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Stack #187900

Townes and Holtfreter experiment, and what it implies like cells from different embryos aggregate, adhere together. implies cell recognition, cell adhesion
__ folding of peptide in transmembrane domain alpha helix
glycocalyx responsible for cell recognition (ABO blood types), cell adhesion
types of CAM (cell adhesion molec) 1.cadherin - homophilic binding 2.integrin - heterophilic binding
how to dissociate cell remove Ca2_, or add trypsin
spatiotemporal distribution lose CAM to migrate, re-express CAM to group into ganglia
cell junctions occluding (tight junction), anchoring , gap
types of anchoring junction 1. adherens - cell-cell or cell-matrix E-cadherin @ catenin attached to F-actin 2. desmosome - cell to cell catenin attached to non contractile IF. 3. hemidesmosome - cell to basal lamina
1 gap junction is 2 connexon, (6 * 2 connexin)
facilated diffusion passive or active passive
difference between facilitated diffusion and simple diffusion through channels both passive, but facilitated diffusion has a binding site
what diffusions are carrier mediated facilitated diff, primary, secondary active
effect of insulin on glucose transpoter increase number of Glut4
Glut 4 is an example of what kind of transport facilitated diffusion
facilitated diffusion move molec against/down conc't grad down
co-transport also called symport
counter-transport also called antiport
example of co-trasport in kidney gluc and Na+ moving into cell from lumen of nephron
example of counter-trasport in kidney Na+ move into cell and H+ out to lumen of nephron
fluid endocytosis called pinocytosis
during endocytosis phagosome fuse with lysosome
during pinocytosis pinocytic vesicle fuses with endosome
pseudopod formed during phago or pinocytosis during phagocytosis
rheumatoid arthritis autoimmune disorder causes release of lysosomal content into joints
lysosomal storage disorder tay sach, missing lipase in lysosome => accum of FA in brain
receptor mediated endocytosis (special pinocytosis) 1.clathrin bdd to vesicle 2.clathrin removed 3.vesicle bdd to endosome
what kind of molec taken in by receptor mediated endocytosis LDL
autocrine cell affect itself
paracrine cell affects target cell
where does RNA polymerase bind to promotor region
exons are expressed genes
introns are garbage ie not expressed
familial hypercholesterolemia LDL receptor defective, high LDL in blood causing atherosclerosis
intracellular protein made in free ribosome
protein destined for secretion made in RER
protein destined for integration into membrane made in RER
co-translational modification translation starts in free ribosome. signal sequence binds SRP(signal recognition particle) which takes the complex to ribosome on RER, where translation is continued.
how does steroid hormone (lipid soluble ligand) bring about intracellular response? steriod diffuse accross PM, and bdd to intracellular receptor. the receptor-ligand complex moves into the nucleus, where it will act as a transcription factor.
subunit of MF actin
subunit of MT alpha, beta tubulin
what is the strongest cytoskeletal filament microtubule
what forms a hollow filament MT
IF forms nuclear lamina
MT radiates from centromere
dynein moves tward __ this is called -ve, reterograde
kinesin moves toward __ this is called +ve, anterograde
what acts as a track for dynein, kinesin MT
endothelial cells of nephron held together by tight junction
integrin heterophilic CAM
cadherin homophilic CAM
Created by: honghee



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