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PTA Cardio Pharm

Pharmacology for Cardio Disease Management

Alpha Adrenergic Antagonist Agents Reduce perohperal vascular tone by blocking alpha-1-adrenergic receptors; causes dilation and decrease in BP
ACE Inhibitor Agents Suppresses enzyme that converts angiotensin 1 into angiotensin 2; decreasing blood pressure and after load
Anticoagulant Agents Inhibit platelet aggregation and thrombus formation
Anti-hyperlipidemia Agents AKA Statins; lipid modifying agents; inhibit anzyme aciton in cholesterol synthesis, break down LDL, decrease triglycerides, and increase HDL
Anti-thrombotic Agents inhibit platelet aggregation and clot formation
Beta Blocker Agents Block B-Adrenergic receptors; decrease myocardial oxygen demand by decreasing heart rate and contractility
Calcium Channel Blocker Agents Decrease calcium entry int o calcium smooth mm cells resulting in diminished myocardial contraction, vasodilation, and oxygen demand
Diuretic Agents Increase secretion of sodium and urine => reduction in plasma volume and BP; thiazide, loop and potassium sparing
Nitrate Agents Decrease ischemia through smooth mm relaxation and dilation of peripheral vessels
Positive Inotropic Agents Increase force and velocity of myocardial contraction; slow the heart rate; decrease conduction velocity through AV node and decrease degree of activation of SNS
Thrombolytic Agents Facilitate clot dissolution through conversion of plasminogen to plasmin; plasmin breaks down clots and opens blood vessels
Created by: ashleighobrien
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