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OMM cardio/pulm

OMM cardio pulm chapman OMS2 Exam 1

QuestionAnswer
Chapman myocardial reflex, anterior 2nd intercostal space at sternal border (B/L)
Chapman myocardial reflex, posterior T2 and T3, between spinous and transverse process (B/L)
Chapman adrenal reflex, anterior Lateral aspect of rectus abdominus, at inferior costal margin (B/L)
Chapman adrenal reflex, posterior T11 and T12, between spinous and transverse process (B/L)
Scoliosis at ____ degrees compromises CV funvtion 60 to 75
Kyphosis at ____ degrees compromisises CV function 60 or more
Flattening of thoracic kyphosis or postural crossovers results in _____ Tachyarrythmia
Triggerpoint in _____ muscle initiates/perpetuates _______ R. Pec major, supraventricular tachyarrhythmia
Sympathetic innervation of the heart, levels T1 to T6, L side dominant
SA node sympathetic innervation; disfunction causes R sided T1 to T6, supraventricular tachy
AV node sympathetic fibers; dysfunction causes L sided T1 to T6, ectopic foci, V-fib
Sympathetic supply to organ controls Function and vascular tone
Symp. supply to arm T2 to T8
Symp. supply to leg T11 to L2
Parasympathetic fibers consist of Vagus nerve + C1 and C2 roots
Vagus stimulation to the heart ____ rate. It is most affected by ____ branches Slows, pulmonary
Reflex slowing of heart can be acheived by pressure on the 1)___ , 2)____ and 3)___ Carotid body, globe of the eye, laryngeal irritation
Deep pressure over this bone can slow the heart Occipitomastoid suture
Parasympathetic effect on peripheral vasculature Vasoconstriction
Parasympathetic special effect on vasculature Dilation in submaxillary, parotid, blush region, tongue, penis and clitorus
Heart lymph drains to Right lymphatic duct
Sympathetic effect on lymph flow constricts the duct, reducing flow
Post MI OMM; focus on OA release, C1, C2, thoracic inlet, rib raising, T1 to T6, lymphatics (lower sympathetic tone)
Post MI, use soft tissue for 72 hrs
HTN is SBP >140 and DBP >90 on 2 occasion
Primary essential is ___% of total HTN 85
HTN caused by (4 things) 1) hypersensitivity to sympathetic stim 2) prolonged symp. to kidneys -> retention 3) vasoconstriction -> increased cardiac output 4) baroreceptors reset to high value
HTN treated by Sympathetic bed manipulation; T1 to L2
In CHF, lymphatic flow is Increased 3 to 40 times
In CHF, treatment directed to Lymphatics
What to look out for before applying lymphatic pumps? That CV system can handle increased fluid load
Diameter of thoracic duct is under Sympathetic control
Anterior MI, changes in T2, T3
Posterior MI, changes in T3-T5
Acute MI treatment (what not to do; position) NO DIRECT, pumps are too vigorous, semi recumbent
Chronic MI treatment Upper thoracic, diaphragm, OA/C2, fascia and thoracic inlet
Gait abnormality can increase cardiac work by 300%
HTN dysfunction pattern levels C6, T2, T6
Pectoralis triggerpoint eponym Travells
Facilitation is driven by Nociception
Faciliation is a ___, ___ threshold loop, lowers
Due to facilitation, normal stimuli can be pain, pain can drive sympathatic output
Heart suregery affects the __ sided ribs Left
Rib spreaders moves ribs into ___ dysfunction Inhalation
Surgeons spread ___ ribs most Lower sternal
Rib heads are ___ into the vertebral bodies Compressed
Posterior cervical chapman points C1 Flex, C3 Flex and STRAW. All others, extend and SARA
Anterior cervical tenderpoints C1 rotate away, C7 Flex and STRAW. All others, flex and SARA
Anterior T1 tenderpoint Midline episternal notch
Anterior T2 tenderpoint Midline Angle of Louis
Anterior T3 to T5 tenderpoint Respective ribs,
Anterior T6 tenderpint Xiphisternal junction
Anterior T7 tenderpoint Tip of xiphoid
Anterior T8 tenderpoint 3 cm below xiphoid at level of T12
Anterior T9 tenderpoint 1–2 cm above umbilicus at level of L2
Anterior T10 tenderpoint 1–2 cm below umbilicus at level of L4
Anterior T11 tenderpoint 5–6 cm below umbilicus below level of iliac crests at superior L5 level
Anterior T12 tenderpoint Superior, inner surface of iliac crest at mid-axillary line
Describe Chapman points Small, smooth, firm, subcutaneous, 2-3 mm
Treatment of Chapman points 15 seconds of rotary force; up to 2 minutes
Still Technique Indirect, then direct
Thoracic khyposis increases during _ and decreases during _ exhalation, inhalation
Respiratory rate is manipulated by Chemo and baro receptors; aortic and carotid bodies + respiratory center in medulla
Scoliosis and khyposis _ the lung space Decrease
1st rib ME muscles Anterior and Middle scalene
2nd rib ME muscle Posterior Scalene
Rib 3-5 ME muscles Pec Major
Rib 6-8(9) ME muscles Serratus Anterior
Rib 9-12 ME muscles Lat Dorsi
Rib 12 ME muscle Quadratus Lumborum
Pulmonary Sympathetics T1-T6
Pulmonary Parasympathetics CNX, OA, C1/C2
Pulmonary Lymphatics R lymphatic duct
Diaphragmatic flattening causes Reduced movement [of lymph]
Anterior Chapman point, bronchial 2nd inter space, parasternal
Anterior Chapman point, upper lung 3rd inter space, parasternal
Anterior Chapman point, lower lung 4th inter space, parasternal
Anterior Chapman point is for Diagnosis
Posterior Chapman point is for Treatment
Ciliated epithelium does what remove particulates
Musculoskeletal pulmonary tx rib motion, myofascial, abdominal diaphragm
Sympathetic innervation to the lungs, trachea, bronchi and visceral pleura T1 to T6 ( T2 to T7)
Sympathetic innervation to the parietal pleura T1 to T11
Sympathetic stimulation _ goblet cells and _ ciliated cells increase goblet, decrease ciliated
Sympathetic stimulation _ secretion inhibits, decreased motility
Posterior Chapman point, bronchial between spinous and transverse process, T2
Posterior Chapman point, upper lung between spinous and transverse process T3 and T4
Posterior Chapman point, lower lung between spinous and transverse process, T4 and T5
Carotid body innervation and function BP, CO2, O2 regulation, CNIX and CNX
Stretch receptor transmit via the _ nerve Vagus
Stretch receptor are excitatory or inhibitory inhibitory
Carotid body causes _ respiratory rate increased
Hering Breuer reflex to congestion decreased excursion (due to fluid stretch) + increased breathing (due to fluid affecting O2) result in shallow rapid breathing
Treating bronchospasm, 1st treat the_ because parasympathetics, prevent acute bronchospasm
Diaphragm’s greatest excursion is in _ position supine
Diaphragm attachments (3) xiphoid, ribs 6-12, T12 to L3
Brachial plexus emerges between anterior and middle scalenes
Normal quiet resp requires _ body work 2-3%
Exercise resp requires _ total body work 3-4%
Asthma, pulmonary dx requires _ total body work 33% or more
Intercostal neuralgia w/out structural dsfx means organic pathos
Inhalation dsfx key rib is bottom (holds up)
Exhalation dsfx key rib is top (pins down)
Inhalation dsfx causes spine to extend
Exhalation dsfx causes spine to flex
Rib 1 axis horizontal
Rib 7 axis 45 degrees below horizontal
Thoracic inlet is made of (4) manubrium, clavicles, ribs, T1
Asthma reflex is T2 rotated left
Bronchial mucosa, typical somatic dsfx T2 and 3, rotated right
How to prevent pneumonia, pre and post op pre C3 to C5, post C3 to C5 AND ribs raising
COPD diaphragm shape is flattened
Level of sternal notch T2
Level and rib for angle of Louis Rib2, T4
Rib for spine of scapula rib 3
Rib for xiphoid and inferior angle of scapula rib 7
Counterstrain technique position of ease for 90 seconds
HVLA on thoracic, hand placed _ dysfunctional segment below
HVLA thoracic, thrust is directed 45 degrees cephelad
When doing thoracic muscle energy, which hand should be placed behind the head? The rotated side
When doing thoracic muscle energy, where should the DO's hand be placed in type I mechanics and in type II mechanics Under in I; Over in II
When doing indirect respiratory cooperation for the thoracic and lumbar spine, the doc should be sitting on the _ side of the curvature Covexity
Created by: asindhidude