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N170 Respiratory Study Aid

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Question
Answer
orthopnea   abnormal condition in which a person must use several pillows when lying down  
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atelectasis   Collapse of the alveoli that prevents normal resp. exchange of oxygen and CO2  
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hypoxia   inadequate tissue oxygenation at the cellular level  
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chest physiotherapy   A group of therapies used in combination to mobilize pulmonary secretions  
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postural drainage   positioning techniques that draw secretions from specific segments of lungs and bronchi into the trachea:  
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pneumothorax   The collection of air in the pleural space  
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hemothorax   An accumulation of blood and fluid in the pleural cavity between the parietal and visceral pleurae, usually a result of trauma:  
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pursed-lip breathing   This involves deep inspiration and prolonged expiration through pursed lips to prevent alveolar collapse  
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diaphragmatic breathing   This technique requires the client to relax intercostal and accesory resp. muscles while taking deep inspirations  
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Nasal Cannula: Flow rate and %O2 delivered   24%-44% of concentrated oxygen delivered at 1-6 Liters per minute; Oxygen delivered is not affected by mouth breathing. Oxygen delivered is dependent on amount client respiratory effort and amount of room air inhaled with each breath  
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Simple face mask: Flow rate and %O2 delivered   Low flow oxygen mask - 5-10 liters per minute: delivers 35-50% O2; Used for short term therapy  
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Venturi Mask: Flow rate and %O2 delivered   High flow mask - 24%-60% concentrated oxygen delivered at 4-10 liters per minute. Most precise and consistent mask device.  
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Non rebreather mask: flow rate and % O2 delivered   Low flow oxygen face mask that delivers highest concentration of oxygen. 10 liters per minute; 60-80% oxygen  
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Diffusion   Process for exchange of respiratory gases in the alveoli & the capillaries of the body tissue  
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Partial Rebreather Mask   Low flow oxygen mask with resevoir bag allows mix of room air and O2- 6-10 liters per minute; 40-70% O2  
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Base Excess   indicates the amount of bicarb available in the ECF normal value: +/- 2 mEq/L  
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Hemothorax   An accumulation of blood and fluid in the pleural cavity between the parietal and visceral pleurae, usually a result of trauma  
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Pneumothorax   Collection of air in the pleural space  
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HCO3- (bicarb)   normal 22-26 mEq/L (decreased in acidosis, increased in alkalosis)  
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pH   *negative logarithm of H+ ion concentration in mEq/L (as H+ ion concentration increases, pH decreases) *normal values 7.35 -7.45 (less is acidotic, more is alkalotic)  
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SaO2   the percent of Hb saturated with O2, a calculated value  
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PaO2   amount of oxygen available to bind with hemoglobin, amount of pressure exerted on O2 by plasma  
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BE "base excess"   indicates the amount of bicarb available in the ECF normal value: +/- 2 mEq/L  
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PaCO2   *partial pressure of CO2 *reflects adequacy of alveolar ventilation, regulated by lungs, alterations indicate resp disturbance *normal values 35-45 mmHg (less is alkalotic, more is acidotic)  
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respiratory alkalosis CV signs   tachycardia, palpitations, increased myocardial irritability  
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respiratory alkalosis respiratory signs   rapid shallow breathing (trying to retain CO2, oxygenate), chest tightness  
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Causes of respiratory alkalosis   hyperventilation, sepsis/infection, over ventilation, hepatic cirrhosis  
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respiratory acidosis cardiac signs   hypotension, peripheral vasodilation weak thready pulse, tachycardia, warm flushed skin  
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respiratory acidosis respiratory signs   dyspnea, slow shallow respirations, hypoxia and hypoventilation, cyanosis  
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Causes of respiratory acidosis   respiratory depression/arrest, inadequate chest expansion, airway obstruction, interference with alveolar capillary exchange  
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respiratory acidosis CNS signs   HA, seizures, altered LOC, papilledema, twitching/tremors, drowsy --> coma  
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Respiratory acidosis CNS signs   paresthesia, dizzyness, confusion, tetany, convulsion, numb/tingling, light headed, anxiety/panic, Loss of consciousness, hyperactive reflexes  
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metabolic alkalosis respiratory signs   hypoventilation, respiratory failure  
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metabolic alkalosis CV signs   tachycardia, HTN, PVC, atrial tachycardia, dysthrythmias  
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Causes of metabolic alkalosis   vomiting, NG suctioning, eating bicarb-based antacids, diuretics  
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metabolic acidosis respiratory signs   Kussmaul/deep/rapid respirations, trying to blow off CO2  
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metabolic acidosis CNS signs   HA (from cerebral edema), lethargy, coma, confusion/restless, weakness  
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metabolic alkalosis GI signs   n/v, anorexia, paralitic ileus (hypokalemia)  
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metabolic acidosis GI signs   n/v, diarrhea, abdominal pain  
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Causes of metabolic acidosis   chronic diarrhea, malnutrition, starvation, renal failure, DKA, trauma, shock, sepsis, fever, salicylate toxicity  
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Cheyne Stokes Respiration   Rhythmic waxing and waning of respirations from very deep to very shallow breaths and temporary apnea  
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Kussmaul’s breathing   A form of hyperventilation demonstrated by an Increased rate and depth of respirations associated with metabolic acidosis  
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Stridor   Shrill harsh sound heard during inspiration with laryngeal obstruction  
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CO2   Stimulator of the respiratory center  
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Hypoventilation   occurs when alveolar ventilation is inadequate to meet the body's oxygen demand or to eliminate sufficient CO2  
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Hyperventilation   state of ventilation in excess of that required to eliminate the normal venous CO2 produced by cellular metabolism  
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