CM- Derm -5- bacteria- vesicular/pustular disease
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| what type of infection can bacterial skin infections typically be | primary cutaneous process or
secondary manifestation of infection of some other organ
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| how do you get primary cutaneous infections | break down of skins barriers that allow infection in
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| Superficial bacterial infection of the outer layers of the epidermis generally caused by staph aureous and group A strep | impetigo
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| What are the two types of impetigo | nonbullous and bullous
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| What is the treatment for impetigo | topical bactroban or oral penicillinase resistant antibiotics.
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| What is classic signs and symptoms of impetigo | honey colored crusts which contain bacteria so wipe away and discard
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| bacterial skin disease affects the desmosomes causing wide spread superficial blistering and loss of upper layers of epidermis | Staph scalded skin syndrome
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| What is folliculitis | inflamation of the epithelium of the hair follicle, usually do to ostial occlusion
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| What are the common pathogens of folliculitis | S. aureus, pseudomonas, candida, pityrosporum, tinea, demodex
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| What are the types of folliculitis | superficial, deep, hot tub
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| what is the treatment for folliculitis | topical anitbodies, oral abx, and none depending on how bad it is
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| what are the three types of laymen's boils | furuncle, carbuncle, abcess
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| deep seated inflammatory nodule about a follicle usually developed from folliculitis | Furuncle
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| What is the pathogen and the treatment for a furuncle | Caused by S. Aureus and treated by warm compress or I&D
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| Before perfroming an incision and drainange on a boil what state does it need to be in | it needs to be flucuant before draining
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| Larger deeper more inflammatory nodule with deeper base generally involves multiple follicles | Carbuncle
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| Pathogen that cuases carbuncles and the treatment | Caused by S. Aureus and treatment is I&D w/ systemic antibiotics
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| Painful tender red fluctuant soft tissue mass not associated with a follicle caused by anerobic bacteria | abcess
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| What is the treatment for an abcess | I&D with systemic antibiotics for severe cases
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| this is an abcess that occurs in axilla and anogenital region associated with apocrine follicles and can become chronic and indolent (causing little or no pain) with sub q extensions with induration, sinus, and fistula formation | Hidradenitis Suppurativa
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| When is hidradenitis suppurativa most common | occurs more in women in the 3rd decade of life
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| What is the pathogenesis of hidradenitis suppurativa | kerain comedones-> Occlusion of the aporcrine duct -> superimposed inflammation and infection -> abscess formation -> chronic infection and spread -> induration, and sinus and fistula formation
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| How many stages of hidradenitis suppurativa are there | 3 stages
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| what stage of hidradenitis suppurativa is marked by single or multiple abscesses without sinus tracts or cicatrization | Stage 1 hidradentitis suppurativa
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| What stage of hidradenitis suppurativa is marked by recurrent abscesses with sinus tracts and cicatrization may be a single to multiple wide spread SEPERATED lesions | stage 2 hidradentitis suppurativa
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| What stage of hidradenitis suppurativa is marked by diffuse interconnected tracts and abscesses across the entire area | stage 3 hidradentitis suppurativa
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| what is the treatment for acute and chronic hidradentitis suppurativa | acute- antibiotics 2 weeks with intralesional steroids
Chronic- long term antibiotics, high dose systemics steroids
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| What type of surgery or treatment is suggested for each stage of hidradentitis suppurativa | Stage 1- I&D w/ antibiotics
stage 2- exterirization and laying open of tracts and electrocoagulation, or exicision and primary closure such as pollock procedure
stage 3- total wide excision and healing with secondary intention or flaps and grafts
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| infection fo the epidermis that extends down to the dermis often from neglected impetigo | ecthyma
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| what pathogens cause ecthyma and what is suggested treatment | ecthyma can be caused by group A strep and S. aureus or a combo. Treatment is with oral antibiotics
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| Superficial infection of upper dermis and lymphatics w/ sharply demarcated raised boarders | erysipelas
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| what pathogens can cause erysipelas and what is the treatment | Group A strep and sometime group C or G. treatment anti strep antibiotics
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| how can you distinguish between erysipelas and cellulitis | erysipelas is more erythematous and has a raised boarder
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| Infectious disease of the dermis and sub q tissues without raised boarders | cellulitis
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| What pathogens can cause cellulitis | S. Aureus and Group A strep
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| What is the treatment of cellulitis | local skin care, elevation, and systemic antibiotics
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| common cause of facial cellulitis in kids | H. flu
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| causes cellulitis w/trauma and abcess | S. Aureus
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| cellulitis from a dog or cat bite | pasteurella
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| cellulitis from salt water | vibrio vulnificus
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| cellulitis in fresh water or soil | aromonas hydrophilia
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| skin disease when neisseria meningitides caused bacteremia spreads or manifests at the skin | meningococcemia
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| what are the s/sx of meningococcemia | transient macular eruption, purpura, and purulent petechiae
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| what is the treatment for meningococcemia | get the IV ceftriaxone fast because it is life threatening
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| aggressive cellulitis in superficial fascia and sub q tissues generally external lesions are small compared to the large lesions under the skin very life threatening | Necrotizing fasciitis
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| What are the usual pathogens that cause necrotizing fasciitis | strep pyogenes, enterobacter, bacteroides, vibrio
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| What is the treatment for necrotizing fasciitis | wide local debridment with broad spectrum antibiotics. Look for sub q gas in mri/ct to see how far out to debride
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