Major clinical syndromes of the MCA, ACA, and PCA territories
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| Left ACA | Right leg weakness of the UMN type and right leg cortical-type sensory loss. Grasp reflex, frontal lobe behavioral abnormalities, and transcortical aphasia can also be seen. Larger infarcts may cause right hemiplegia.
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| Right ACA | Left leg weakness of the UMN type and left leg cortical-type sensory loss. Grasp reflex, frontal lobe behavioral abnormalities, and transcortical aphasia can also be seen. Larger infarcts may cause left hemiplegia.
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| Left PCA | Right homonymous hemianopia. Extension to the splenium of the corpus callosum can cause alexia without agraphia. Larger infarcts including the thalamus and internal capsule may cause aphasia, right hemisensory loss and right hemiparesis.
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| Right PCA | Left homonymous hemianopia. Larger infarcts including the thalamus and internal capsule may cause left hemisensory loss and left hemiparesis.
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| Left MCA stem | Combo of the above (sup/inf/deep), w/R hemiplegia, R hemianesthesia, R homonymous hemianopia, & global aphasia. Often a left gaze preference, esp at the onset, caused by damage to the left hemisphere cortical areas important for driving the eyes to the R.
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| Right MCA stem | Combo of the above (sup/inf/deep), w/L hemiplegia, L hemianesthesia, L homonymous hemianopia, & profound L hemineglect. Often a R gaze preference, esp at the onset, caused by damage to the R hemisphere cortical areas important for driving eyes to the L.
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