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Stack #36569
Heart
Question | Answer |
---|---|
transverse pericardial sinus | allows access to pass ligatures around aorta & pulmonary trunk |
Pericarditis | inflammation of pericardium |
Pericarditis pain located at | top of shoulder b/c sensory innervation of pericardium from phrenic n. (friction heard w/stethoscope) |
Cardiac tamponade | accumulation of fluid inside pericardium puts pressure on heart & makes it difficult to pump blood |
R. atrium contains | pectinate mm., crista terminalis, sinsus venarum, fossa ovalis, ostium for coronary sinus |
R. ventricle contains | tricuspid valve, chordae tendinae, papillary mm., septomarginal trabeculum(moderator band), conus arteriosus, pulmonary valve |
R. ventricle experiences | hypertrophy when vascular resistance in lung is high as w/COPD |
L. ventricle contains | thickest wall, mitral valve, chordae tendinae, papillary mm., aortic valve |
L. ventricle experiences | hypertrophy in congestive heart failure |
R. Coronary a. contains | SA nodal, R. marginal, AV nodal, posterior interventricular |
L. Coronary a. contains | (SA nodal), anterior interventricular aka "LAD", circumflex |
Great cardiac vein | runs with anterior interventricular a. |
Middle cardiac vein | runs with posterior interventricular a. |
Small cardiac vein | runs with right marginal a. |
Cardiac sinus | recieves drainage from great, middle, small cardiac vv. |
Cardiac sinus opens into | r. atrium |
anterior cardiac v. drains | directly into r. atrium |
single lymphatic vessel drains | lymph from heart to inferior tracheobronchial nodes |
Sinoatrial (SA) node | located in wall of r. atrium at end of crista terminalis |
atrioventricular (AV) node | located on floor of r. atrium next to septum |
AV node transmit signal to | inferior aspect of ventricles via r. & l. bundle branches. |
Moderator band has | many fibers from the r. bundle pass through it |
Heart Sympathetics | preganglionic and postganglionic fibers |
Preganglionic fibers | exit from T1-T4 |
Postpanglionic fibers | located in local chain ganglion & inferior (stellate), middle, & superior cervical ganglia |
Heart Parasympathetics | from vagus n. |
Cardiac Plexus | Superficial & Deep |
Superficial Cardiac Plexus | anterior to arch of aorta & projects mainly to l. ventricle |
Deep Cardiac Plexus | near bifurcation of trachea, projects mainly to posterior, Right SA node |
Surface anatomy of heart | superior border from inferior border of 2nd costal cartilage to right of sternum to top of 3rd costal cartilage on left (2nd intercostal space: hight on right, low on left) |
Inferior heart surface anatomy | inferiorly from 6th costal next to sternum on right passing along xiphisternal junction at midline |
Apex located | in 5th intercostal space near midclavicle line |
Atrial Septal Defects (ASDs) | result of incomplete closure of fossa ovalis & small ones (probe patent) |
Atrial Septal Defects (ASDs) occur | in about 1/5 of population |
Ventricular Septal Defects (VSDs) | most common heart malformations |
Ventricular Septal Defects (VSDs) occur | btwn. membranous & muscular portion of septum. Cause a L. to R. shunt which may result in pulmonary hypertension |
Mitral valve | most commonly damaged |
Mitral valve damage due to | rheumatic fever |
Mitral valve damage results in | incomplete closure results in backflow |
Murmur | backflow is audible through stethoscope at apex of heart referred to as a murmur |
Aortic valve problems | cause collapsing pulse w/characteristically high pulse pressure (difference btwn. systolic & diastolic BP) |
Coronary Artery Disease | impaired blood flow to heart due to atherosclerosis & may cause angina pectoris (chest pain) |
Sudden occlusion of coronary vessel produces | ischemia (lack of sufficient blood) |
Ischemia of heart | may result in death of heart m. |
myocardial infarction (MI) | heart attack |
common site of infarct | vascular tree of LAD |
infarction involving AV node | atria continu in normal rhythm, but ventricle beat at slower inherent rate w/no coordination of function |
Bundle branch block | unaffected side beats firts then impulse passes to other side causing temporal separation of ventricular contraction |