Pharmacology for Cardio Disease Management
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| Alpha Adrenergic Antagonist Agents | Reduce perohperal vascular tone by blocking alpha-1-adrenergic receptors; causes dilation and decrease in BP
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| ACE Inhibitor Agents | Suppresses enzyme that converts angiotensin 1 into angiotensin 2; decreasing blood pressure and after load
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| Anticoagulant Agents | Inhibit platelet aggregation and thrombus formation
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| Anti-hyperlipidemia Agents | AKA Statins; lipid modifying agents; inhibit anzyme aciton in cholesterol synthesis, break down LDL, decrease triglycerides, and increase HDL
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| Anti-thrombotic Agents | inhibit platelet aggregation and clot formation
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| Beta Blocker Agents | Block B-Adrenergic receptors; decrease myocardial oxygen demand by decreasing heart rate and contractility
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| Calcium Channel Blocker Agents | Decrease calcium entry int o calcium smooth mm cells resulting in diminished myocardial contraction, vasodilation, and oxygen demand
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| Diuretic Agents | Increase secretion of sodium and urine => reduction in plasma volume and BP; thiazide, loop and potassium sparing
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| Nitrate Agents | Decrease ischemia through smooth mm relaxation and dilation of peripheral vessels
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| Positive Inotropic Agents | Increase force and velocity of myocardial contraction; slow the heart rate; decrease conduction velocity through AV node and decrease degree of activation of SNS
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| Thrombolytic Agents | Facilitate clot dissolution through conversion of plasminogen to plasmin; plasmin breaks down clots and opens blood vessels
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