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KIN 3600
Lec 11
| Question | Answer |
|---|---|
| Steady state exercise | -Continuous exercise for long period of time (40mins – 1hr) -Curvilinearly |
| O2 consumption | -ATP supply -Creatine Phosphate -Anaerobic glycolysis ^ for supplying energy for ATP |
| 98% Exclusive through Aerobic Phosphorylation | No more reliance on anaerobic energy transformation system |
| O2 deficit | difference in O2 demand & O2 supply during exercise bout |
| Energy Equivale Oa (EEOa) | 1L O2 = 5kcal energy (aerobic) |
| Incremental Phase | Curvature always goes up -Hydrolysis of ATP & CP |
| Anaerobic glycogenolysis | there may be some accumulation of lactate (HLA) |
| Small contribution of energy by the aerobic phosphorylation through utilization of stored oxygen | -O2 in capillary blood and interstitial fluid -O2 present in sarcoplasm in combination with myoglobin -O2 present in mitochondria |
| O2 deficit | Oxygen equivalent of energy supplied anaerobic metabolic system plus extra oxygen extracted from body’s oxygen stores |
| OXYGEN IS ENERGY | ENERGY IS OXYGEN |
| Factors O2 deficit | -O2 Equivalent energy supplied by ATP/CP system. -O2 Equivalent energy supplied by anaerobic glycogenolytic -Extra O2 extracted from storage: Myoglobin, Capillary blood, Interstadial fluid, Present in mitochondria |
| Why doesn’t O2 consumption increase instantaneously to level required in exercise? | System is slow to accelerate -Aerobic Phosphorylation |
| Why is the system slow? | Enzyme are required -> Pos & Neg Modifications |
| Time required to ^increase concentration of Krebs Cycle intermediaries | -2nd slowest -Depends on intensity & duration 5-15-fold |
| Q10 effect | Time required to ^increase body temperature |
| Aerobic energy transformation system has “high inertia” – it is a sluggish system | -3 mins to reach O2 level required. -Single muscle fiber |
| Plateau Phase | -Energy demand of exercise is completely met by aerobic phosphorylation OR -All ATP required for the exercise is resynthesized by aerobic phosphorylation -O2 demand = O2 supply |
| What happens if we stop rapidly? | -Excess O2 - ^ slow = v slow |
| For any given absolute submaximal powerout (watts) of steady state exercise | rate of O2 consumption is the same regardless of fitness, health, gender, age, etc. |
| Recovery Phase | “Oxygen debt” |
| Recovery Phase factors | -2 required to resynthesize ATP & CP from ADP & Pi -O2 required to replenish O2 storage of the body -O2 required for the resynthesize of glycogen from lactic acid in liver (gluconeogenesis) |
| Recovery Phase extra oxygen | -consumed by heart -consumed by respiratory muscles -consumed following exercise due to higher body temp (Q10 effect) - consumed due to hormonal stimulation-->Hormones make cell membranes leaky -not exclusively used to pay back the energy “borrowed” |
| EPOC | Excess Post-Exercise Oxygen Consumption |
| Oxygen kinetics during low to moderate intensity & high intensity submaximal steady state exercise | Higher intensity of the exercise the higher the oxygen deficit & EPOC |
| O2 deficit & EPOC larger in sprints or marathon? | - Sprinting - Marathon: not much EPOC - 100m dash: largest O2 deficit |
| EPOC depends on body temperature | Largest component of EPOC is ^ body temperature because it takes a long time to bring temperature back to normal |
| Difference in oxygen deficit between aerobically fit & unfit individuals | ^ fitness = v EPOC & O2 deficit Due to faster response of aerobic E.T. v fitness = ^ EPOC & O2 deficit |
| Why is Aerobic E.T. becomes less sluggish? | - ^increase activity of rate limiting enzymes - ^increase TCS intermediaries = ^ fast Krebs = v EPOC |
| ^ increase fitness level | v decrease O2 deficit & EPOC |
Created by:
rmart11
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