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Motor Speech Disorders (Lecture 5)

Quiz yourself by thinking what should be in each of the black spaces below before clicking on it to display the answer.
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Question
Answer
Cognitive-Linguistic Processes   show
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Sensorimotor Planning/Programming   show
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show Direct activation of motoneurons,Muscle contraction, Movement, SPEECH!!!!  
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show Aphasia: Defect of Language Generation, Word-finding problems, Agrammatism, Neologisms, Auditory comprehension deficits, Reading and writing deficits  
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show Apraxia of speech, Difficulty sequencing syllables, Articulatory groping, “know what you want to say but can’t make mouth say it”, No weakness or paralysis  
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Neuromotor Execution Defects   show
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show neurologic lesion of CNS or PNS, movement disorders classified based on how they sound; and how they sound corresponds with underlying neuropathology  
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show Impairment of sensorimotor programming, not attributable to muscular weakness or language difficulties  
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Flaccid Dysarthria   show
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show Breathiness, Hypernasality, Imprecise articulation, Nasal air emission, monopitch  
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Conditions associated with Flaccid Dysarthria   show
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show inability to make a labial seal or elevate tongue for lingual sounds  
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Spastic Dysarthria   show
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Spastic Dysarthria Etiologies include   show
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show Imprecise consonants, Strained-strangled voice (vocal stenosis), Slow rate, Monopitch and loudness, Hypernasality, Pitch breaks  
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show Lesion to cerebellum or cerebellar circuits; Several possible patterns of deficit including “robotic quality” and “drunken quality”  
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show neoplasms, progressive cerebellar degeneration, trauma, encephalitis, MS, ETOH toxicity, CVA, congenital conditions  
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show Imprecise consonants, Robotic quality (equal and even stress), Irregular articulatory breakdown (drunken quality), Harsh voice  
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Hypokinetic Dysarthria   show
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show Parkinson’s disease (idiopathic PD), and parkinsonism due to drugs, CVAs or other disease processes  
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Clinical Presentation of Hypokinetic Dysarthria:   show
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show Lesions to the basal ganglia that result in too little inhibition (too much facilitation); Slow hyperkinesia, with a “build up” of involuntary movement activity  
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Hyperkinetic Dysarthria (Dystonia) Etiologies include:   show
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show Imprecise consonants, Harsh voice, Strained-strangled voice, Monopitch and loudness, Vowel and consonant breakdowns  
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show BG lesion causing too much facilitation; Quick hyperkinesia results in irregular, unsustained, random, unpatterned, rapid movements  
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show Huntington’s, Sydenham’s chorea, encephalitis, Gilles de la Tourette syndrome  
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Clinical Presentation of Hyperkinetic (chorea) Dysarthria:   show
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Flaccid dysarthria, possible site of lesion   show
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show Facial resulting in slurred speech  
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Flaccid dysarthria, possible site of lesion   show
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Flaccid dysarthria, possible site of lesion   show
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Flaccid dysarthria, possible site of lesion   show
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show UUMN lesion; weakness, incoordination, usually mild and transient to bilateral innervation  
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show imprecise articulation only sometimes present  
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show Lesion to cortical tissue (maybe insula), Sensorimotor planning/programming deficit  
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Apraxia of Speech Etiologies include   show
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show articulatory errors (perseverative, anticipatory, metathetic errors), articulatory groping, slowness, dysprosody, automatic speeech, inability to program sequences of sounds (especially consonants)  
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