grcc bi240 immunity
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| What is the early warning system of infection? | inflammation- redness, swelling, fever, pain.
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| What are the mediators of acute inflammation | mast cells and basophils
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| histamine is released from mast cells | increases permeability of capillaries
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| capillaries become leaky causing | accumulation of complement, enzymes and phagocytes at sites of infection.
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| Pathogens are eliminated and healing is initiated | via phagocytosis, etc
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| Complement components is activated and induce | acute inflammation
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| Complement C system triggers | histamine release from mast cells, increases vascular permeability and induces chemotaxis which triggers phagocytes towards sites of infection.
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| macrophages, neutrophils & eosinophils are now known as | mediators of acute inflammation.
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| Macrophages, neutrophils, eosinophils secrete | chemicals that are proinflammatory cytokines triggering increased vascular permeability to maintain acute inflammation.
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| phagocytosis process begins where | macrophages, dendritic cells, and polymorphonuclear leukocytes (PMN)
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| Phagocytosis engulfs | and destroys particles that don't belong in the area.
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| phagocytosis: adhesion | attachment or binding of phagocyte and pathogen using noncovalent interactions.
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| Phagocytosis: oponization | enhancement of phagocytosis due to better binding medated by complement or antibody receptors on the phagocytes surfaces.
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| ingestion (engulfment) cells take in via | pseudopodia and phagosomes
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| pseudopodia | surround the pathogen, then fuse to form a vacuole called a phagosome
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| Phagosomes are converted into phagolysosomes by | fusion with lysosomes (vacuoles containing a broad spectrum of digestive enzymes) and then break down.
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| Ingestion of bacterium happen via phagosomes as they are converted to | phagolysosomes that contain digestive enzymes that help with breakdown of bacteria.
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