6-19-10 Patho II CAD
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| What percent of all deaths in western society are caused by CAD | show 🗑
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| How do you know if your old | show 🗑
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| show | 225ml/min (8ml of o2/100g of tissue/min)
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| What percent of cardiac output becomes coronary blood flow | show 🗑
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| show | 4 times
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| How many ml of O2 is required to maintain cell life | show 🗑
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| show | the local metabolism
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| show | O2 demand
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| show | increase HR, strength, and vasoconstriction
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| What does PNS (parasymp) stimulation do to the heart | show 🗑
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| 70% of the metabolism of the cardiac muscle uses what as its energy source | show 🗑
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| show | Acute occlusion or Cardiac Fibrillation
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| show | Atherosclerosis
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| Atherosclerosis starts at what age | show 🗑
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| Atherosclerosis causes - besides lipid accumulation, what are 3 other causes | show 🗑
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| show | Connective tissue matrix (collagen and elastic fibers
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| Name 3 independant predictors for the development of athrosclerosis | show 🗑
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| What Theory is this: some form of injury in the endothelium will trigger the events by changing the fx activity of the endothelium which combined with incr. cholest, smooth muscle prolif. and platlet activity lead to the atherosclerosis plaque | show 🗑
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| What theory is this: each lession of athrosclerosis is originated from a single smooth muscle cell that serves as a source of all of the cells within a lesion - each lesion is a benign neoplasm - caused by viruses, chemicals, or mutants | show 🗑
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| ??? is when coronary demand is greater than supply | show 🗑
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| show | Hypoxia
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| Zero O2 supply with adeqate perfusion is called | show 🗑
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| Ischemia is the result when what 3 things decrease | show 🗑
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| show | Ischemia (not necrosis, no pain with necrosis)
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| show | hot, pressing, and constricting
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| Per slide 13, which of the following 2 are not related to myocardial ischemia pain: [Throat, neck jaw][shoulder][retrosternal][epigastric][right chest pain][left chest pain][back pain] | show 🗑
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| show | Give Fernandez's cell phone a ring and get the scoop (IN BOLD LETTERS, NO!!!!)
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| What are the classes of angina (3 of them) | show 🗑
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| Angina that starts gradually and gets worse over minutes than dissipates after trigger is gone is classified as ? angina | show 🗑
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| show | Increase O2 demand over supply (physical activity, emotions, eating, Mary Beth, hurrying, fever, chills, tachycardia, motion of hands over head)
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| In chronic or stable angina there is a "fixed coronary occlusion" which means | show 🗑
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| show | False, it results from increased demand with fixed supply
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| show | True
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| Other diagnosis that could mimic angina (MI)- (6 of them) | show 🗑
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| Chronic angina findings include (3 things) | show 🗑
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| show | Stres thalium 201 Myocardial perfusion imaging - it shows defects
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| show | MI
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| show | Coronary arteriography with left ventricular arteriography
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| show | Lifestyle changes, drug tx, revascularization
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| show | PTCA, CABG, Laser angioplasty, Coronary atherectomy
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| What is the difference between Acute (unstable) Angina and Chronic (Stable) Angina | show 🗑
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| show | False (that is the definition of Chronic/Stable angina). A true statement would be: fixed O2 demand with decreased O2 supply
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| What are the clinical symptoms that differ between Acute and Chronic angina | show 🗑
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| What things (2 of them ) do not relieve symptoms in Acute angina but do in Chronic angina | show 🗑
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| show | In acute angina there is less collateral flow, higher number of vessles dz (more than one), increased incidence of coronary thrombi, faster progression of atherosclerosis, increased platlet aggregation, and thrombosis + coronary spasms
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| What are the different diagnosive test between Acute and Chronic angina | show 🗑
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| Management of Acute angina (8 of them) | show 🗑
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| show | Dead tissue (from prolonged ischemia)
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| show | The whole thickness of the ventricular wall
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| Sub-endocardial infarction includes | show 🗑
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| show | Acute coronary thrombosis
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| What are the variables with Acute coronary occlution that determine whether the damage will be transmural, subendocardial or no damage | show 🗑
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| Enzymes tested related to MI (4 of them) | show 🗑
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| show | Drugs (b-blockers, nitrates, analgesics, and O2), Controll of physical activity, Thrombolitics (intracoronary and intravenously), and PTCA (easier through fresh thrombus)
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| T/F: In a CABG, the harvested vessle can be from either a venous or arterial source | show 🗑
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| In a CABG, revascularization occurs by connecting the harvested vessle distal to the coronary blockage and into the ? or the ? | show 🗑
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| show | When the PTCA was not successful (or could not be performed), or multiple vessles need intervention
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| CABG is containdicated in what patients | show 🗑
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| How many hours after an AMI should a CABG be performed to be most successful. | show 🗑
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| show | Coronary artery spasm
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| T/F: Variant Angina is like Acute angina, it can occur at rest | show 🗑
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| show | False, Varient angina does is not related to physical activity or emotional stress; Chronic angina is
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| Does anything happen with the ST segment with Variant Angina | show 🗑
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| show | False, Variant angina pain does not get worse (Acute angina pain gets worse)
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| The key component to diagnosing Variant angina is | show 🗑
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| show | Ergonovine
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| What will the administration of Ergonovine cause in a patient with Variant angina | show 🗑
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| What is the best management treatment for patients with Variant angina | show 🗑
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| show | When the spasm is present with obstruction (PTCA and CABG are contraindicated in Variant angia without obstruction)
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Created by:
smorrissey1
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