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Advanced Respiratory: Oxygenation/Ventilation

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Answer
ICU patients are very ill and that's why there're in the ICU   Sedated... In a severe amount of pain. Changed LOC. Respirator and Cardiac status are compromised  
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Although we have technology   Assess the patient-identify what's happening with them and implement if needed  
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Be prepared-What can potentially go wrong   Assess-know patient's history and what can I anticipate, be on the lookout for Expect more serious things can happen with these patients  
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With a higher acuity patient   Anything can happen at any minute  
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If an alarm goes off or a number looks abnormal   First assess the patient Look before you intervene-adding oxygen or repositioning Look at history, why they're there and how they look What do I need to do right now to take care of this patient  
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If a patient looks like they are having difficulty breathing   Oxygen therapy Reposition - move up in bed if slumped Cough/deep breath suction Medications- look at standing orders or current orders  
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Social history   Smoking-how long? Smokers tend to do much worse d/t damage to lungs and mucosa, don't have as good responses, especially older smokers Young smokers in traumas harder to manage  
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Cardiopulmonary history   Do they have an underlying heart failure Taxing causes ↑ HR and exacerbation of HF symptoms, perfusion  
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Elimination history   Listen for crackles, assess urine output,  
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Sleep/Rest history   Some people have issues lying down Be aware of things like orthopnea , obstructive apnea  
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Dyspnea assessment   At rest or upon exertion? Postop pts can have dyspnea upon exertion-are they tolerating it? Sats still ok?  
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Chest pain assessment   Commonly ask-could have obstacles to communication ie lines, tubes  
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Cough / Sputum assessment   listen to cough, productive?, thick?, wheezy sounding? how does sputum look  
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Voice changes assessment   More hoarse, dry sounding with distress  
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Fatigue assessment   Overwhelming tiredness even when person is resting Could be another issue  
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Nursing Assessment Past   Disease processes (past and current) Flu season? Immunizations current? Environmental exposures/Behaviors (past and current) Medical/surgical hospitalizations  
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cardinal s/sx of respiratory distress   Hypoxia Restlessness Diaphoresis Tachycardia Cool skin  
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S/SX if cardiac involvement of respiratory distress   Dyspnea, wheezing, cough, sputum, palpitations, swollen feet Don't rely on monitor-s/sx can be masked ie., tachycardia masked by beta blockers Fatigue Chest pain Anxiety-anxiety is big Dizziness Bradycardia  
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Key Factors that cause hypoxia or impede pts breathing   Blocked airway Secretions Underlying condition asthma, COPD, PE Allergies, allergic reaction Meds-watch SE Sedatives can impede breathing Age Elderly ↓elastic recoil, ↓ ability of the chest cage to move in and out Positioning  
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Observing the chest   Shape of the chest Equal in expansion Observe abdomen when they breath-assess for belly breathing Chest is going one way, belly the other using abdominal muscles d/t weak diaphragm-ominous sign What's normal for the patient?  
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Signs of respiratory distress What you may see……….   Pursed lip breathing Pallor, clammy, cool skin ↓ cap refill Clubbing-long-term sign Barrel chest Respiratory rate (12-20) However, all pts are different  
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A pt with COPD expiratory time?   A pt with COPD has a longer expiratory time, trying to keep the alveoli open COPD can also have cardiac involvement-observe for JVD and distant heart tones  
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Cheyne-stokes   Respirations gradually increase in depth, then become more shallow, followed by a period of apnea.  
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Biot's   Highly irregular breathing pattern with abrupt pauses between efforts  
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Kussmaul's   Respiration faster and deeper without pauses  
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Apneustic   Respirations prolonged, gasping, followed by extremely short, inefficient expiration  
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Factors that alter a good wave form on a monitor   Nail polish, Cold fingers, lotions  
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Respiratory Assessment   Will I see this, What does it mean? How will I respond? Chest wall excursion, symetrical Rib fracture, pneumothorcias Tracheal deviation Chest wall tenderness Crepitus Tactile Fremitus  
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Normal breath sounds include:   bronchial bronchovesicular Vesicular  
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bronchial   bronchial  
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bronchovesicular   bronchovesicular  
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Vesicular   Vesicular  
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Adventitious breath sounds include:   Crackles, Wheezes, Rhonchi, Pleural friction rub, Stridor  
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Crackles   Fluid or mucous moving through the smaller airways Crackles can't be cleared with coughing, need loop diuretics, ↓ fluids  
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Wheezes   High-pitched musical sounds  
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Rhonchi   More course and larger airway than crackles Can be cleared with coughing and suctioning  
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Pleural friction rub   Course, grating leather on leather rubbing sound Pneumonia, TB, pleural effusions  
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Stridor   Narrowing of the large airways Anaphylactic reaction or extubated pt  
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Restlessness and agitation   Look at the pulse ox, what is the pt restlessness and agitated for, it there something else going on? Could it be a pain situation?  
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Decreased LOC   Underlying medication causing LOC? Or hypoxic? We can give oxygen, but it doesn't mean they will perfuse it.  
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Change in breathing pattern   Reposition. Did it help?  
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Cyanosis or dusky   Usually a late sign - can happen quickly sometimes  
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Accessory muscles   Ominous sign-don't want to see  
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Dyspnea or orthopnea   Turn our pts every 2 hours-chg quickly If they don't recover put in fowlers position, administer ↑ O2 if low sats  
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Adventitious sounds   Changes from assessment to the next things are moving around Be prepared for issues that might arise  
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Onset of S/S distress Early s/sx   Restlessness/irritability & confusion Tachypnea/DOE Tachycardia/HTN  
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Onset of S/S distress Late s/sx   Combativeness Dyspnea at rest Cyanosis  
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Dysrhythmias   early or late  
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SV02   Mixed venous oxygen saturation How well does the oxygen saturate with the hemoglobin at the venous level "ABG at venous level" Tells us if they need more oxygen if on supplemental O2  
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SV02 tissue perfusion   At what rate is the body using oxygen, is the body demanding more oxygen to perfuse the tissues than what we're giving them? Manytimes used with pts w/ resp and cardiac problems  
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Don't start ? before Sputum tests   Don't start ABX before new sputum sample  
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Pulmonary angiograms   Femoral artery to pulmonary vasculature Most accurate to diagnose and locate PE's, CAT scan 2nd  
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ETCO2 monitoring (end tidal)   At the end of respiration we assess CO2 levels because: Gas exchange takes place at the end of expiration down in the alveoli  
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V/Q scans   Ventilation perfusion scan, how well are they ventilating and then are they perfusiing that oxygen and exchanging gas  
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Balance and Imbalance in Ventilation Perfusion (VQ)   We want a normal balance of our ventilation and perfusion  
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Alterations in Ventilation:   Blockage Gravity  
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Alterations in Perfusion:   Pressures (airway/PA)  
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Ventilation-Perfusion Relationships V/Q ratio   Relationship between_the alveoli to _the flow of blood of the lungs__ -ventilation to flow of blood  
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Emphysema and COPD   Emphysema and COPD reduces the surface area of the alveoli  
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V/Q is greatest in   V/Q is greatest in __base of the lungs because that where the majority of our gas exchange takes place  
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Possible VQ States   Normal-ventilation matches perfusion Low ventilation/ perfusion exceeds High ventilation/ low perfusion  
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Tidal volume (VT)   Amount of inhaled and exhaled air in mL, normally 6 - 10 mL/kg  
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Inspiratory capacity (IC)   Measured with incentive spirometer  
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Placement of ET tube Proper placement   Endo-tracheal - about 4 cm above the carina  
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How would you know the tube was not in the appropriate position?   Bilateral breath sounds O2 sats come up nicely End-tidal CO2 within normal limits Then, look at chest x-ray for final confirmation Note and chart ET tube depth measurement at lips  
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Improper Placement   With improper ET tube placement too low you will hear going into one lung but not the other Common to get placed in the right stem bronchus  
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If a patient extubates themselves   Assess pt-need more O2? Bag, non-rebreather? Call for physican  
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Ambu bag   Need in room! Need bag and valve! Need O2 wall or cylinder.  
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MECHANICAL Ventilation Indications:   Inability to breath or apneic Severe impaired ABG imbalance-not primary reason Severe failure, hypoxia despite O2 therapy Muscle fatigue  
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Benefits of MV   Decreases system & MVO2 requirements Helps L ventricle, decreases O2 requirements of the L ventricle Permit sedation Reduce ICP Hyperventilation reduces ICP Prevent atelectasis Secure airway Too much sedation causes respiratory depression  
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Goals of MV   Reduction in work of breathing Assurance of patient comfort Synchrony with ventilator Adequacy of ventilation and oxygenation  
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