lecture 19 hoffmann
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| show | mucosal hemorrhages and petechiae, OJO - risk of CNS hemorrhage! failure to stop bleeding after initial injury, bleeding time is prolonged and plt # or morphology is abnormal
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| examples of primary hemostasis disorders | show 🗑
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| pseudothrombocytopenia | show 🗑
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| information that mean plt volume (MPV) gives clinician | show 🗑
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| one cause of thrombocytopenia = bone marrow infiltration by leukemia/lymphoma, metastatic ca, TB, CMV/HIV infection, etc, which implies what about plts? | show 🗑
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| reasons for increased destruction of plts, one cause of thrombocytopenia | show 🗑
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| causes of sequestration of plts or altered distribution | show 🗑
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| show | microangiopathic processes - there is RBC shearing by fibrin deposition within vessels as seen in DIC or TTP
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| dacrocytes/nucleated RBCs + thrombocytopenia suggest? | show 🗑
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| spherocytes + thrombocytopenia suggest? | show 🗑
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| macrocytes + thrombocytopenia suggest? | show 🗑
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| microcytes, hypochromia, aniso- and poikilocytosis + thrombocytopenia suggest? | show 🗑
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| spur cell anemia | show 🗑
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| show | PCNs, cephalosphorins, sulfa drugs // heparin // furosemide, thiazides, methyldopa // carbamaszepine, Depakote // H2 receptor agonists, cocaine
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| most common cause of plt dysfunction | show 🗑
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| show | acts as a bridge btwn exposed vascular subendothelium and plt glycoprotein receptors (GPIb); acts as transport protein for VIII in blood, protects it from inactivation and increases its half-life
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| features of vW dz | show 🗑
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| lab diagnostic tools for vW dz | show 🗑
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| scurvy | show 🗑
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| leukocytoclastic vasculitis | show 🗑
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| Osler-Weber-Rendu dz aka hereditary hemorrhagic telangiectasia | show 🗑
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| show | hemarthrosis (deep tissue and joint bleeding), delayed onset, deep hematomas in muscle compartments and retroperitoneum
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| PT extrinsic pathway (starting at VIIa), PTT intrinsic pathway (starting at XII) | show 🗑
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| show | factor VII has shortest half-life in plasma
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| information that a 1:1 dilution of pt plasma with nl plasma tells you | show 🗑
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| show | caused by factors VIII/IX respectively, both X-linked recessive
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| TT or thromboplastin time | show 🗑
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| show | severe vit K deficiency, warfarin overdose, malabsorption of vit K/pt on TPN, DIC, liver dz, congenital factor I II V or X deficiencies
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| how to distinguish btwn liver dz and DIC when both cause PT/INR and PTT to be elevated | show 🗑
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| show | b/c it turns over so quickly especially in liver dz
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| show | IV Ig and prendnisone; Ig binds macrophages and distracts them from eating up plts until the prendnisone can work to decrease immune response
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