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Session 2 CM derm5
CM- Derm -5- bacteria- vesicular/pustular disease
| Question | Answer |
|---|---|
| what type of infection can bacterial skin infections typically be | primary cutaneous process or secondary manifestation of infection of some other organ |
| how do you get primary cutaneous infections | break down of skins barriers that allow infection in |
| Superficial bacterial infection of the outer layers of the epidermis generally caused by staph aureous and group A strep | impetigo |
| What are the two types of impetigo | nonbullous and bullous |
| What is the treatment for impetigo | topical bactroban or oral penicillinase resistant antibiotics. |
| What is classic signs and symptoms of impetigo | honey colored crusts which contain bacteria so wipe away and discard |
| bacterial skin disease affects the desmosomes causing wide spread superficial blistering and loss of upper layers of epidermis | Staph scalded skin syndrome |
| What is folliculitis | inflamation of the epithelium of the hair follicle, usually do to ostial occlusion |
| What are the common pathogens of folliculitis | S. aureus, pseudomonas, candida, pityrosporum, tinea, demodex |
| What are the types of folliculitis | superficial, deep, hot tub |
| what is the treatment for folliculitis | topical anitbodies, oral abx, and none depending on how bad it is |
| what are the three types of laymen's boils | furuncle, carbuncle, abcess |
| deep seated inflammatory nodule about a follicle usually developed from folliculitis | Furuncle |
| What is the pathogen and the treatment for a furuncle | Caused by S. Aureus and treated by warm compress or I&D |
| Before perfroming an incision and drainange on a boil what state does it need to be in | it needs to be flucuant before draining |
| Larger deeper more inflammatory nodule with deeper base generally involves multiple follicles | Carbuncle |
| Pathogen that cuases carbuncles and the treatment | Caused by S. Aureus and treatment is I&D w/ systemic antibiotics |
| Painful tender red fluctuant soft tissue mass not associated with a follicle caused by anerobic bacteria | abcess |
| What is the treatment for an abcess | I&D with systemic antibiotics for severe cases |
| this is an abcess that occurs in axilla and anogenital region associated with apocrine follicles and can become chronic and indolent (causing little or no pain) with sub q extensions with induration, sinus, and fistula formation | Hidradenitis Suppurativa |
| When is hidradenitis suppurativa most common | occurs more in women in the 3rd decade of life |
| What is the pathogenesis of hidradenitis suppurativa | kerain comedones-> Occlusion of the aporcrine duct -> superimposed inflammation and infection -> abscess formation -> chronic infection and spread -> induration, and sinus and fistula formation |
| How many stages of hidradenitis suppurativa are there | 3 stages |
| what stage of hidradenitis suppurativa is marked by single or multiple abscesses without sinus tracts or cicatrization | Stage 1 hidradentitis suppurativa |
| What stage of hidradenitis suppurativa is marked by recurrent abscesses with sinus tracts and cicatrization may be a single to multiple wide spread SEPERATED lesions | stage 2 hidradentitis suppurativa |
| What stage of hidradenitis suppurativa is marked by diffuse interconnected tracts and abscesses across the entire area | stage 3 hidradentitis suppurativa |
| what is the treatment for acute and chronic hidradentitis suppurativa | acute- antibiotics 2 weeks with intralesional steroids Chronic- long term antibiotics, high dose systemics steroids |
| What type of surgery or treatment is suggested for each stage of hidradentitis suppurativa | Stage 1- I&D w/ antibiotics stage 2- exterirization and laying open of tracts and electrocoagulation, or exicision and primary closure such as pollock procedure stage 3- total wide excision and healing with secondary intention or flaps and grafts |
| infection fo the epidermis that extends down to the dermis often from neglected impetigo | ecthyma |
| what pathogens cause ecthyma and what is suggested treatment | ecthyma can be caused by group A strep and S. aureus or a combo. Treatment is with oral antibiotics |
| Superficial infection of upper dermis and lymphatics w/ sharply demarcated raised boarders | erysipelas |
| what pathogens can cause erysipelas and what is the treatment | Group A strep and sometime group C or G. treatment anti strep antibiotics |
| how can you distinguish between erysipelas and cellulitis | erysipelas is more erythematous and has a raised boarder |
| Infectious disease of the dermis and sub q tissues without raised boarders | cellulitis |
| What pathogens can cause cellulitis | S. Aureus and Group A strep |
| What is the treatment of cellulitis | local skin care, elevation, and systemic antibiotics |
| common cause of facial cellulitis in kids | H. flu |
| causes cellulitis w/trauma and abcess | S. Aureus |
| cellulitis from a dog or cat bite | pasteurella |
| cellulitis from salt water | vibrio vulnificus |
| cellulitis in fresh water or soil | aromonas hydrophilia |
| skin disease when neisseria meningitides caused bacteremia spreads or manifests at the skin | meningococcemia |
| what are the s/sx of meningococcemia | transient macular eruption, purpura, and purulent petechiae |
| what is the treatment for meningococcemia | get the IV ceftriaxone fast because it is life threatening |
| aggressive cellulitis in superficial fascia and sub q tissues generally external lesions are small compared to the large lesions under the skin very life threatening | Necrotizing fasciitis |
| What are the usual pathogens that cause necrotizing fasciitis | strep pyogenes, enterobacter, bacteroides, vibrio |
| What is the treatment for necrotizing fasciitis | wide local debridment with broad spectrum antibiotics. Look for sub q gas in mri/ct to see how far out to debride |
Created by:
smaxsmith
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