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Vocabulary

Quiz yourself by thinking what should be in each of the black spaces below before clicking on it to display the answer.
        Help!  

Question
Answer
Tardative dyskinsia   Involuntary movement of jaw - call doctor immediately or could become permanent  
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CTZ   Chemoreceptor trigger zone  
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Serotonin Agents   Ondanestron (Zofran)  
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Serotonin Action   Blocks the serotonin receptor in our brain that makes us feel nauseous when it's stimulated  
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Metoclopramide (Reglan)   Dopamine & Prokinetic  
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Dopamine Antagonists   Prochlorperazine (Compazine) Promethazine (Phenergan) Metoclopramide (Reglan)  
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Dopamine Action   Blocks the dopamin receptor in our brain that makes us feel nauseous when it's stimulated  
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Ondanestron   Zofran  
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Metoclopramide   Reglan  
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Promethazine   Phenergan  
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Prochlorperazine   Compazine  
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Prokinetic   Metoclopramide (Reglan)  
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Prokinetic Action   Dopamine blocker Increase tone of lower esophageal sphincter Increase peristalsis  
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Serotonin Admin   PO 30 - 40 mins. before Surgery or expected nausea  
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Serotonin Indications   Nausea r/t pregnancy, chemo, cancer  
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Serotonin Side Effects   Headache, Dizziness, Diarrhea  
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Serotonin Teaching   No driving bc sedation  
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Dopamine Indications   Prevents Nausea  
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Dopamine Admin   PO, IV (slow-over 15 mins.), Rectal, No IV Push  
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Dopamine Side Effects   Muscle spasms, tremors, rigidity Hypotension Sedation Dry mouth, urinary retention, constipation  
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Dopamine Teaching   Encourage fluids Make Pt. aware of side effects  
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Prokinetic Indications   Nausea, Vomitting, Pt's w/reflux (moves things through quickly)  
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Prokinetic Admin   IV or PO, usually 30 mins before meals  
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Prokinetic Side Effects   Sedation, diarrhea, dry mouth, Tardative dyskinesia  
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Prokinetic Teaching   No alcohol or driving bc of sedation Monitor BP & hypotension  
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Antihistamines   Dimenhydrinate (Dramamine)  
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Dimenhydrinate   Dramamine  
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Antihistamine Action   Blocks histamine receptors in the inner ear and CTZ - Prevents Motion Sickness  
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Antihistamine Indications   Motion sickness  
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Antihistamine Admin   PO, IV, IM, Rectal Give 30 mins prior to activity that causes sickness  
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Antihistamine Side Effects   Sedation, Drowsiness, Dizziness, Dry mouth, constipation, Urinary retention  
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Antihistamine Teaching   Teach about side effects  
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Antiemetics   Seotonin Antagonists Dopamine Antagonists Prokinetic Antihistamines  
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Seotonin Antagonists Dopamine Antagonists Prokinetic Antihistamines   Antiemetics  
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Histamine2 Receptor Agents   Cimetidine (Tagamet) Famotidine (Pepcid) Ranitidine (Zantac)  
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Histamine2 Receptor Agents Action   Block histamine receptors in stomach to decrease acid in stomach (doesn't prevent reflux but increases pH)  
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Histamine2 Receptor Agents Indications   Reflux, Gastic & duodenum ulcers  
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Histamine2 Receptor Agents Admin   Admin w/or w/o food, don't admin antacids w/in 1 hr. of giving antagonist (effects absorption rate) IV must be given slowly or can cause bradycardia  
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Histamine2 Receptor Agents Side Effects   Nausea, vomiting, diarrhea, constipation Decreased WBC's (long term use) Tagamet: Seizures, Lower labido, Lethargy  
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Histamine2 Receptor Agents Teaching   Antacids affect absorption rate of Antagonist, Smoking also interferes with absorption  
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Cimetidine   Tagamet  
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Ranitidine   Zantac  
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Famotidine   Pepcid  
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Proton Pump Inhibitors (PPIs)   Omeprazole (Prilosec) Lansoprazole (Prevacid) Pantoprazole (Protonix) Esomeprazole (Nexium)  
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PPI Action   Decreases the acids in the stomach  
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PPI Indications   Reflux, duodenum & gastic ulcers  
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PPI Admin   Empty stomach, usually 1x daily before breakfast  
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PPI Side Effects   Nausea, vomiting, diarrhea Long term use = bone loss  
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PPI Teaching   Antacids do not affect absorption ^ Vit D ^ Ca+ ^ Exercise  
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Omeprazole   Prilosec  
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Lansoprazole   Prevacid  
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Pantoprazole   Protonix  
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Esomeprazole   Nexium  
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Mucosal Protectants   Sucralfate (Carafate)  
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Sucralfate   Carafate  
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Mucosal Protectants Action   Creates gel to coat stomach (good w/pt's already have ulcer)  
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Mucosal Protectants Indications   Pt.'s w/ Ulcers  
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Mucosal Protectants Admin   Empty stomach - wait 2hrs before/after meals or other meds - affects absorption  
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Mucosal Protectants Side Effects   Constipation, nausea, diarrhea, dyspepsia  
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Mucosal Protectants Teaching   Teach pt how to take  
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Antacids   Aluminum hydroxide (Amphojel) Calcium carbonate (Tums) Magnesium hydroxide (Milk of Magnesia)  
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Aluminum hydroxide   Amphojel  
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Calcium carbonate   Tums  
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Magnesium hydroxide   Milk of Magnesia  
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Aluminum hydroxide Action   Neutralize gastric acid  
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Aluminum hydroxide Indications   Ulcers and reflux  
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Aluminum hydroxide Admin   PO  
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Aluminum hydroxide Side Effects   Amphojel & Tums: Constipation Milk of Magnesia: Diarrhea  
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Aluminum hydroxide Teaching   Know what other medications are being taken  
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Decreased Blood flow + Increased stomach acid =   Perfect environment for an ulcer  
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Prostaglandin E Analogs   Misoprostol (Cytotec)  
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Misoprostol   Cytotec  
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Prostaglandin E Analogs Action   Works to increase pH, protect against ulcer, allow adequate blood flow to the gastric wall  
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Prostaglandin E Analogs Indications   Prevents gastric ulcers from long-term NSAID use  
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Prostaglandin E Analogs Admin   Take w/meals while taking NSAID  
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Prostaglandin E Analogs Side Effects   Diarrhea, Ab pain, nausea Women: uterine cramps, spotting  
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Prostaglandin E Analogs Teaching   Do not take if pregnant - spontaneous abortion  
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What drug can cause a spontaneous abortion in women if taken during pregnancy?   Misoprostol  
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Cancer of Oral Cavity Risk Factors   Alcohol/smoking Greater in Males >50yrs  
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Cancer of Oral Cavity Pathophysiology   Lips, Tongue, Floor of mouth  
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Cancer of Oral Cavity Clin. Manifestations   Painless lesion that doesn't heal  
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Cancer of Oral Cavity Diagnostic Tests   Biopsy to confirm  
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Cancer of Oral Cavity Treatment   Depends on type/extent  
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Cancer of Oral Cavity #1 concern   Airway  
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A stroke is the   3rd leading cause of death in USA  
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Up to 80% of strokes are   preventable  
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Strokes are the leading cause of   long-term disability  
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_______ are more likely to have a stroke.   Men  
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_______ are more likely to die from a stroke.   Women  
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Cerebrovascular Disorder   Any interruption of blood supply to brain causing CNS Functional abnormality  
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Stroke   O2 supply to the brain is disrupted  
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Stroke has 4x higher incidence in   left hemi-sphere  
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"STR"   Can a person... Speak simple sentence? Talk? Raise both arms?  
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"F A S T"   Facial Arms Speech Time  
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Neuroplasticity   Neuron's ability to rearrange connections  
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Brain adaption   brain saturated w/blood or deprived of O2 = find alt. pathways  
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Transient Ishemic Attack (TIA's)   Cerebral ischemic event lasting < 24 hrs May even last just a few minutes No permanent neurological chgs.  
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Evolving Stroke   Worsening of deficits over minutes Suggests widening area of ischemia  
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Completed Stroke   Acute cerebral ischemic event w/deficits  
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#1 risk factor for stroke   Hypertension  
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TIA aka   "warning strokes"  
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Blood clot temporarily clogs artery so part of brain doesn't get blood it needs   TIA  
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S/S of TIA   Similar to stroke, usually last 1 or 2 mins.  
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Ischemic Stroke   Obstruction of cerebral blood flow  
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88% of all strokes are   Ischemic Stroke  
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"Worst headache of my life"   Subarachnoid hemorrhage  
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Receptive Aphasia   Person doesn't know what's going on  
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Expressive Aphasia   Person knows what's going on, but can't express  
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Broca's Area located in   Left hemisphere  
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Controls tongue, lips, vocal cords & ability to phonate (form words)   Broca's Area  
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Global Aphasia   Expressive & receptive aphasia  
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Dysphagia   Difficulty swallowing  
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Dysarthia   Impaired speech  
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Ataxia   Gait disturbance  
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Nystagmus   Rapid Eye Movements  
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Hemianesthesia   Pain on one half of body that doesn't completely register  
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Wernicke's Area   Can be in whichever hemisphere is dominant for pt.  
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Recalls, recognizes & interprets words & other sounds in the process of "phonating"   Wernicke's Area  
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Dmg. to Wernicke's Area (Temporal lobe of dominant hemisphere)   Inability to comprehend spoken or written words  
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If a Pt. is newly diagnosed with Afib, do TEE test to see if   There are clots behind the heart  
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Trans-esophageal echocardiogram   TEE test  
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Tissue Plasminogen Activator (tPA)   Time of onset is known and < 3 hrs  
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Pt not a candidate for tPA if   Pt is rcvg IV Heparin  
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Angioplasty   Any endovascular procedure to reopen narrowed blood vessels & restore forward blood flow  
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Atherectomy   Opens occluded, scarred or calcified vessels by removing atherosclerotic plaques with rapidly rotating drills  
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Balloon angioplasty   Relies on the inflation of high-pressure balloons within blocked arteries to force open  
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Laser & radiofrequency waves   Vaporize or ablate atheroscleroptic plaques  
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Endovascular stents   Hold vessels open with expandable lattices inserted across narrowed section of the artery  
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Cerebral Angioplasty   Coils surgically placed in cerebral vascular malformations to assure patency and prevent rupture (Aneurysm)  
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Hemorrhagic Strokes   Vessel ruptures & bleeds into brain or spaces around brain  
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Intracerebral Hemorrhage   Always hypertension related  
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Craniectomy   Removal of part of the brain  
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Craniotomy   Redirection - keep from getting to brain stem  
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Subarachnoid Hemorrhage   Bleeding into subarachnoid space  
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Arteriovenous Malformation   A tangle of blood vessels in the brain or on the surface of brain that bypasses norm brain tissue and diverts blood from arteries to the veins  
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Mandibular Fractures Clin. Manifestations   Dull ache -> throbbing pain Restricted jaw movement  
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Mandibular Fractures Diagnostic   X-ray or MRI  
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Mandibular Fractures Treatment   Rigid plate fixation Bone grafting  
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Mandibular Fractures Nurse Mngmnt.   #1 Airway #2 Nutrition  
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Absent/ineffective peristalsis of distal esophagus Failure of esophageal sphincter to relax in response to swallowing   Achalasia  
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Achalasia   Food cannot get into the stomach  
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Achalasia Diet   Small frequent meals/soft/encourage fluids  
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Achalasia Meds   Ca+ channel blockers relax sphincters  
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Achalasia Pneumatic dilation   Balloon to try and open sphincter up  
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Achalasia Esophagomyotomy   Enter esophagus & cut muscle fibers  
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90% of Hiatal Hernias are   Sliding esophageal hernias  
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sliding Hernia Manifestations   Heartburn Regurgitation Dysphasia Belching Reflux*  
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Reflux Occurs w/sliding hernia because   Sphincter is NOT intact - food easily regurgitates into esophagus.  
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Paraesophageal manifestations   Sense of fullness after eating Indigestion May be a symptomatic  
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The sphincter in a paraesophageal hernia   Is still intact.  
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Hiatal hernia complications   Strangulation  
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Gastroesophageal Reflux Disease (GERD)   Backflow of gastric contents into esophagus  
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Avoid these if you have GERD   High-fat diet Caffeine, tobacco, alcohol, spearmint, peppermint, carbonated bevs Eating 2 hrs before bed Tight clothing  
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Meds for GERD   Antacids Histamine Receptor Blockers PPIs Prokinetic Agents  
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Treatment of GERD   Nissen fundoplication  
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Nissen fundoplication   Surgery to wrap stomach around esophagus = No reflux  
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Barrett's Esophagus   Chgs cells in esophagus bc constant burning of lining from reflux -> pre-cancerous condition  
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S/S Barrett's Esophagus   Same as GERD -> ppl usually don't know it's happening  
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Barrett's Esophagus Diagnosis   Endoscopy  
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Barrett's Esophagus Treatment   Minor cell chgs Photodynamic therapy (PDT) Prohylactic esophagectomy  
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Photodynamic therapy (PDT)   Take out piece of esophagus & replace w/jejunum or part of stomach  
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Risk factors for Cancer of Esophagus   Barrett's Alcohol Tobacco 3x more common in men  
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Cancer of Esophagus Mgmnt   Radiation Chemotherapy Surgery (Esophagectomy)  
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Gastritis   Inflamation of the stomach  
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Gastritis Patho   Lining can become swollen & ulcerate Can cause perferation of lining = hemorrhage  
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Acute Gastritis Clin. Manifest   Abdominal discomfort Nausea Vomiting  
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Chronic Gastritis Clin. Manifest   Abdominal discomfort Nausea Vomiting Heart burn Pain  
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Chronic Gastritis   Find the trigger  
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The most common location for ulcers is   the duodenum.  
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Peptic Ulcer Disease   Increased acid & decreased mucus allow acid to erode mucosal wall  
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Peptic Ulcer Disease occurs in the   Stomach Pylorus Duodenum Esophagus  
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Increase gastric acid production   Caffeine Smoking Alcohol Stress  
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H. pylori   Bact will burrow into the lining allowing acid in to create an ulcer  
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H. pylori requires   2 different Ab's to cure  
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Intractable Ulcers   Incurable Ulcers - (perform Vagotomy)  
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Vagotomy   Cut Vagus nerve to decrease production of stomach acid - (incurable ulcers)  
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Antrectomy - Billroth I   Take out lower portion of stomach to decrease stomach acid  
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"Coffee Grounds" in stomach/vomit   Hemorrhage  
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Peritinitis   Stomach leak out causing bacterial infection - sepsis  
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Perforation complications can lead to   Peritinitis  
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Any leakage into the abdominal cavity...   Requires Ab's  
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Weight indications for surgery   BMI >40 or over 100 lbs overweight  
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3 Categories of Bariatric Procedures   Malabsorptive Restrictive Combined Malabsorptive & Restrictive  
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Rstrictive Surgery:   Reduces food intake Decrease in appetite Early satiety Control of stoma Craving control (Ghrelin)  
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Laparoscopic Gastric Banding   Inflatable silicone band completely around upper portion of stomach  
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Vertical Sleeve Gastrectomy (VSG)   85% of stomach removed - shaped like small banana  
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Gastric Bypass   Limits size of stomach and bypasses some of the small intestine  
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Secretory Diarrhea   Bowel doesn't reabsorb water - it excretes water  
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Osmotic Diarrhea   Pulling of water into the bowel  
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Infectious Diarrhea   C-diff  
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Antidiarrheals   Loperamide (Imodium) Diphenoxylate (Lomotil)  
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Loperamide   Imodium  
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Diphenoxylate   Lomotil  
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Constipation   Abnormal infrequency or irregularity of defications Abnormal hardening of stools Retention of stool in rectum  
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Chronic Constipation   <3 bowel movements/week for 12 weeks in a 12 month period  
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Bulk forming Laxatives   Psyllium (Metamucil) Methylcellulose (Citrucel)  
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Psyllium   Metamucil  
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Methylcellulose   Citrucel  
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Bulk forming Laxative Action   Promotes large soft stools by absorbing water into the intestines Stimulates peristalsis  
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Lubricant Laxatives   Mineral oil Glycerin suppository  
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Lubricant Laxative Action   Softens fecal matter and lubricates intestinal tract  
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Stimulant Laxatives   Bisacodyl (Dulcolax) Senna (Senokot)  
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Bisacodyl   Dulcolax  
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Senna   Senokot  
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Stimulant Laxative Action   Irritates colonic epithelium by stimulating sensory nerve ending and increasing peristalsis  
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Fecal softener Laxatives   Docusate (Colace)  
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Docusate   Colace  
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Fecal softener Laxative Action   Promotes incorporation of water into the stool resulting in softer fecal mass More of daily treatment 1-5 days to take effect  
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Osmotic Agent Laxatives   Polyethylene glycol and electrolytes  
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Osmotic Agent Laxative Action   Rapidly pulls H2O into colon -> diarrhea out  
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Common Hernia Locations   Inguinal Femoral Incisional Umbilical  
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Volvulus - Mech. Obstruction   Twisting of the bowel - caused by congenital abnormality  
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Intussusception - Mech. Obstruction   Tunneling of bowel (folds in on itself)  
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Paralytic illeus - Functional obstruction   No blockage, lack of movement (caused by surgery)  
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Diverticulum   Sac like pouches -> sigmoid colon, usually  
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Diverticulosis   Multiple Diverticulum  
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Diverticulitis   Food or infection stuck in diverticula & cause inflammation  
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Diverticular Disease   Submucosal layers of the colon herniate through the muscular wall  
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Diverticular Disease Complications   Peritonitis Abscess formation Fistulas Bleeding  
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End-to-end anastomosis   Cut out bad bowel & reconnect  
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Appendicitis   Food or infection gets lodged in the appendix  
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Diarrhea is common in   Chrohn's Disease  
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In Chrohn's Disease, constipation is   Occasional  
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Blood in stool in Chrohn's Disease is   Occult Common  
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Abdominal pain in Chrohn's Disease is   Typically mid-abdomen and right lower quadrant pain  
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Fever in Chrohn's Disease is   Common  
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Weight loss/growth failure in Chrohn's Disease is   Common May occur for many months to years before diagnosis  
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Perianal disease in Chrohn's Disease is   Common for: Fistula/fissures Abscesses/skin tags  
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Diarrhea in Ulcerative Colitis is   Common, often with urgency  
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Constipation in Ulcerative Colitis is   Rare without obstruction  
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Blood in stool in Ulcerative Colitis is   Very common  
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Abdominal pain in Ulcerative Colitis is   Cramping pain, often with stools  
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Fever in Ulcerative Colitis is   Rare  
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Weight loss/growth failure in Ulcerative Colitis is   Rare  
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It is rare to have Perianal disease with   Ulcerative Colitis  
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Chrohn's disease...   often recurs after surgery  
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Ulcerative colitis...   is curable with a colectomy  
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Right hemisphere   right now, present pictures, sound, sight, smell  
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Left hemisphere   past, future associate present with past to prepare future Broca's area - speech center  
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Broca's area   Speech center, left hemisphere  
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Magnetic Resonance Imaging   MRI  
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Second largest part of brain Balance and coordination, assists in controlling body movements   Cerebellum  
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Largest most advanced part of brain higher intellect, speech, movement, emotion, and integration or sensory stimuli   Cerbrum  
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Handles automatic functions Respiration, heart rate, BP, arousal, wakefulness & attention   Brain Stem  
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Artistic, creative, intuitive   Right hemisphere  
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Logic, reasoning, linear   Left hemisphere  
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Cerebral edema   any swelling of the brain  
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Frontal lobes   Voluntary muscle movements  
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Parietal lobes   regulate taste and touch  
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Temporal lobes   regulate hearing & smell  
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Occipital lobes   regulate vision  
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3 ways Neurological Changes may affect the body   Structural Metabolic Drug Related  
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Cardiovascular Respiratory Infection   3 ways Neurological Changes may affect the body  
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AEIOU Tips = A   acidosis, alcohol  
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AEIOU Tips = E   epilepsy  
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AEIOU Tips = I   infection  
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AEIOU Tips = O   overdose  
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AEIOU Tips = U   uremia  
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AEIOU Tips = T   trauma  
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AEIOU Tips = i   insulin  
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AEIOU Tips = p   psychogenic  
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AEIOU Tips = s   stroke  
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Often, the first sign that a neurological event is already in progress   Restlessness  
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Progressive dilation of pupils   increasing ICP  
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Fixed pupils   very serious injury at midbrain or lower  
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Corneal reflex is   unilateral or absent in coma  
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Cheyne-stokes respirations mean that   lesion or injury is deep within cerebral hemispheres (death is usually imminent)  
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CT Scan, MRI   Show displacement of brain structures  
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EEG   studies brain's electrical activity  
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Cerebral angiography   detects aneurysms, occluded vessels & tumors w/in cerebral arts/veins  
🗑
Transcranial Doppler   assesses cerebral blood flow  
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Lumbar puncture   CSF detects blood, WBCs, meningitis  
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The 2 essential things the brain needs to survive are   O2 & glucose  
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Brain   80% of skull's contents  
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CSF   10% of skull's contents  
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Blood   10% of skull's contents  
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Hypercapnia   Increased cerebral vasodilation then increased blood flow then increased ICP  
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Hypoxemia   Cerebral vasodilation, which raises ICP  
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Vasodilator drugs   increase cerebral blood flow -> temp increased ICP (Nitroglycerin)  
🗑
Cerebral Perfusion Pressure   Pressure needed to perfuse brain cells  
🗑
Normal CPP   70-100 mm Hg  
🗑
CPP < 50   irreversible brain dmg  
🗑


   

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