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Unit A Congestive Heart Failure Study Notes

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Question
Answer
Classifications of CHF   show
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a state in which the heart is no longer able to pump sufficient blood to meet all metabolic needs of the body systems.   show
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show low output HF  
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cardiac output remains above normal or normal; caused by increased metabolic needs or hyperkinetic conditions- septicemia, anemia, hyperthyroidism   show
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show systolic HF  
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show systolic HF  
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The percentage of blood ejected from the heart during systole.   show
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decreased ejection fraction leads to what?   show
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Left ventricle is unable to relax adequately during diastole. Represent 20-40% of all heart failures.   show
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Inadequate relaxtion equals what to the heart?   show
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What is the regular ejection fraction of the heart?   show
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show ejection fraction less than 45%, no symptoms, only dx with echo, cardiac cath, cardiac test.  
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Class II   show
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show symptoms of heart failure with no exertion  
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show symptoms at rest.  
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show Acute MI  
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show decrease filling of ventricles, decrease stroke volume, and increase oxygen demand.  
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thyroid disorders cause heart failure how?   show
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hypertensive crisis   show
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Rupture of the papillary muscles affects the heart how?   show
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show hole between 2 ventricles  
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what are the acute onset of CHF   show
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Chronic onset of heart failure   show
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Risk factors of heart failure   show
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show stroke volume  
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show preload, contractility, afterload  
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the degree of fiber stretch at the end of diastole, heart is relaxed; corresponds to end diastolic volume and pressure   show
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show contractility  
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the pressure that the ventricles must pump against to empty the chamber effectively;systemic bld pressure   show
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show valves, bld volume, ventricular wall compliance, venous tone, starlings law.  
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the ability of the cardiac fibers to stretch and snap back appropriately(fibers lengthen and shorten in response to need)   show
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show increased SNS stimulation, positive inotropes, decreased with hypoxemia, decreased negative inotropes. increased preload = increased contractility  
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afterload depends on:   show
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What happens when the heart has increased afterload?   show
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_________ in aorta and carotid responds to increase CO2 and sends impulses to basomotor center in the medulla which stimulate nervous system to respond.   show
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show decreased stoke volume because with increased afterload, the left ventricle requires more energy to eject its contents.  
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show disintergration of cells into membrane-bound particles that are eliminated by phagocytosis or shedding. (programmed cell death.)  
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name the neurohormones that are released in heart failure   show
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show hormone produced and released by the ventricles, increased to counterbalance the RAS, thus decreasing preload. Determines the degree of HF.  
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RAS? And how is RAS activated in HF clients?   show
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What occurs in the body when RAS is activated?   show
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which hormone from the RAS system contributes to ventricular remodeling?   show
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What happens to the heart once it begins the ventricular remodeling process?   show
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show Adequate perfusion, pressure, and cardiac output.  
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show Increased SNS= beta 1, beta 2 stimulation; increased peripheral vasoconstriction, RAS activated  
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show is not meant to be longterm control  
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show beta 1: Tachycardia  
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show increase heart rate to increase cardiac output.  
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show the heart rate will be so rapid that the filling time of the ventricles will decrease.  
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Ventricular Dilation   show
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show Muscle fibers begin to lengthen, increase vol. in the ventricles, stretched muscle fibers contracts more forcefully, heart requires more o2, less bld flow, and compensate more than tachycardia.  
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What eventually happens to the heart muscle during ventricular dilation.   show
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Third and final mechanisms, initially increases the force of contraction and enlarges the heart and increase oxygen demand.   show
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show the heart muscles enlarges, begins to cause ventricular remodeling- increase force of contraction, afterload, O2 demand, and wall thickness.  
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What happens to the heart as the heart enlarges and the wall thickens?   show
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show asymptomatic.  
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show Signs/symptoms of CHF.  
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show 0-100  
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BNP levels of 100-400   show
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BNP > 400   show
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Usually the first to fail; the ventricle is unable to pump the sufficient blood in a forward direction.   show
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Blood build up in left ventricle causes:   show
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show bld flow back in to LA, then to lungs causing SOB, and crackles.  
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Early clinical manifestations of Left HF:   show
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show Crackles- d/t leaking of plasma into lungs and pulmonary artery engorgement; orthopnea, PND, cheyne-stokes respirations, cardiac asthma  
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The inability to lay flat with comfortable breathing.   show
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show PND- Paroxysmal Nocturnal Dyspnea  
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irregular breathing pattern- apnea/tachypnea breathing   show
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show Cardiac asthma.  
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show S3 and S4  
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show anxiety, irritability, confusion, memory loss.  
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show Nocturia-urinate at night, early sign. And oliguria- decrease urine output, in late stage.  
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show lying down promote kidney perfusion at night.  
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During the late state of HF, oliguria occurs, why?   show
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show the heart is larger which moves the PMI  
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show become diminished or weak  
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Pulsus alternans   show
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Left sided HF is the most common cause of this type of heart failure.   show
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How does left sided heart failure cause right sided heart failure?   show
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Describe the events that occur during rt sided hf.   show
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In rt ventricular heart failure, peripheral congestion in venous system causes what to the body?   show
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Which organs are mostly affected by Rt. sided HF?   show
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show Weakness, JVD, anorexia, nausea and bloating, hepatomegaly, dependent edema, cyanosis of nailbeds.  
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show sticks out. Jugular venous distention JVD  
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show nausea, anorexia, bloating.  
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Client with RHF c/o abdominal pain, and have jaundice, due to?   show
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show dependent edema, pitting edema.  
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while assess the client with rt sided heart failure you noticed the nailbeds are cyanotic and capillary refills >3, in analyzing these symptoms, you realized these symptoms are due to:   show
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