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Stack #36569
Heart
| Question | Answer |
|---|---|
| transverse pericardial sinus | allows access to pass ligatures around aorta & pulmonary trunk |
| Pericarditis | inflammation of pericardium |
| Pericarditis pain located at | top of shoulder b/c sensory innervation of pericardium from phrenic n. (friction heard w/stethoscope) |
| Cardiac tamponade | accumulation of fluid inside pericardium puts pressure on heart & makes it difficult to pump blood |
| R. atrium contains | pectinate mm., crista terminalis, sinsus venarum, fossa ovalis, ostium for coronary sinus |
| R. ventricle contains | tricuspid valve, chordae tendinae, papillary mm., septomarginal trabeculum(moderator band), conus arteriosus, pulmonary valve |
| R. ventricle experiences | hypertrophy when vascular resistance in lung is high as w/COPD |
| L. ventricle contains | thickest wall, mitral valve, chordae tendinae, papillary mm., aortic valve |
| L. ventricle experiences | hypertrophy in congestive heart failure |
| R. Coronary a. contains | SA nodal, R. marginal, AV nodal, posterior interventricular |
| L. Coronary a. contains | (SA nodal), anterior interventricular aka "LAD", circumflex |
| Great cardiac vein | runs with anterior interventricular a. |
| Middle cardiac vein | runs with posterior interventricular a. |
| Small cardiac vein | runs with right marginal a. |
| Cardiac sinus | recieves drainage from great, middle, small cardiac vv. |
| Cardiac sinus opens into | r. atrium |
| anterior cardiac v. drains | directly into r. atrium |
| single lymphatic vessel drains | lymph from heart to inferior tracheobronchial nodes |
| Sinoatrial (SA) node | located in wall of r. atrium at end of crista terminalis |
| atrioventricular (AV) node | located on floor of r. atrium next to septum |
| AV node transmit signal to | inferior aspect of ventricles via r. & l. bundle branches. |
| Moderator band has | many fibers from the r. bundle pass through it |
| Heart Sympathetics | preganglionic and postganglionic fibers |
| Preganglionic fibers | exit from T1-T4 |
| Postpanglionic fibers | located in local chain ganglion & inferior (stellate), middle, & superior cervical ganglia |
| Heart Parasympathetics | from vagus n. |
| Cardiac Plexus | Superficial & Deep |
| Superficial Cardiac Plexus | anterior to arch of aorta & projects mainly to l. ventricle |
| Deep Cardiac Plexus | near bifurcation of trachea, projects mainly to posterior, Right SA node |
| Surface anatomy of heart | superior border from inferior border of 2nd costal cartilage to right of sternum to top of 3rd costal cartilage on left (2nd intercostal space: hight on right, low on left) |
| Inferior heart surface anatomy | inferiorly from 6th costal next to sternum on right passing along xiphisternal junction at midline |
| Apex located | in 5th intercostal space near midclavicle line |
| Atrial Septal Defects (ASDs) | result of incomplete closure of fossa ovalis & small ones (probe patent) |
| Atrial Septal Defects (ASDs) occur | in about 1/5 of population |
| Ventricular Septal Defects (VSDs) | most common heart malformations |
| Ventricular Septal Defects (VSDs) occur | btwn. membranous & muscular portion of septum. Cause a L. to R. shunt which may result in pulmonary hypertension |
| Mitral valve | most commonly damaged |
| Mitral valve damage due to | rheumatic fever |
| Mitral valve damage results in | incomplete closure results in backflow |
| Murmur | backflow is audible through stethoscope at apex of heart referred to as a murmur |
| Aortic valve problems | cause collapsing pulse w/characteristically high pulse pressure (difference btwn. systolic & diastolic BP) |
| Coronary Artery Disease | impaired blood flow to heart due to atherosclerosis & may cause angina pectoris (chest pain) |
| Sudden occlusion of coronary vessel produces | ischemia (lack of sufficient blood) |
| Ischemia of heart | may result in death of heart m. |
| myocardial infarction (MI) | heart attack |
| common site of infarct | vascular tree of LAD |
| infarction involving AV node | atria continu in normal rhythm, but ventricle beat at slower inherent rate w/no coordination of function |
| Bundle branch block | unaffected side beats firts then impulse passes to other side causing temporal separation of ventricular contraction |