cardio emergen yo Word Scramble
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Question | Answer |
Stable Plaque | Hard thick fibrous cap, less likely to rupture -collateral circulation develops due to this |
Vulnerable plaque | Soft,lipid rich material,thin cap pf fibrous tissue that seperates from vessel lumen |
Plaque ruptures due to what? | -force of blood flow -coronary spasm |
how long does it take for platelets to start adhearing once a rupture occurs | 1-5 seconds |
Thromboxane | A chemical that causes platelet aggregation and vasoconstriction |
ASA | a glycoprotein inhibitor |
Fibrin develops how | 1.injury releases clotting factors,2.prothrombin activator,3.calciumcauses it to convert to thrombin,4.thrombin +fibrinogen=fibrin |
Heparin | inactivates thrombin,no fibrin development |
Coumadin | interferes with the clotting factors,decreases synthesis of prothrombin |
VLDL | carries fat(cholesterol) and triglyceides from liver to body= should be <30 mg/dl |
LDL | 70% of total serum cholesterol (stick easy to vessel walls) should be <160 mg/dl-richest in fat |
HDL | <60,mostly protein, returns LDL's to the liver for removal |
Triglycerides | form of fat that comes from food, should be less than 200 mg/dl |
These organs require high levels of cholesterol to funtion | Brain,liver,kidneys,adrenal glands, myelin sheathe, reproductive organs |
Vessels commonly affected by athersclerosis | Aorta and its branches,coronary arteries and cerebral arteries,renal arteries and legs |
Purpose of the endothelium tissue | lines the interior surface of blood vessels,fluid diltration, blood vessel tone and hemeostasis |
Atherosclerosis effects of blood vessels | 1.vessel elasticity is lost 2.diameter is decreased |
Cholesterol | soapy, white chemical found in body tissue, made in the liver, produces 1.5-2 grams/day |
Types of angina | Prinzmetal, stable, unstable |
What 2 processes cause angina tp develop | 1/Athersclerosis. 2/coronary vasospasm |
Prinzmental Angina | (vasospastic,atypical, varient) due to vasospasms-at rest,when sleeping or when exposed to cold weather |
Stable angina | due to stress and physical activity-located substernally/radiating-pain less than 1-5 min,lasts up to 15 min |
Unstable Angina | occurs at rest, during light exercise-substernally-lasts more than 10 mins |
Angina develops due to what | the build up of Co2/lactic acid in the ischemic tissue , activating nerve endings causing pain |
LCA supplies how much blood to the heart | 85% |
Left coronary occlusion leads to | Anterior,lateral and septal wall MI's |
Right coronary occlusion leads to | inferior, posterior ,right ventriclular and damage to the conduction system |
Extent of MI (SUBENDOCARDIAL) | not a full muscle thickness,non patholigical Q wave |
Extent of MI (TRANSMURAL) | Full thickness infarct,pathological Q wave |
Death secondary to MI is caused by | 1.Fatal dysrhythmias/sudden death,2.Heart failure-chf.3.Cardiogenic shock 4.Ventricular aneurysm.5,ventricular aneurysm |
Inferior Wall infart causes what response | PSN response |
Anterior wall infarct causes what response | SNS response |
The most common cause of a AMI | Coronary thrombus |
Where do dyrythmias develop | they develop around ischemic and becrotic tissue |
What is a warning dysrhythmia | PVC"s |
GOals of management of MI pt's | -pain relief-repurfusion therapy-rapid, safe transport-thrombolytic therapy-prevent dysrhythmias |
Created by:
rebeccabelleth
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