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neuroanamtomy, dsyarthrias, respiration, phonation

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Answer
Major anatomical levels of the brain   Supratentorial, posterior Fossa (infratentorial), spinal, peripheral  
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Supratentorial Boundaries   above the tentorium cerebelli membrane upper border of posterior fossa, cerebellum seperates anterior, middle fossae and posterior fossa  
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Supratentorial includes   cerebral hemisphere (frontal, temporal, parietal, occipital lobe pairs basal ganglia, thalamus, hypothalamus Cranial nerve I (olfactory) and II (optic)  
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supratentorial possible motor speech disorders   AOS, Dysarthrias - spastic, unilateral UMN, hypokinetic, hyperkinetic  
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Posterior Fossa Level Boundaries   infratentorial, below tentorium cerebelli  
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Posterior Fossa includes   brainstem - pons, medulla, and midbrain cerebellum origins of Cranial nerves III - XII  
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Posterior Fossa Motor speech disorders   spastic, unilteral UMN, hyperkinetic, ataxic, flaccid dysarthria  
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Spinal level boundaries   foramen magnum  
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spinal level includes   vertebral column - 7 cervical, 12 thoracic, 5 lumbar 31 pairs spinal nerves - dorsal/posterior roots = sensory - ventral/anterior roots = motor  
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peripheral nerves includes   spinal nerve and cranial nerves serving speech  
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speech production occurs from complex interaction of   cognitive, linguistic and motor processes  
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MSD are speech disorders resulting from   impairments in planning and execution of neuromotor control  
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dysarthria is marked by   impaired execution resulting from abnormalities in strength, steadines, tone, or accuracy of movements required for control of the respiratory, phonatory, resonatory, articulatory and prosodic aspects of speech production  
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Pathophysiologic disturbances are due to CNS/PNS abnormalities and reflect   weakness, spasticity, incoordination, involuntary movements; or excessive, reduced, or vairables muscle tone of the speech musculature  
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need to take speech production that we hear and evaluate the subsystems   (respiration, phonation, resonance, articulation, prosody) for strength, range, steadiness, tone, and accuracy  
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Apraxia of Speech   neurologic speech disorder caused by impaired planning/programming sensorimotor commands needed to direct movements of phonetically and prosodically standard speech  
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ideation   begin with thoughts, feeling and emotions  
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linguistic processes   retrieve words from storage and perform phonologic encoding and assemble syntactic frame  
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motor planning   intended message is organized for nueromuscular execution  
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neuromuscular execution   execution involves direct activation of motor neurons, muscle contraction, and movement. CNS & PNS combine to regulate and execute all of the processes by which motor plan results in  
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neuromotor signal includes   CNS, PNS, basal ganglia, propriceptive and tactile sensory, descendign motor tracts and cerebellar control circuits  
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prevelance of acquired communication disorders that fall in neurological domain   41%  
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Comprehension involves   intelligibility - speech signal only comprehensbility - info independent of the speech signal, context communication conditions - complexity of message both conceptually and linguistically and adversity of listening conditions  
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Clinical Process of speech disorders   1. identify the perspectives 2. Identify treatment candidates 3. setting treatment goals 4. Assessing treatment efficacy  
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World Health Organization - International Classification of Functioning includes   Body Structure Body Function Activity Participation Environmental factors  
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Body function integrity   physiological function of body systems (speech, language, cognitive and respiratory) Impairment - problems in body function (respiration, phonation, articulation, velopahryngeal function)  
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Body structure integrity   anatomical parts of body (tongue, lips teeth) impairments - problems in body structure (cleft palate, flaccid tongue, flaccid muscles)  
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Activity   execution of task or action (learning, applying knowledge, communication) activity limitation - difficulty executing activities  
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Participation   involvement in life situations, ability and desire to participate in real life situation  
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environmental factors   physical, social, attidunal environmental factors (living situation, rehab situation) environmental barrier - noise, distance, lighting, limited access, societal bias  
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Personnel Framework includes   finder/identifier facilitator general practice clinician specialist expert  
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Evidenced Based Practice levels   Authoritative - expert opinion, accept rationale Observational - case studies (single or series), qualitative research, structured behavioral observations Experimental - randomized controlled studies, studies with controls, single subject designs  
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Purpose of Clinical Evaluation   -detect or confirm suspected problem - establish differential diagnosis - classify with a specified disorder group - determine site of lesion/disease process - specify severity of involvement - establish prognosis -specify precise treament focus  
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purpose of clinical eval cont'd...   - -establish criteris for tx termination - measure change resulting from Tx, lack of Tx or exacerbation  
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clinical examination components   3 major processes - history of speech problem - physical examination - motor speech examination  
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Assess for differential diagnosis   detect problem classify with a specific disorder group (cognitive, language, motor planning, motor execution) document severity  
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Asses for dif. diagnosis cont'd...   is communication disorder consistent with medical diagnosis? characteristics, severity determine necessary referral  
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monitor impact of medical intervention   communication characteristics, severity, secondary symptoms  
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Assess for intervention Planning   Assesment of impairment, activity, participation Plan intervention strategy  
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Assessment of impairment   communication characteristics subsystem performance (respiratory, laryngeal, velopharyngeal, oral articulation)  
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Assessment of functional communication   Activity - speech only , clinical Participation - in contextualized social situations  
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Assessment of Activity - speech signal only   intelligibility speaking rate naturalness (prosody)  
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intelligibility   a measure of the inderstandability of the speech signal only  
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AIDS   Assesment of intelligiblity of dysarthric speech - standardized assessment tool to measure intelligibility  
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AIDS measures   intelligiibiltiy of single word intelligibility of sentences speaking rate (on sentence task) communication efficiency ratio intelligible word per minute/190WPM  
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Tally for Windows   Computerized measurement of habitual speaking rate. reading passages aloud  
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Pacer for Windows   standard passages and sentences for computerized test  
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typical adult speaking rates   paragraph out loud 160-180 wpm sentences 190 wpm conversation - highly variable  
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naturalness/prosody   the extent to which speech conforms to a listener's standards of rate rhythm intonation and stress patterning  
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impairment assessment - functional components approach   what aspects of the speech motor activity are impaired? how have weakness, slowness, incoordination or abnormal tone affected speech?  
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oral peripheral exam   assess structure assess non-speech movement assess speech (like) movement assess movement during speech  
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subsystem impairment   respiration phonation velopharyngeal function oral articulation  
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Standarized tests used   Iowa Oral Pressure Instrument Experimental Phonetic Intelligibility test Phoneme indentification test optical motion capture Electromagnetic Articulography  
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purposes of assessment   screening - detect or confrim problem differential diagnosis specify severity and prognosis plan treatment measure changethat occurs as a result of treament  
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CAS - Child Apraxia of Speech   speech disorder due to delays or deviances in those processes involved in planning and programming movement sequences for speech  
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dysarthria   disrupted or distorted oral communication due to paralysis, weakness, abnormal tone or incoordination of the muscles used in speech  
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dysarthria processes affected   phonation respirations resonance articulation prosody  
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movements may be affected such as   force, timing, endurance, direction and range of motion  
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dyskinesias   involuntary movements  
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sites of lesions include   bilateral cortical damage, cranial nerve involvement, spinal nerve involvement (respiration), basal ganglia and cerebellum  
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dysarthria characteristics   slurred speech imprecise articulation weak respiratory support and low volume incoordination of the respiratory system hypernasality involuntary dyskinesias of the oral facial muscles spasticity of flaccidity of the oral facial muscles  
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respiratory system   source of aerodynamic energy for speech  
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essential parameters of respiration   air pressure lung volume air flow repiratory shape  
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Tidal Volume - TV   total of resting inspiration and expiration  
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resting expiratory   at end of expiration during resting breathing  
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expiratory reserve volume - ERV   air that is left after bottom of expiration during tidal volume  
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inspiratory reserve volume - IRV   what remains after resting inhalation  
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Vital Capacity - VC   the total amount of air that can be exhaled following maximal inhalation TV+IRV+ERV  
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inspiratory checking   ability to produce low sunglottal air pressure at high lung volume levels  
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residual volume - RV   volume remaining after forced exhalation  
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TOtal Lung capacity - TLC   volume lungs can be expanded with greatest inspiration TV+IRV+ERV+RV=VC+RV  
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FUnctional Residual Capacity - FRC   ERV+RV  
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Inspiratory Capacity - IC   TV+IRV  
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Forced vital capacity FVC   amount of air that can be forcefully expelled from a fully inflated lung position  
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subglottal air pressure levels   4-8 cm H2O Conversational speech, 1 to 1 relationship btwn sunglottal air pressure and speech intensity  
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breath groups   10-20% of vital capacity across time average of 15 syallables per breath group 50cc of airflow per syllable (1/3rd of a mouthful of air)  
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breath pattern   Inspiratory :expiratory ratio for speech 1:6  
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overall goal for respiratory treatment   consisten, adequate subglottal pressure during speech produced with minimal fatigue and appropriate breath group lengths  
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vocal intensity   sound energy percieved as loudness  
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fumndamental frequency   rate of VF opening / closing perceived as pitch  
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vocal quality   regularity of VF vibratory cycle perceived as roughness, harshness, hoarseness  
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hypoadduction   weakness/reduced closure of vocal folds from inflammatory myopathies, muscular dystrophies, breathy, quiet, aperiodic, hoarse, nasal  
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hyperadduction   closure is too tight - strained, strangled, pressed, harsh, loud/soft/normal, HUntington's, Pseudobulbar Palsy, some brain injuries  
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phonatory instability   variations in frequency/intensity = tremors, rough, hoarse with pitch breaks and fry  
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mixed phonatory impairments   aspects of above = MS, ataxic (cerebellar), dyspohonia  
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phonatory coordination impairment   poor coordination of phonatory system with articulation results in lack of voiced-voiceless distinctions or aspiration anomalies  
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hypoadduction - myotonic muscular dystrophy (MMD)   aperiodic, hoarse, hypernasal  
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hypoaddcution - myasthenia gravis   hypernasal, stridor, "vocal weakiness"  
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hypoaddcution - peripheral nerve (recurrent laryngeal) damage   unilateral - hoarse, breathy voice, reduced loudness OR voice may be normal  
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hypoaddcution - preipheral nerve (superior laryngeal) damage   mild hoarseness, vocal fatigue  
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hypoadduction- Xth cranial nerve injury to LMN   laryngeal paralysis,  
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hypoadduction - brainstem stroke and Parkinson's disease   reduced loudness, monotone, hoarse, tremor  
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hypoadduction - closed TBI   hypophonia  
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hyperadduction   UMN lesions, quick hyperkinesia of Huntington's disease, dysphonia, adductor spasmodic dysphonia  
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Phonatory assessment   history, structural integrity perceptual quality voiced, voiceless, pitch, loudness acoustic - range of frequency physiologic - laryngeal function  
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range of frequency in men and women   men 110-150 Hz women 175-210 Hz 25-50dB  
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Phonatory assessment cont'd...   intelligibility - with/without background noise predictable/unpredictable changes in phonation - voice breaks, ptich shifts, monotone timing of laryngeal - articulatory function - voiced, voiceless, distinguish cognates, aspirated sounds?  
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evaluating reflexive phonation   crying, laughing, coughing, choking  
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flaccid dysarthria   vocal fold immobility, incomplete glottal closure, reduced palatal movement, reduced pharyngeal wall movement, breathy, diplophonic, reduced loudness, decreased pitch and range  
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which disorders present with flaccid dysarthria   myasthenia gravis, vascular disorders (brainstem CVA affecting CN nuclei), infections (polio, herpes, meningitis), demyelnating disease (Guillain-Barre), muscle disease (Muscular Dystrophy), degenerative disease (progressive bulbar palsy, ALS)  
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Flaccid dysarthria treatment   head turn, digital manipulation of thyroid artilage, speak at onset of exhalation, use of optimal breath groups portable amplification, vocal fold injection, pharmacologic  
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Spastic dysarthria   reduced inhalatory/exhalatory volume, hyperadduction of vocal folds, slow velopharyngeal movements, reduced speech and range of tongue, lip and jaw, reduced tongue strength, incomplete lingual articulatory contacts  
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spastic dysarthria - what do we hear?   harsh, strained quality, pitch breaks, monopitch, monoloudness, low pitch  
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which disorder present with spastic dysarthria   vascular (brainstem CVA), lacunar CVA (deep:basal ganglia, thalmus, brainstem, deep cerebral white matter), degenerative disease, TBI  
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behavior mod for spastic dysarthria   speak at onset of exhalation, speak on deep breath, increase ptich, breathy onset, optimize breathing group, relaxation or massage  
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ataxic dysarthria   irregular chest wall movement, voice tremor, slow lip, jaw and tongue movement  
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ataxic dysarthria - what do we hear?   harsh, monopitch and mono loudness  
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which disorder presents with ataxic dysarthria   degenerative diseases, vascualer disorders, tumors, trauma, toxic disorders, chronic alcoholism  
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hypokinetic dysarthria   difficulty altering automatic breathing patterns, bowed vocal folds, tremulousnessof arytenoid cartilages, reduced lip and jaw amplitude, lip rigidity  
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hypokinetic dysarthria - what do we hear?   monopitch and loudness, reduced loudness, breathy  
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which disorders present with hypokinetic dysarthria?   degenerative desease (Parkinson's disease), vascular (multiple or bilateral CVA), certain medications, trauma TBI, infections  
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hypokinetic dysarthria treatment   behavior mod - effortful closure technique, Lee Silverman voice treament, speak at onset of exhalation, high phonatory effort, optimize breath groups, surgical fixes  
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hyperkinetic dysarthria   abnormal involuntary movements, may be rapid or slow, maybe irregular or rythmic  
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hyperkinetic dysarthria - what do we hear?   strained quality, excessive loudness variation, reduced ptich and variability, dysphonis, adductor voice arrests, vocal tremor, contiuous or intermittent aphonia  
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hyperkinetic dysarthria presents in which disorders?   idiopathic, tardive dyskinesia, HUntington's chorea, MS, Tourette's syndrome  
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unilateral UMN dysarthria   mild to moderate dysphonia, in single hemisphere CVA (cortical or lucanar), usually mild  
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ataxic site of lesion   cerebellar circuit  
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flaccid site of lesion   lmn, one or more cranial nerves  
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hyperkinetic site of lesion   basal ganglia circuit  
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hypokinetic site of lesion   basal ganglis circuit  
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spastic site of lesion   umn (usually bilateral)  
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Articulation assessment   speaking mode speaking task speaking context cueing level  
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speaking mode   habitual, clear  
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speaking task   imitative, reading, spontaneous  
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speaking context   single word, carrier phrase, sentence, paragraph  
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cueing level   simultaneous imitation (choral), immediate imitation, delayed imitation, spontaneous, answer questions  
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