Test 2 - Chapters 5,6,10
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| What morphological features is classic in the megaloblastic anemias? | macroovalocytes
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| A macrocytosis that is not megaloblastic in origin can be seen in all of the following: | Chemotherapy, Postsplenectomy, and reticulocytosis
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| A macrocytosis that is not megaloblastic in origin can be seen in all of the following except: | Hypoglycemic patients
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| Ineffective erythropoiesis is defined as: | Premature destruction of red cell precursors
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| Intrinsic factor is secreted by the: | Parietal cells of the stomach
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| One of the most common pathophysiologies that contribute to folic acid deficiency is: | Tropical sprue
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| The Schilling test continues to be an historic reference method for the determination of: | Pernicious anemia
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| Macrocytic red blood cells are routinely seen in all the following: | Pernicious anemia, liver disease, and response to anemic stress
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| Macrocytic red blood cells are routinely seen in all the following except: | Thalassemia
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| Thalassemia are characterized by: | Decreased rate of globin synthesis
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| When platelets concentrate at the edges and feathered end of a blood smear, it is usually due to: | poorly made wedge smear
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| Irregular clumping of platelets is usually due to: | inadequate mixing of blood and anticoagulant
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| The anemia of chronic infection is characterized by: | decreased serum iron levels
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| Factors commonly involved in producing anemia in patients with chronic renal disease include: | ineffective erythropoiesis
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| A pt has the following blood values: RBC 6.5 x 10^6/uL Hgb 14.0 g/uL Hct 42.0% MCV 65 fL MCH 21.5 pg MCHC 33% These results are compatible with: | thalassemia minor
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| What sets of laboratory findings is consistant with hemolytic anemia? | decreased erythrocyte survival; increased catabolism of heme
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| Evidence indicates that the genetic defect in alpha thalassemia usually results in: | a quantitative deficiency in RNA resulting in decreased globin chain production
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| Hemoglobin H disease results from: | absence of 3 of 4 alpha genes
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| What represents characteristic features of iron metabolism in patients with anemia of a chronic disorder? | serum iron is decreased, transferrin saturation is decreased, TIBC is normal or decreased
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| What is most closely associated with idiopathic hemochromatosis? | iron overload in tissue
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| What is seen most often in thalassemia? | target cells
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| What is most likely to be seen in lead poisoning? | basophilic stippling
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| A 17-year-old boy is admitted to the hospital with a fever of unknown origin. His WBC count is 20.0 x 10^9/L. All of the following can be seen on his peripheral smear: | toxic granulation, increased band neutrophils, and dohle bodies
Can NOT be seen: reactive monocytes
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| A typical blood picture in infectious mononucleosis is an absolute: | Lymphocytosis without anemia and many reactive lymphocytes
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| A patient who presents with low white cell count, thrombocytopenia, elevated liver enzymes, and mulberry-like inclusions in the granulocytes is most likely suffering from: | Human ehrlichiosis
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| What statement is common to most lipid storage disease? | They are caused by a missing metabolic enzyme
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| An autosomal disorder that manifests itself with large lysosomal inclusions, recurring infections, and albinism is: | Chediak-Higashi
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| What is an unusual complication that may occur in infectious mononucleosis? | Hemolytic anemia
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| Toxic vacuolization occurs most frequently in: | Neutrophils
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| Toxic granulation in neutrophils is a direct result of: | Enhanced lysosome enzyme production
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| Pelger-Huet anomaly is characterized mainly by: | Hyposegmentation of the nucleus in the neutrophils
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| Dohle bodies consist of: | Ribosomal RNA
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| In which white cell disorder will you see prominent deposition of granules in every cell line? | Alder's anomaly
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| In which white cell disorder will you see gray-green cytoplasmic granules in the neutrophils? | Chediak-Higashi sydrome
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| All of the following are lipid storage syndromes/disease: | Gaucher's disease, Tay-Sachs disease, Niemann-Pick disease
NOT: Chediak-Higashi syndrome
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| What shows the three stages of iron defiency anemia in order form first to third? | storage, transport, functional
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| What is not a cause of iron deficiency anemia? | chronic infection
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| In anemia of chronic inflammation, which is increased? | Storage iron
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| In sideroblastic anemia, where is there a decreased amount of iron? | Hemoglobin
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| Lead poisoning is associated with: | Sideroblastic anemia
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| What is the cause of primary iron overload? | defective genes
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| Iron overload damages all of the following cells: | liver tissue, white blood cells, pancreas tissue
Not damaged: red blood cells
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| What is the common treatment for patients with hereditary hemochromatosis? | phlebotomy
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| What is the common treatment for patients with transfusion-induced hemochromatosis? | chelation
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| What can cause a B12 deficiency? | pernicious anemia
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| Lab finding common for megaloblastic anemia include all of the following: | pancytopenia, increased MCV, and nuclear-cytoplasmic asynchrony
Not included: schistocytes
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| Long term megaloblastic anemia leads to what change in all the blood cell counts? | Decreased
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| Megaloblastic anemias result in cells that are: | macrocytic, normochromic
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| In a megaloblastic anemia, what result would be expected for a neutrophil lobe count? | increased
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| In a megaloblastic anemia, what result would be expected for a bilirubin test? | increased
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| In a megaloblastic anemia, what result would be expected for a lactate dehydrogenase? | Increased
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| A bone marrow aspiration/examination is not necessary for patients with megaloblastic anemia. | True
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| Hypersegmented neutrophils | megaloblastic anemia--specifically: pernicious anemia
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Created by:
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