Busy. Please wait.

show password
Forgot Password?

Don't have an account?  Sign up 

Username is available taken
show password


Make sure to remember your password. If you forget it there is no way for StudyStack to send you a reset link. You would need to create a new account.
We do not share your email address with others. It is only used to allow you to reset your password. For details read our Privacy Policy and Terms of Service.

Already a StudyStack user? Log In

Reset Password
Enter the associated with your account, and we'll email you a link to reset your password.

Remove Ads
Don't know
remaining cards
To flip the current card, click it or press the Spacebar key.  To move the current card to one of the three colored boxes, click on the box.  You may also press the UP ARROW key to move the card to the "Know" box, the DOWN ARROW key to move the card to the "Don't know" box, or the RIGHT ARROW key to move the card to the Remaining box.  You may also click on the card displayed in any of the three boxes to bring that card back to the center.

Pass complete!

"Know" box contains:
Time elapsed:
restart all cards

Embed Code - If you would like this activity on your web page, copy the script below and paste it into your web page.

  Normal Size     Small Size show me how

cardio emergen yo

cardiac emergencies

Stable Plaque Hard thick fibrous cap, less likely to rupture -collateral circulation develops due to this
Vulnerable plaque Soft,lipid rich material,thin cap pf fibrous tissue that seperates from vessel lumen
Plaque ruptures due to what? -force of blood flow -coronary spasm
how long does it take for platelets to start adhearing once a rupture occurs 1-5 seconds
Thromboxane A chemical that causes platelet aggregation and vasoconstriction
ASA a glycoprotein inhibitor
Fibrin develops how 1.injury releases clotting factors,2.prothrombin activator,3.calciumcauses it to convert to thrombin,4.thrombin +fibrinogen=fibrin
Heparin inactivates thrombin,no fibrin development
Coumadin interferes with the clotting factors,decreases synthesis of prothrombin
VLDL carries fat(cholesterol) and triglyceides from liver to body= should be <30 mg/dl
LDL 70% of total serum cholesterol (stick easy to vessel walls) should be <160 mg/dl-richest in fat
HDL <60,mostly protein, returns LDL's to the liver for removal
Triglycerides form of fat that comes from food, should be less than 200 mg/dl
These organs require high levels of cholesterol to funtion Brain,liver,kidneys,adrenal glands, myelin sheathe, reproductive organs
Vessels commonly affected by athersclerosis Aorta and its branches,coronary arteries and cerebral arteries,renal arteries and legs
Purpose of the endothelium tissue lines the interior surface of blood vessels,fluid diltration, blood vessel tone and hemeostasis
Atherosclerosis effects of blood vessels 1.vessel elasticity is lost 2.diameter is decreased
Cholesterol soapy, white chemical found in body tissue, made in the liver, produces 1.5-2 grams/day
Types of angina Prinzmetal, stable, unstable
What 2 processes cause angina tp develop 1/Athersclerosis. 2/coronary vasospasm
Prinzmental Angina (vasospastic,atypical, varient) due to vasospasms-at rest,when sleeping or when exposed to cold weather
Stable angina due to stress and physical activity-located substernally/radiating-pain less than 1-5 min,lasts up to 15 min
Unstable Angina occurs at rest, during light exercise-substernally-lasts more than 10 mins
Angina develops due to what the build up of Co2/lactic acid in the ischemic tissue , activating nerve endings causing pain
LCA supplies how much blood to the heart 85%
Left coronary occlusion leads to Anterior,lateral and septal wall MI's
Right coronary occlusion leads to inferior, posterior ,right ventriclular and damage to the conduction system
Extent of MI (SUBENDOCARDIAL) not a full muscle thickness,non patholigical Q wave
Extent of MI (TRANSMURAL) Full thickness infarct,pathological Q wave
Death secondary to MI is caused by 1.Fatal dysrhythmias/sudden death,2.Heart failure-chf.3.Cardiogenic shock 4.Ventricular aneurysm.5,ventricular aneurysm
Inferior Wall infart causes what response PSN response
Anterior wall infarct causes what response SNS response
The most common cause of a AMI Coronary thrombus
Where do dyrythmias develop they develop around ischemic and becrotic tissue
What is a warning dysrhythmia PVC"s
GOals of management of MI pt's -pain relief-repurfusion therapy-rapid, safe transport-thrombolytic therapy-prevent dysrhythmias
Created by: rebeccabelleth