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preadlscent-postmeno
OH3-preadolescent-postmenopausal
| Question | Answer |
|---|---|
| what is menopause | without menstruation, end of fertility, 12 months amenorrhea in females 45 and older, occurs between 47-55, |
| dysmenorrhea | no menstruation |
| what is LAP | localized aggressive periodontitis, 5-47%, actinobacillus actinomycetemcomitans, 1st diagnosed at puberty, 1st molars and incisors |
| what is GAP | generalized aggressive periodontitis, caused by microflora, occurs in younger than 30yrs, proximal surfaces attachment loss in 3 teeth other than 1st molars and incisors |
| factors that affect menstruation cycle | climate, work schedule, emotional trauma, acute/chronic illness, weight loss/excessive weight gain |
| when does PMS occur | 7-10 days prior |
| dysmenorrhea-primary (functional) | normal w/ symptoms of hyperactivity/contractions |
| dysmenorrhea-secondary (acquired) | abnormal or result endometriosis |
| dysmenorrhea-physiologic/psychologic | inadequate prep for puberty |
| menopause effects | skin and mucous decrease thickness, become fragile, osteoporosis, predisposed to atherosclerosis, diabetes, hypothyroidism, dysguisia, tissue appear shiny and dry, saliva substitute for xerostomia |
| papillon le-fevre syndrome | palmoplantar keratodermas, type 4 (19 diff. types), hyperkerintization on hands and feet and early onset of perio. disease, severe inflammation, collagen fibers of PDL destroyed, alve. bone lost=tooth lost genetic, autosomal, recessive disorder |
| chediak-higashi syndrome (defects) | cellular disorder: neutropenia, defective chemotaxis, defective platelets=prolonged bleeding time defects lymphocytes, monocytes, macrophages, partial albinism |
| chediak-higashi syndrome (infections) | recurrent infections: skin, respiratory tract, mucous mem., photophobia, neurological manifestations, autosomal gene disorder w/ onset in early childhood |
| cohen syndrome | small head, short upper lip, high narrow palate -obesity, decrease muscle tone, mod. learning disabilities, decrease WBC, genetic autosomal recessive disorder |
| Ehlers danlos syndrome | genetic disorder, autosomal dominant/recessive, x-linked affect collagen structure and function |
| EDS-classical | skin fragility, extensive scarring, poor muscle tone, sprains |
| EDS-hypermobility | excessively loose joints, joint dislocation (jaw/knee/shoulder), chronic limb and joint pain |
| EDS-vascular | weak CT in intestines, arteries, uterus= organ/blood vessel rupture, bruise easily, risks: complications following surgery, collasped lung, premature death |
| EDS-kyphoscoliosis | joint looseness, scoliosis |
| EDS-arthrochalasia | loss of bone mass, dislocation of hip |
| EDS-dermatosparaxis | hernias may occur, aged appearance to young adults, fragile skin, bruise easily |
| hypophosphatasia | metabolic bone disease, caused by low levels of alkaline phosphatase, inorganic pyrophosphate accumulate= Rickets (children) and osteomalacia (adults) |
| H- perinatal | =stillbirth |
| H- infantile | before 6mths w/ inadequate lbs. poor appetite, Rickets, premature tooth loss before 5yrs |
| H- childhood | *see infantile delayed walking, bowed legs, knock knees, muscle pain and weakness in lower limbs |
| odontohypophosphatasia | premature tooth loss without skeletal problems |
| pseudohypophosphatasia | *see infantile with normal ALP activity |
| adult hypophophatasia | middle age, stress fractures in feet, thigh, hips, early secondary tooth loss, arthritis, degeneration prevalent in Manitoba, Canada |
| syphilis-treatment | antibiotics: penicillin (1st), erythromycin, cephalosporin |
| gonococcal stomatitis-treatment | antibiotics- penicillin (1st), tetracycline |
| NOMA gangrenous stomatitis-treatment | antibiotics, conservative debridement |
| syphilis-bacteria | treponema pallidum |
| syphilis | primary- chancre appears, ~3wks after infection, ulcer secondary-6-8wks after ulcer appearance, mucous patches on palate + gingiva, systemic: malaise, fever, headache, sore throat, myalgia, lymphadenopathy tertiary-begins 4-7yrs |
| gonococcal stomatitis-bacteria | neisseria gonorhoeae |
| gonococcal stomatitis | invovles urinary tract and anorectal area, oral charac.:erythema, edema on pharyngeal, tonsils, uvula, soft palate |
| NOMA gangrenous stomatitis-bacteria | f. nucleatum, p.intermedia, b.vincenti, streptococcus species, staph. aureus |
| NOMA gangrenous stomatitis | oral charac. NUG, systemic- fever malaise, halitosis, lymphadenopathy |