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KIN 3600

Lec 11

Steady state exercise -Continuous exercise for long period of time (40mins – 1hr) -Curvilinearly
O2 consumption -ATP supply -Creatine Phosphate -Anaerobic glycolysis ^ for supplying energy for ATP
98% Exclusive through Aerobic Phosphorylation No more reliance on anaerobic energy transformation system
O2 deficit difference in O2 demand & O2 supply during exercise bout
Energy Equivale Oa (EEOa) 1L O2 = 5kcal energy (aerobic)
Incremental Phase Curvature always goes up -Hydrolysis of ATP & CP
Anaerobic glycogenolysis there may be some accumulation of lactate (HLA)
Small contribution of energy by the aerobic phosphorylation through utilization of stored oxygen -O2 in capillary blood and interstitial fluid -O2 present in sarcoplasm in combination with myoglobin -O2 present in mitochondria
O2 deficit Oxygen equivalent of energy supplied anaerobic metabolic system plus extra oxygen extracted from body’s oxygen stores
Factors O2 deficit -O2 Equivalent energy supplied by ATP/CP system. -O2 Equivalent energy supplied by anaerobic glycogenolytic -Extra O2 extracted from storage: Myoglobin, Capillary blood, Interstadial fluid, Present in mitochondria
Why doesn’t O2 consumption increase instantaneously to level required in exercise? System is slow to accelerate -Aerobic Phosphorylation
Why is the system slow? Enzyme are required -> Pos & Neg Modifications
Time required to ^increase concentration of Krebs Cycle intermediaries -2nd slowest -Depends on intensity & duration 5-15-fold
Q10 effect Time required to ^increase body temperature
Aerobic energy transformation system has “high inertia” – it is a sluggish system -3 mins to reach O2 level required. -Single muscle fiber
Plateau Phase -Energy demand of exercise is completely met by aerobic phosphorylation OR -All ATP required for the exercise is resynthesized by aerobic phosphorylation -O2 demand = O2 supply
What happens if we stop rapidly? -Excess O2 - ^ slow = v slow
For any given absolute submaximal powerout (watts) of steady state exercise rate of O2 consumption is the same regardless of fitness, health, gender, age, etc.
Recovery Phase “Oxygen debt”
Recovery Phase factors -2 required to resynthesize ATP & CP from ADP & Pi -O2 required to replenish O2 storage of the body -O2 required for the resynthesize of glycogen from lactic acid in liver (gluconeogenesis)
Recovery Phase extra oxygen -consumed by heart -consumed by respiratory muscles -consumed following exercise due to higher body temp (Q10 effect) - consumed due to hormonal stimulation-->Hormones make cell membranes leaky -not exclusively used to pay back the energy “borrowed”
EPOC Excess Post-Exercise Oxygen Consumption
Oxygen kinetics during low to moderate intensity & high intensity submaximal steady state exercise Higher intensity of the exercise the higher the oxygen deficit & EPOC
O2 deficit & EPOC larger in sprints or marathon? - Sprinting - Marathon: not much EPOC - 100m dash: largest O2 deficit
EPOC depends on body temperature Largest component of EPOC is ^ body temperature because it takes a long time to bring temperature back to normal
Difference in oxygen deficit between aerobically fit & unfit individuals ^ fitness = v EPOC & O2 deficit Due to faster response of aerobic E.T. v fitness = ^ EPOC & O2 deficit
Why is Aerobic E.T. becomes less sluggish? - ^increase activity of rate limiting enzymes - ^increase TCS intermediaries = ^ fast Krebs = v EPOC
^ increase fitness level v decrease O2 deficit & EPOC
Created by: rmart11