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WVSOM -- Physio

Integrated CV Control

Medullary CV center Vasomotor (ventrolateral medulla) Cardioacceleratory Cardioinhibitory
Blood Pressure Control Baroreflex
High pressure receptors Carotid sinus Aortic Arch
Low Pressure Receptors Cardiopulmonary receptors pulmonary artery Junctino of atria w/ corresponding veins
Response to orthostatic pooling of blood Decreased Stretch -> Increased SNS Decreased PNS
SNS on B1 in orthostatic hypotension increased HR Increased contractility Increased SV
SNS on arterioles Consticts Increased Peripheral Resistance
SNS on Veins Increased Vascular Resistance Increased Stroke Volume Increased Cardiac Output
SNS on Kidneys Increased Na Increased H2O Increased BV increased VR Increased CO
Baroreflex to increased arterial Pressure NTS decreases SNS and Increases PNS resulting in decreased HR
Increased Stretch Leads to increased HR VD to kidnes Decrease in ADH Myocytes release ANP
Decreased stretch leads to no effect on HR Increased SNS to kidneys Increased release of ADH, AVP
Stretch of high-pressure receptors Due to increased BP reflex works to decrease BP
Stretch of Low-pressure receptors increased volume relex works to dump fluid
Hormonal Control Epinephrine AVP ADH ANP
AVP or ADP Vasopressin Anti-diuretic Hormone Synthesized in the hypothalamus and released from pituitary
Stimulus for release of vasopressin Increased plasma osmolality Decreased volume Circulating Ang II
AVP Actions vasoconstrict retention of fluid by kidney to increase volume
RAAS Renin-Angiotensin-Aldosterone-System
Angiotension I -> Angiotension II by enzyme ACE
Angiotensin II results in Increased Aldosterone Decreased Na Excretion Increased Systemic Vascular Resistance
Role of chemoreceptors in AP regulation Important role in severe hypoxia Decreased pO2, Increased pCO2, and Decreased pH Central Sense: elevated CO2 leading to decreased pH
Created by: tjamrose



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