thermod notes
Quiz yourself by thinking what should be in
each of the black spaces below before clicking
on it to display the answer.
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| show | diagnosis or findings of physical examination
recommendaed treatment procedures and rehab plan
prognosis if the recommended treatment is administered or rehab plan is complete
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| show | risks and benefits of the recommended treatment and rehab
reasonable alternatives to proposed plan, with potential risks, benefits and prognosis
prognosis if not treatment is administered
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| show | 1. control pain and swelling, protect injury 2. restore ROM 3. restore neuromuscular control and muscular endurance 4. restore strength and power 5. resume sport specific training 6. return to physical activity
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| show | epithelial, connective, muscle, nervous
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| Epithelial tissue | show 🗑
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| Types of injury to epithelial tissue | show 🗑
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| Connective Tissue types | show 🗑
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| show | supports and protects the body
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| Injuries to connective tissue | show 🗑
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| Types of muscle tissue | show 🗑
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| Skeletal muscle | show 🗑
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| show | slow twitch-type 1
intermediate fibers type II A
fast twitch type II B
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| Types of injuries to skeletal muscle | show 🗑
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| Nerve tissue | show 🗑
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| Types of injury to nerve tissue | show 🗑
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| show | Cancellous or spongy bone
Cortical or compact bone
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| show | Redness, heat, swelling, pain, loss of function
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| Redness | show 🗑
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| Chemical mediators | show 🗑
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| show | causes vasodilation, heat and redness
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| Bradykinins | show 🗑
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| Prostoglandins | show 🗑
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| show | increases capillary membrane permeability, swelling
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| show | increase blood flow to the injured area increases heat, chemical activity increases metabolic activity
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| show | GOAL is to releive athlete of noxious stimulous=physical, chemcial and cell anoxia
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| show | from the trauma to the area
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| Chemcial Pain | show 🗑
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| Cell anoxia | show 🗑
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| show | secondary to the increased blood flow to the area and broken blood vessels, exudate, tranudate
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| Exudate | show 🗑
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| transudate | show 🗑
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| show | torn blood vessels cell death-increases osmotic extracellular pressure(drives fluid from capillaries) increaded protien gravity chemical mediators-alters cell permeability to protien and fluid
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| Loss of Function | show 🗑
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| show | inflammation(injury to 4 days), proliferation/fibroblastic(4 days to 3 weeks), remodelling(3 weeks to one year)
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| Goal of the inflammation phase | show 🗑
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| show | Tissue injury Vasoconstiction-increase in sympathetic actiivity(shock lasts for 5 sec-10 min), decreases blood flow so clotting can occur release of chemical mediators by mast cells-increases bf, vasodilation, block lymph channels to prevnt systemic inf
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| show | PMNL-polymorpho nuclear luekocytes-leukocytes(macrophages and neutrophils)-chemotaxis occurs Diapedesis-movement of WBC and other substances through damaged cessels into the tissue phagocytosis
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| Chemotaxis | show 🗑
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| show | Neutrophils-clean up dead cells-kill off other cells in the process Macrophages-form lysomes to destroy and increse phagocytosis
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| Treatment during inflammation phase | show 🗑
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| show | impart strength to wound increase epitheliazation increase collagen production increase healing
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| Proliferation/fibroblastic phase | show 🗑
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| show | US-helps increase fibroblastic activity
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| Proliferation phase(collagen) | show 🗑
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| show | NSAIDs Antibiotics debridement RICE proper positioning early motion-make sure that collagen fibers do not line up in a shortened position, early movement allows collagen fibers to properly align
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| Remodelling phase Goal | show 🗑
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| show | regeneration of new cells Reduction in fibroblast, macrophages Type III collagen coverting to type I reduction of capillaries
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| show | Controlled motion-motion of the injured tissue will influence thier strucures when they heal Modalities-STM, ultrasound, lazer Exercise
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