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Oral Path Test 1
Oral Pathology Test 1
| Question | Answer |
|---|---|
| what does familial mean? | runs in families but not directly inherited |
| what does congenital mean? | present at birth does not imply genetic |
| what is aplasia | with out development |
| what does hypoplasia mean? | under development |
| what are the features that suggest something is developmental? | young age or present at birth asymptomatic bilateral symmetry |
| what are syndromes? | combinations of findings which define a condition |
| what is agnathia? | no jaw |
| what is micrognathia? | small jaw |
| what is macrognathia? | large jaw |
| how do you treat cleft lip and palate? | treat lip lesions early palate have to wait until the child is ready to speak |
| what are fordyce granules? | sebaceous glands yellowish plaques that secrete a fatty substance not seen in children |
| what is hereditary fibromatosis gingivae? | autosomal dominant and inherited generalized gingival hyperplasia |
| what are the three things that can cause generalized gingival hyperplasia? | medications, hereditary, syndromes |
| what is aglossia? | no tongue |
| what is microglossia? | small tongue |
| what is ankyloglossia? | fusion of tongue to the floor of the mouth |
| what does it mean to be tongue tied? | lingual frenum is too far attached |
| what is cleft tongue? | lack of complete fusion of lateral halves |
| what is fissured tongue? | NOT DEVELOPMENTAL age related deep grooves that occasionally related to xerostomia |
| what is benign migratory glossitis? | geographic tongue or erythema migrans atrophic red areas with yellowish white borders |
| what causes hairy tongue? | elongated filiform papillae caused by drugs, radiation, smoking NOT DEVELOPMENTAL |
| what is Stafney's bone cavity? | submandibular salivary gland, radiolucency corticated borders below the inferior alveolar canal |
| what is microdontia? | small teeth |
| what is macrodontia? | large teeth |
| what is an enamel pearl? | excess of enamel at bi or trifurcation of molars at the CEJ |
| what is gemination? | division of tooth germ with incomplete formation of two teeth, tooth splits in two, almost always crown of the tooth usually single root split coronal |
| what is fusion of teeth? | union of two tooth germs by dentin commonly has two roots |
| what is concrescence of teeth? | fusion by cementum |
| what is a dilaceration of teeth? | bend or curve, possibly from trauma |
| what is dens in dente? | tooth within a tooth, shell of enamel and dentin inside tooth |
| what is taurodontism? | block shaped teeth with large pulps |
| what are two reasons for taurodontism? | phenotypic expression, development from ancestors Klinefelter's syndrome |
| what is anodontia? | no teeth at all hereditary |
| what is partial anodontia or oligodontia? | some but not all teeth are missing |
| what are the congenitally missing teeth? | 3rd molars maxillary lateral premolars |
| what are mesiodens? | between maxillary centrals and is most common |
| how can you tell if the enamel defect is hereditary? | all teeth affected, both dentitions family history |
| how can you tell if the enamel defect is environmental? | turns on and off, one dentition is affected no family history |
| what is amelogenesis imperfecta? | inherited defect of ameloblast abnormal enamel created by ameloblasts autosomal dominant, recessive |
| what are the environmental defects for enamel? | nutritional: calcium exanthematous: virus in your epithelium congenital syphilis local infection or trauma fluorosis, tetracycline |
| what is the exception of environmental enamel defect that will affect both dentitions? | fluorosis |
| what is dentinogenesis imperfecta? | inherited defect of odontoblasts autosomal dominant |
| what causes dental caries? | formation of plaque, adherence of microorganisms to plaque, microorganisms ferment carbohydrates of the plaque, acid dissolves dental hard tissue |
| what are the microorganisms that adhere to plaque to cause cavities? | streptococcus mutans lactobacillus |
| what do early lesions look like on the tooth? incipient caries | lusterless or chalky appearing white spot which is produced by early demineralization of enamels |
| what do later lesions that turn into cavities look like? | produce a depression, often discolored |
| where do cavities occur on teeth? | pits and fissures, smooth enamel surfaces, root surfaces |
| what are the most common cavities? | pit and fissure cavities |
| how do pit and fissure cavities look on a radiograph? | diamond shape radiolucency |
| how do smooth surfaces look on a radiograph? | double triangle |
| why do smooth surface caries appear mostly on the facial surfaces of teeth? | because the height of contour is closer to the occlusal surface |
| what do root surface caries look like on the radiographs? | triangle radiolucency |
| how does a meth mouth look? | black extensive decay focused initially around gingival line leaves on roots of teeth as it amputates crown dry mouth |
| what is pulpitis? | inflammation involving the dental pulp this happens by dilation of the blood vessels |
| what causes pulpitis? | caries, trauma, dental restorations |
| what is reversible pulpitis symptoms? | pain mostly on cold, pain fades away after stimulus is removed pain is not always there or reproducible, radiographs appear normal |
| how do you treat reversible pulpitis? | remove the cause if identifiable give the tooth time |
| what are the symptoms of irreversible pulpitis? | pain typically lingers with removal of stimulation, varying degree of pain that increases with time, mostly hot produces pain and have percussion and chewing pressure pain. |
| what do radiographs show about irreversible pulpitis? | slight widening of the periodontal ligament, inflammation in the pulp |
| what is the treatment for pulpitis? | endodontic therapy or extraction |
| what is pulp necrosis? | death of pulp tissue due to overwhelming inflammation in a confined environment |
| why does a tooth that undergoes pulp necrosis appear discolored? | bleeding in pulp chamber spills red blood cells into dentinal tubules the breakdown causes hemosiderin or brown pigment |
| what does pulp necrosis look like radiographically? | thickening of the periodontal ligament area and destruction of bone in periodical region |
| what is chronic hyper plastic pulpitis? | pulp polyp, large carious lesions with exposure of the pulp pulp grows out from inside the tooth to produce red soft tissue mass in the crown area |
| what are iatrogenic? | mistakes or injuries produced by healthcare professionals |
| what are factorial lesions? | self induced and may have psychogenic component patients do these themselves, nervous habit |
| what is frictional keratosis? | most common lesion, physically rubbing on your tissue protective phenomenon that your surface epithelium will make keratin usually a white plaque |
| what is morsicatio bucarum? | cheek biting |
| what is a traumatic ulcer? | most common cause of intraoral ulceration loss of surface epithelium from physical injury tan: covered by fibrinous exudate |
| where are the most common places to find traumatic ulcers? | tongue and lower lip |
| what is a traumatic granuloma? | traumatic ulcer with injury to underlying muscle doesn't heal, ulcers that don't go away, most common on tongue |
| what is a amalgam tattoo? | slate bluish gray discoloration from traumatic implantation of amalgam producing subsurface discoloration |
| what is a hematoma? | bite yourself and break a blood vessel so blood leaks out extravascular bleeding onto tissue |
| what are petechiae? | pinpoint bleeding from capillaries |
| what do we see in acute radiation therapy? | dermatitis/stomatitis |
| what do we see in chronic radiation therapy? | salivary glands sensitive, decreased salivary flow dryness and discomfort, burning that may lead to candidosis difficulty eating, speaking and swallowing increased cervical caries |
| what is osteoradionecrosis? | bone breaks down, radiation damages osteocytes very prone to infection |
| what do burns in oral mucosa look like? | white |
| what are the medications that cause generalized gingival hyperplasia? | phenytoin (Dilantin) cyclosporine calcium channel blockers |
| what is type I hypersensitivity? | IgE mediated, histamine potent vasodilator red lesions on skin causing anaphylaxis |
| what is type IV hypersensitivity? | delayed hypersensitivity T cell mediated white lesions |
| what is angioedema? | lip swelling |
| what is stomatitis medicamentosa? | swallowing something and you react to it systemically |
| what is stomatitis venenata? | put something inside your mouth and react to it topically |
| what does a cinnamon allergy look like? | red and white combination lesion increased desequamative gingivitis |
| what is attrition? | loss of tooth structure occlusally and interproximally due to direct tooth to tooth contact |
| what is physiological attrition? | eating, tooth to tooth contact |
| what is pathological attrition? | bruxism, non functional habits |
| why does inter proximal attrition happen? | because the teeth move normally in the bone |
| what is abrasion? | loss of tooth structure from mechanical habit |
| what is the most common form of abrasion? | cervical abrasion caused by horizontal brushing most apparent in canine and premolar area presents as a V shape in cervical area of the tooth |
| what is erosion? | loss of tooth structure from chemical process, most often non-bacterial acid dissolution intrinsic or extrinsic, looks like polished surface with sharp edge of enamel |
| what is perimylosys? | erosion of teeth due to gastric secretions |
| what is the pattern for erosion for eating disorders? | palatal surfaces of the maxillary later surfaces but not any other teeth |
| why does erosion not happen on mandibular teeth? | protected by tongue and saliva pools in the floor of the mouth |
| what is abfraction? | loss of tooth structure due to repeated tooth flexure caused by occlusal stress always found in cervical third |
| what is demastication? | combined effect of attrition and abrasion |
| what is secondary dentin? | physiologic deposition of dentin throughout life formed after root completion, responsible for making pulp chamber smaller |
| what is reparative dentin? | localized formation of dentin on pulp formed under pathological conditions: injury |
| what are dead tracts? | dentinal tubules devoid of cytoplasmic processes of odontoblasts dentin is beyond repair |
| what makes up physiological dentin? | primary and secondary dentin |
| what causes pulp calcifications? | denticles, pulp stones, diffuse linear calcifications |
| what is hypercementosis? | abnormal thickening of cementum |
| what causes localized hypercementosis? | inflammation, excessive occlusion, fracture, loss of antagonist, idopathic |
| what causes generalized hypercementosis? | Paget's disease of bone |
| what are cementicles? | calcifications in PDL |
| what can cause pathologic external resorption of roots? | pager's disease of bone |
| what is pink tooth of mummery indicate? | internal resorption, inflammed pulp |
| what is a tumor? | swelling or inflammation |
| what is a neoplasm? | abnormal mass of tissue growth which exceeds and is uncoordinated with that of normal tissue |
| what is pyogenic? | pus forming |
| what is a granuloma? | microscopic aggregation of macrophages that are transformed into epithelia cells |
| what is hyperplasia? | increase in the size of an organ associated with an increase in the number of cells |
| what is a hamartoma? | excessive but focal overgrowth of cells and tissues native to the organ in which it occurs |
| what causes a fibroma? | local irritation or trauma can occur in any location, usually older individuals |
| what does a fibroma look like? | smooth surfaced pink nodule |
| what is another name for inflammatory fibrous hyperplasia? | eulis fissuratium or denture epulis |
| where does inflammatory fibrous hyperplasia develop? | flange of an ill fitting denture folds of tissue in the alveolar vestibule |
| what does an inflammatory fibrous hyperplasia look like? | firm fibrous tissue, can be ulcerated most commonly in females |
| what are the two that occur on the gingiva only? | peripheral ossifying fibroma peripheral giant cell granuloma |
| where is it common to see peripheral ossifying fibroma? | young patients, mostly female |
| what causes peripheral giant cell granulomas? | local irritation and trauma |
| what does a peripheral giant cell granuloma look like? | red blue or red nodular mass occurring only in the soft tissue |
| what population does a pyogenic granuloma occur in? | children, young adults and pregnant women |
| what does a pyogenic granuloma look like? | smooth lobulated mass, pedunculated or sessile, bleeds a lot ulcerated pink or red purple |
| what is a hemangioma? | benign tumor of blood vessels most common in head and neck region |
| what does a hemangioma appear like? | blue or reddish blue |
| what is a lymphangioma? | benign tumor of lymphatic vessels occurring in the head and neck region most common on the tongue |
| what does a lymphangioma look like? | tapioca pudding or frog egg appearance |
| what causes inflammatory papillary hyperplasia? | ill fitting, poor denture hygiene, wearing denture 24 hours a day found mostly below the denture |
| what causes a verruciform xanthoma? | response to localized epithelial trauma or damage |
| what does a verruciform xanthoma look like? | hyper plastic epithelial lesion found on the oral mucosa skin or genitialia white, yellow white or red in color, papillary verrucoid or flat topped surface |
| what causes traumatic neuroma? | benign tumor, reactive prolieration of nerver tissue after injury or trauma |
| where is traumatic neuroma most commonly found? | mental foramen area, tongue or lower lip pain or numbness |
| what are the most common tumor like proliferations? | fibroma inflammatory fibrous hyperplasia peripheral ossifying fibroma peripheral giant cell granuloma pyogenic granuloma |
| what are the vascular lesions? | hemangioma lymphangioma |