RESP
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| During inspiration, thorasic cavity volume increases | show 🗑
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| During inspiration, intrapulmonary pressure decreases to -1mmHg | show 🗑
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| During inspiration, air (gases) flows into lungs down its pressure gradient until intrapulmonary pressure is 0 (or equals atmospheric pressure) | show 🗑
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| As the external intercostals contract ribs elevate and sternum flares | show 🗑
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| During expiration, inspiratory muscles relax (diaphragm rises and rib cage descends due to gravity. | show 🗑
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| during expiration, elastic lungs recoil passively; intrapulmonary volume decreases | show 🗑
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| During expiration, intrapulmonary pressure rises to +1mmHg | show 🗑
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| During expiration, air (gases) flow out of lungs down its pressure gradient until intrapulmonary pressure is 0 | show 🗑
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| During expiration, ribs and sternum are depressed as external intercostals relax | show 🗑
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| During expiration, diaphragm moves superiorly as it relaxes | show 🗑
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| Inspiration is an active process | show 🗑
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| VENTRAL RESP. GROUP: VENTROLATERAL PORTION OF MEDULLA. INSP. OR EXPIR. DEPENDING ON THE NERVES STIMULATED | show 🗑
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| THE MOST IMPORTANT ROLE IN CONTROLLING RESP. IS PLAYED BY THE DORSAL RESP. GROUP WHICH RECEIVES INFORMATION FROM THE CHEMORECEPTORS, BARORECEPTORS AND OTHER RECEPTORS IN THE LUNGS THAT HELP IN THE CONTROL | show 🗑
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| RAMP SIGNAL STOPS FOR 3 SECONDS AND STARTS AGAIN THUS CAUSING A STEADY INCREASE IN THE PULMONARY VOL. DURING INSP. INSTEAD OF INSPIRATORY GASPS. | show 🗑
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| THE DURATION OF EXPIRATION IS DETERMINED BY THE DURATION OF INSPIRATION. | show 🗑
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| STRONG SIGNALS FROM THIS CENTER REDUCE THE TIME OF INSPIRATION WHILE WEAK SIGNALS INCREASE the time of inspiration. ` | show 🗑
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| THE VENTRAL GROUP IS MOSTLY INVOLVED IN INCREASED LEVELS OF VENTILATION | show 🗑
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| H+ AND CO2 DIRECTLY STIMULATE THE RESP. CENTER CAUSING INCREASE OF THE INSPIRATORY AND EXPIRATORY SIGNALS. | show 🗑
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| O2 DOESN’T HAVE A DIRECT EFFECT ON THE CENTER BUT ACTS THROUGH THE PERIPH. CHEMORECEPTORS LOCATED IN THE CAROTID AND AORTIC BODIES. | show 🗑
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| PRESS. AGAINST A SURFACE IS CAUSED BY THE CONSTANT IMPACT OF KINETICALLY MOVING MOLECULES AGAINST THAT SURFACE THE TOTAL PRESS. OF A GAS IS DIRECTLY PROPORTIONAL TO THE CONC. OF THE GAS MOLECULES | show 🗑
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| IN THE CASE OF RESP. PHYSIOLOGY WE DEAL WITH MIXTURES OF GASES (O2, CO2, N2). | show 🗑
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| EACH GAS CONTRIBUTES TO THE TOTAL PRESS. IN DIRECT PROPORTION TO ITS [ ] | show 🗑
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| GASES DISSOLVED IN H2O OR THE BODY TISSUES ALSO EXERT PRESSURE BECAUSE THE DISSOLVED MOLECULES ARE MOVING RANDOMLY AND HAVE KINETIC ENERGY. | show 🗑
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| THE PRESSURE OF A GAS IN A SOLUTION IS DETERMINED NOT ONLY BY ITS CONC. BUT ALSO BY THE SOLUBILITY COEFFICIENT OF THE GAS | show 🗑
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| THE PARTIAL PRESS. OF EACH GAS TENDS TO FORCE MOLECULES OF THAT GAS INTO SOLUTION, FIRST IN THE ALVEOLAR MEMB. AND THEN IN THE BLOOD OF THE ALVEOLAR CAPILLARIES. | show 🗑
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| THE RATE AT WHICH THEY ESCAPE FROM ONE MEDIUM TO ANOTHER IS DIRECTLY PROPORTIONAL TO THEIR PARTIAL PRESSURE IN THE BLOOD. | show 🗑
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| THE NET DIFFUSION IS DETERMINED BY THE DIFFERENCE BETWEEN THE TWO PARTIAL PRESS | show 🗑
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| THE GAS PRESS. DIFFERENCE BETWEEN TWO AREAS IS KNOWN AS PRESSURE DIFFERENCE FOR DIFFUSION. | show 🗑
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| O2 IS GOING TO THE BLOOD | show 🗑
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