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Digestion

Digestion summary

QuestionAnswer
breakdown of starch in mouth Ptyalin (salivary amylase)
starts breakdown of fat in mouth sublingual lipase
glands that give rise to parietal cells (oxyntic cells), and chief cells (peptic cells) gastric glands
cells produce pepsinogen chief/peptic cells
converts pepsinogen to pepsin for protein digestion HCl
Produces HCl Parietal/ oxynitic cells
Produces intrinsic factor Parietal/ oxynitic cells
Where does B12 absorption occur terminal ileum
mucous producing cells in the stomach mucous neck cells AKA cardiac gastric cells
glands give rise to gastric cells pyloric glands
produce gastrin gastric cells
mucous secreting cells in the small intestine, duodenum Brunner's glands
buffers acid chyme from stomach bicarbonate
helps cause release of pancreatic enzymes water
secretin triggers from pancreas bicarbonate and water
causes production of pancreatic amylase, lipase, and trypsinogen cholecystokinin
closes pyloric sphincter in response to fats cholecystokinin
inhibits gastrin in stomach cholecystokinin
causes gallbladder to release bile cholecystokinin
creates micelles which carry digested FAT to the microvilli of the lacteal glands for absorption into LYMPHATICS as CHYLOMICRONS bile
long chain fatty acid absorption lymphatics
medium chain fatty acid absorption lymphatics or portal system
short chain fatty acid absorption portal system to liver
Along with cholecystoknin, also helps close pyloric sphincter in response to fats enterogastrone
converts tryspinogen to trypsin for breakdown of protein enterokinase
trypsin activates wich proteolytic enzymes chymotrypsinogen, proteolastase, carboxypeptidase
breaks down polypeptides chymotrypsinogen
digests elastin fiber that holds meat together proteolastase
helps break into individual amino acids then sent to liver carboxypeptidase
protects pancreas from digesting itsself (pancreatitis) trypsin inhibitor
mucous production small intestine goblet cells
glucose+galactose lactose
glucose+fructose sucrose
glucose+glucose maltose
shut off appetite centers in hypothalamus leptin (from fat cells)
promotes appetite ghrelin (from stomach)
increase appetite from hypothalamus neuropeptide Y
helps insulin bring glucose into cell adiponectin
when you've eaten enough peptide YY
from Delta cells of pancreas somatostatin
somatostatin inhibits: insulin and glucagons, gastin and secretin, and rennin
form argentaffin cells in stomach, satiation serotonin
Created by: polystachya