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Airway Disorders

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Question
Answer
Pulmonary Blood Supply   To lungs for gas exchange  
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Bronchial Blood Supply   From thoracic aorta  
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Conducting Airway   Nasopharynx-Oropharynx-Larynx/Endolarynx-Trachea-Bronchi-Carina-Hili-Pulmonary & Lymphatic Vessels  
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What is Acinus   Cluster of cells, thin walled 16th-23rd divisions, alveolar ducts, the alveolar sacs contain 300 million alveoli  
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Respiratory Functions   Exchange CO2 & O2, maintain acid-base balance  
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Factors Affecting Respirations   *Neurochemical= Medulla @ base of brain-brain stem (pattern) & Pons (rate & debth) *Mechanical= Irritant, stretch & pressure receptors *Hering-Breuer= Keeps us from over stretching  
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Mechanisms Of Breathing-Ventilation   *Inspiration= Air flows into lungs 1-1.5 seconds *Expiration= Longer, gasses flow out of lungs 2-3 seconds *Normal= 12-20 times per minute  
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Examples Of Respiratory Passageway Resistance   Constriction, edema, mucus, tumors, infectious materials, spasms  
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This Happens In Non-Lung Compliance   Use of accessory muscle  
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This Improves Aveolar Surface Tension   Surfactant secretion (sigh)Surfactant lowers surface tension  
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Example Of Diffusion   O2 & CO2 exchange across alveolar capillary membrane, determined by pH, changes from area of high to low concentration, need good perfusion/cardic output  
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Accessory Muscles   *Sternocleidomastoid & Scalenus= Lift up thorax to expand volume * Intercostal and Scalene= Expand A&P (retractions) *Expiration= Abdominal and internal intercostal are compensatory  
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Pulmonary Blood Flow And Gravity   Air rises, blood drops to dependent area, "good lung down" More blood and less O2 lower, less blood and more O2 upper in lobe  
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Cues To Respiratory Illness   SOB, wheezing, pleuritic chest pain, cough, sputum production, hemoptysis , voice change, fatigue  
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Pt. History Assessment   Predominant complaints, family hx, health hx, smoking hx, occupational exposure  
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Patterns Of Breathing   *Kussmauls= Rapid breathing (compensatory) *Tachypnea= Fast >20 (intervine 30-40) *Tachy-Brady= OH SHIT *Brady= <12, start looking for reasons *Biots= Varies in rate, debth & rhythm w/ irregular periods of apnea (sign of brain stem problem)  
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Bronchospasms   Constriction (vagus nerve) Histamine release= >mucus/prostoglandin= more constriction  
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Causes Of Bronchospasms   NSAID, ASA, -olol, ACE, some inhalers  
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Symptoms Of Bronchospasm   SOB, chest tightness, fatigue, "silent chest"  
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Steps In Respiratory Assessment   #1 Inspection #2 Palpation #3 Percussion #4 Auscultation  
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Significance Of Positioning   *Lying=mild distress *Sitting=moderate *Upright=severe *Tripod=increases A&P diameter *Orthopnea="one,two,three pillow"  
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Finger Clubbing   Angle normally 20, occurs when body trying to compensate for hypoxia-develops collateral circulation  
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Significance Of Speech   Sentence= mild/moderate dyspnea *Phrase= moderate *Words= severe "1-2-3 word dyspnea"  
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Palpation   With pads of fingers, crepitus-crackles, nodes-if swollen should move, mediastinal shift- trachea shifts to opposite side of lung injury.  
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Tactile Fremitus   Vibration, ask pt. to say 99, decreased in atelectasis, emphysema, asthma, pleural effusion & pneumothorax, increased in pneumonia, tumor, secretions  
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Sounds Of Percussion   *Flat=solid (sternum) *Dull=no air/fluid (liver) *Tympanic=air (stomach) *Resonant=echo (lung) *Hyperresonant=low pitch, air free  
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How To Auscultation Pt.   Deep breath through mouth, not through gowns, listen laterally with an effusion, R= 3 lobes L= 2 lobes  
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Normal Breath Sound Locations   *Bronchial= loud, high pitched, over large airways, expiratory * Bronchiovesicular= medium pitch, R&L bronchus, i=e *Vesicular= soft/low pitch, i>e *E-I-E-I-I  
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Crackles   Rales, not cleared by coughing, fluid scruntching down on aveoli, ex. pulmonary edema  
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Sonorous Wheezes   Rhonchi-gurgle, heard on expiration, occurs in conducting area, ex. COPD, asthma  
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Stridor   High pitched, harsh, inspiratory ex. laryngeal spasms due to tetany w/ low calcium, croup  
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Friction Rub   Loud, dry, creaking, loss of lubricant, most often heard laterally ex. pleurisy, pleuritis, effusion, poss pneumonia  
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Absent Or Diminished   Ex. atelectasis, pleural effusion, pneumonia, worsening bronchonconstriction  
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Pulse Ox   SaO2= saturation of oxygen on hemoglobin**Does not determine acid-base status  
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Inaccurate Pulse Ox Reading   Hypothermia, hypotension, vasoconstriction, IV dyes, HGB bound with other gas other then O2 eg. coal miner, <70% is + or - 4%  
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Pulse Ox Results   **<90-91% (12 hrs)= Report & corrective nursing action **<80% (4 hrs)= Hurry **<70% (1 hr)= You better run!  
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Normal VQ Scan (Ventilation/Perfusion)   0.8-0.9, Perfect= 1mL O2 per 1mL of blood, Abnormal VQ= hypoxia, most often done for pulmonary embolism or baseline for someone with ARDS  
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Shunt   Low ventilation, "STUNT"= stump  
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Silent Unit   Poor VQ, compensatory-diverts blood to better ventilated areas, ex. PE, chronic alveolar collapse  
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Dead Space Unit   Poor perfusion=horrible blood flow to pick up O2, nothing wrong w/ lungs, ex. PE, decreased CO  
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PFT's   Based on age, height, wt, sex, monitor the course of pulmonary disease, evaluate meds, determine need for mechanical ventilation  
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Pre-Procedure For PFT   No..tobacco,bronchiodilators,heavy meal,sedatives,narcotics,distended abdomen. Instruct pt how to breathe for test= nose clip,tight seal  
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PFT Results   *FeV1= Low in COPD *VC= vital capacity *RV= Increased w/ COPD, the air left in the lungs after expiration *Tidal Volume= How much air w/ each breath the pt. draws in  
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Post PFT   Maintain airway, prevent injury-may be dizzy  
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Sputum Specimines   Gm stain= Identifies Gm + or - organisms, if + need C&S to identify best ATB therapy (lower the # the better) Acid-fast smear= TB identification. Cytology= looking @ cells and identifying maligancies  
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Pre Bronchoscopy procedure   Informed consent, clarify info, NPO, baseline VS, remove dentures/partials, sedative/conscious sedation, sterile set up  
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Post Bronchoscopy Procedure   NPO until gag reflex returns= cranial nerve #9, VS q hr, discourage coughing, smoking, talking for several hrs, expect hoarse voice, low fever common 1st 24 hrs, designated driver, notify dr. persistent cough, bloody, purulent sputum  
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Bronchoalveolar Lavage (BAL)   Diagnose pneumocystic pneumonia  
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Mediastinoscopy   Incisions for lymph node biopsy  
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Methemo-Globinemia   Benzocaine anesthestic spray- sx usually w/in 1st hr= central cyanosis, O2 ineffection, choclate brown blood, impairs Hgb to carry O2 (functional anemia)Allows CO2 binding, need co-oximeter, Antidote= Methyl Blue  
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Thoracentesis   Lg bore needle through chest wall, position client in supine or sitting postion, affected lung accessible  
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Low Flow O2 systems   NC: 24-45% Simple face mask: 40-60% Humidification needed at >4L  
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High Flow O2 systems   Venturi mask: 24-50% Partial/Non rebreather: Up to 100% New O2 cannulas: Up to 15L Heliox  
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Difference Between O2 Flow And O2 Concentration?   Flow= Force (# of liters) Concentration= Percent  
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Oral Airway   Holds tongue away from pharynx, insert with tip pointed upward then rotate  
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Advantages And Disadvantages To Oral Airway   Advantages=easy to insert, preserves airway. Disadvantages=easy to dislodge,unconscious pts,no use in facial/oral surgery  
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Endotracheak Tubes (ET)   Mechanical ventilation up to 100% O2, can be inserted oral or nasal  
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Oral vs. Nasal ET   Oral= Rapid, easier to insert-chip teeth, need bite block, excess salivation. Nasal= More comfortable, more secure-more resistence  
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Indications for ETT & Mechanical Ventilation   Respiratory failure- pH <7.25 CO2>50% O2<50%  
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Nursing Role For Insertion Of ET   Only nurses with training can perform, RN-sets up equpiment, verify placement, check cuff leaks, monitoring location, care vent. settings, teach conscious pt. about gagging/feelings of suffocation  
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Function Of Cuff On ET Tube   Keep secretions from going into lungs/keep O2 from escaping back upwards  
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Rapid Sequence Intubation (RSI)   *Fast acting narcotic ex. Fentyl *Sedative ex. Versed *Paralytic agent ex. curare derivative. **Do not use in narcotic overdose or code**  
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Procedure for RSI   Gather supplies. test cuff inflation, supine position/head extended "sniffing position" Nasal insertion=greater resp. effort  
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Correct ET location   3-5cm above carina (bifurcation of mainstem bronchus) most likely to be accidently intubated in right mainstem  
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Verifying Placement Of ET   #1 CO2 indicator #2 Auscultate breath sounds bilaterally #3 Observe symmetric chest movement #4 Feels warm/exhaled air at end of tube #5 Confirm by chest x-ray  
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Quick Check Verification For ET   "Lip Line" Q-24 hrs, women 21cm, men 23cm, move side to side but not up and down, x-ray is absolute look!  
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O2 AND Ventilation   PaCO2=best indicator of hypo/hyper ventilation  
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Complications of ET   Aspiration and unplanned extubation **Assess, stay with pt. put on call light, airway assistance- bag or NC**  
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Maintaining Tube Patency   Suction PRN NOT routinely, hyper-oxygenate pre and post, hydration not NS spritzer, suction >120=mucosal damage  
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Closed Suction Technique   Useful for PEEP >7-8cmH20,secretions,freq. suctioning need,unstable  
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Nursing Dx   Impaired gas exchange & Decreased cardiac output  
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Negative Pressure Vent   Negative pressure on chect, pulls air into lung, Pneumowrap=Poncho fits over body & creates a vacuum which expands thoracic cage.  
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Positive Pressure Ventilator (PPV)   Inflates aveoli, must be intubated or trach, creates decreased cardiac output  
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Pressure Cycled Ventilators   Preset pressure, if increased airway resistance or decreased compliance=tidal volume may not be delivered  
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Volume Cycled Ventilators   Preset volume, tidal volume delivered regardless of resistance or compliance, more frequently used  
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Tidal Volume (TV or VT)   Amt. of air delivered w/ each ventilator breath, 7mL/kg of body weight  
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FiO2   21-100% ex. ARDS, Can have O2 toxicity (lost nitrogen) or absorbtion atelectasis (alveolar collapse)  
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Continuous Mandatory Ventilation (CMV)   Vent only delivers TV & breaths per min. that have been set, total control for pt. by decreasing work of breathing, but can cause muscle atrophy=harder to wean  
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Assist/Control Ventilation (ACV)   Senses & controls volume for each present breath, moderate control of pt. NOT for use in COPD  
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Synchronized Intermittent Mandatory Ventilation (SIMV)   Least control=used for weaning, pt. can take own additional breaths @ own TV, vent. rate gradually decreased, will not breathe as pt. breaths on their own  
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Continuous Positive Airway Pressure (CPAP)   + pressure applied via facemaskor ET helps maintain open airways and aveoli >5cm Used mostly for weaning and sleep apnea  
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Positive End Expiratory Pressure (PEEP)   3-5cm, increases amt. of air remaining in the lungs during expiratory phase, allows for reduction of FiO2  
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Peak Inspiratory Pressure   Amt. of pressure required to deliver TV, Plateau pressure: 30cm H20 or less, if increased=bad, decreased=good  
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Low Pressure Alarm   Little/No pressure generated duringdelivery of machine breaths**Check for disconnection, placement, cuff leak/tear  
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High Pressure Alarm   Pre-set peak inspiratory pressure limit is reached b4 venthas delivered set TV, Fix the problem! Check for obstruction, placement..  
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If In Distress With An Alarming Vent..   Take care of client first!! Manually vent pt. and have someone else fix vent  
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Pressure Support   5-10cm, pt. receives increased TV  
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Sighs   Prevents microatelectasis, pulses of air delivered at 100-300x per min. or 1-5mL/kg, high frequency but low pressure  
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Hypoventilation   Acidosis  
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Hyperventilation   Alkalosis, check for arrythmias/calcium imbalances, may be caused by overventilation/overinflation  
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V/Q Mismatch   Uneven blood flow in lungs  
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Decreased CO   + pressure in thorax instead of - pressure, decreased CO=decreased BP, increased HR, vasoconstriction. May require Dopamine to increase BP  
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Volu or Barotrauma   Assess crepitus  
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O2 Toxicity   Exposed to >50% FiO2, exposure time and concentration  
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Pressure Necrosis R/T Tubings   Reposition side to sidein mouth, release cuff pressures via protocol  
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Pneumothorax   May be caused by high vent pressure  
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When to draw ABGs   Anytime a vent setting is changed**Wait 15-30 minutes after vent setting change  
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Nursing Assessments   Breath sounds, VS, chest movement, need for suctioning, hypoxia/hypercapnea, vent settings, pule ox, PEEP (decreased CO) skin, stress ulcers, joint mobility, nutrition, bowels, emotions  
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Nursing Implementations   Humidified/warmed air, HOB 30 degrees, position change, suction, ABGs, keep vent alarms on, correst bucking, decrease anxiety, respect sleep/wake cycles, high calorie need  
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Weaning Phase #1 Pre-Weaning   Assess resp. and non-resp. factors, spontaneous breathing trial  
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Weaning Phase #2 Weaning Process   *Short term pt-linear *Long term pt. peaks & valleys *Psych support for pt. and family  
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Weaning Phase #3 Outcome   Continue O2 therapy, oral care, monitor VS & resp. distress, ABGs within 1 hr  
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Ready To Wean?   Stabilized condition, stable chest wall, resp rate 12-20, PEEP <5cms, good nutrition, improved vital copacity, stong cough, no accessory muscle use, lungs clear, ABG WNL, adequate CO, afebrile  
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Weaning Guidelines   Trust=essential, initiate in the AM not PM, watch for compromise (HR >20bpm from baseline, BP changes, RR changes, ABG deterioration) put back on vent on original settings  
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Spontaneous Breathing Trial   30 min-2 hrs, increasing time, stop before fatigue  
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T-Piece   Remove pt. from vent, place on t-piece which delivers specified FiO2 back on vent then gradually increase time (10 min or so) off until pt. breaths spontaneously for prolonged period  
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SIMV   Gradually decrease # of breaths that machine delivers until pt. breathing totally on own. Vent is there for support, do not need to return to original vent settings  
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CPAP   Vent in CPAP mode, used commonly and provides monitoring that t-piece doesn't  
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