🗑
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| show | stage 2, 60-89
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| Mod ↓ GFR | show 🗑
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| Causes of CKD | show 🗑
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| show | sustaining primary dz, HTN, intraglomerular HTN, proteinuria, nephrocalcinosis, Dyslipidemia,
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| show | lipid lowering agent , and ACEi (if they can be) (statins: have a factor that help the kidney “deal” with CKD)
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| how can we slow pregoression of CKD | show 🗑
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| Sxs CKD | show 🗑
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| show | oral Na+ bicarb
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| MC cause of renal failure | show 🗑
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| What are two types of dialysis | show 🗑
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| show | Not done :D
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Created by:
becker15
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