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Mid-Term Physiology

Mid-Term Physiology; Thibodeau, K.P. G. (2018). Anthony's Textbook of Anatomy &

TermDefinition
Homeostasis Correct temperature, pressure, chemical composition
afferent towards
efferent away
Feedkback loop 1) sensor mechanism -senses something is off sends signal to control center 2)integator or control center -recieves input; part of brain; sends efferent signal if change needed 3)effector mechanism -organs influence varibles 4) feedback
Negative feedback oppose change by creating a response that is opposite ex-shivering
Positive feedback -often harmful -amplify change -NOT homostatic
Set point where body level should be at;
Feedforward info flows forward to other processes in anticipation of an event
Intracellular control mechanisms regulate at a cell level
Intrinsic control regulate at tissue or organ level
Extrinsic control operates at system or organism level -nervous and endocrine systems
Functional groups arrangments of atoms attached to carbon core of organic molecules; radicals or R
Free radical unattached functional group; highly reactive
Carbohydrates C, H, O 1:2:1 -sugars and starches -primary source of chemical energy
Monosaccharides -small, simple sugars -MOST IMPORTANT GLUCOSE -ex fructose, galactose, pentose (rna, dna)
Disaccharides -double sugar -bonded by dehydration reaction (removal of water)
Polysaccharides -complex sugars -bonded by dehydration reaction (removal of water) ex- sucrose, maltose, and lactose
Polymer large molecule made of identical smaller molecules ex- glycogen --animal starch
Lipids water-insoluble organic biomolecule -NON-polar -C, H, O less O than carbs -energy, structure, cell membranes
Triglycerides or FATS -most abundant -most concentrated energy -made of glycerol and fatty acids
Saturated fatty acid all bonds are filled with H; no double bonds
Monosaturated fatty acids ONLY 1 double carbon bond
Polysaturated fatty acids More than 1 double carbon bond
Unsaturated fatty acids 1 or more double bonds; not all are filled with H
Phospholipids -P & N head hydrophilic tail hydrophobic
Steroids ex- cholestrol- plamsa membrane around every body cell
Prostaglandins (PG) -tissue hormones -formed and released in response to a stimulus- -regulate hormones, BP, digestive juices, immune system, blood clotting
Proteins C, H, O, N -structure and function (enzymes)
Amino acids -peptide bond, carboxyl + amino acid group
Primary protein structure amino acids in a chain
Secondary protein structure folding and twisting of amino acids
Terriary protein structure 2nd level twists and folds to form 3D
Quaternary protein structure more than one foldled amino acid chain
Membrane potenical positive ions outside the membrane; negative inside
Resting potenical no electrical signals -70mV (resting membrane potenical (RMP)
Stimulus-gated channels ion channels that open by sensory or chemical stimulus
Ligand- gated channels open or close by stimulation of a correct ligand to recceptor -only happen in small locations of the membrane
Depolarization movement of membrane potenical towards zero
Hyperpolarization membrane potenical away from zero
Action potenical steps 1) stimulus triggers Na+ channels to open (depolarize) 2) threshold is reached; voltage gated Na+ channels open 3) more Na+ enters increased depolarization 4)action potencial peaks; voltage gated Na+ closes
Repolarization steps after action potencial 1) voltage gated K+ channels open 2) brief hyperpolarization 3) resting potenical restored by channels returning to OG state
Refractory period brief time local area of axon's membrane resists restimulation
Saltory conduction action potenical flows from node to node; "leaping"
Synapse signal from one neuron to another
Electrical synapse two cells are joined by gap junctions - continue signal without pause ex-cardiac muscles, smooth muscles
Chemical synapse use a chemical transmitter to send signals -synaptic knob, synaptic cleft, postsynaptic membrane
Synaptic knob bulb at end of presynaptic axon -lots of neurotransmitters
Synaptic cleft space between knob and plasma membrane of postsynaptic cleft
Postsynaptic membrane space on postsynaptic cleft with receptors to recieve neurotransmitters
Spatial summation when synaptic knobs have enough leftover neurotransmitters add together to create an action potenical
Temporal summation when synpaptic knobs stimulate a postsynatic neuron in rapid succession
Memory form when flow of info is facilitated by synapses
Short-term memory presynaptic facilitaiton inhibition at particular synapses
Intermediate long term memory presynaptic activity lasts longer by releasing serotonion to block K+ channel from rapidly repolararizing the membrane
Long-term memories change in structure of synapse; increased vesicles stored, increased membrane locations, increase of presynaptic axon terminals, increase in dendritees that allow postsynaptic facilitation
Ionotropic receptors directly change ion permeability when stimulated
Metabotropic receptors binds to receptor that sets a metabolic pathway that opens or closes ion channels -g proteins
Convergence many neuropathways funneled into one
Divergence one neuropathway split or copied
Created by: makayla.davis
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