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Nursing Funds Mod B Oxygen

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Term
Definition
OXYGEN   BASIC HUMAN NEED  
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HEART & LUNGS   SUPPLY THE BODY W/ OXYGEN NECESSARY FOR CARRYING OUT THE RESPIRATORY & METABOLIC PROCESSES NEEDED TO SUSTAIN LIFE  
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CARDIOPULMONARY SYSTEM   TO PROVIDE OXYGEN TO THE TISSUES AND REMOVE CARBON DIOXIDE & WASTE PRODUCTS FROM THE BODY  
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VENTILATION   THE MOVEMENT OF AIR IN & OUT OF THE LUNGS  
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DIFFUSION   MOVEMENT OF GASES BETWEEN AIR SPACES & THE BLOODSTREAM AT THE ALVEOLI  
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RESPIRATION   THE EXCHANGE OF O2 & CO2 DURING CELLULAR METABOLISM  
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PERFUSION   MOVEMENT OF BLOOD INTO & OUT OF THE LUNGS TO THE ORGANS & TISSUES OF THE BODY  
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RIGHT LUNG   3 LOBES UPPER, MIDDLE, LOWER  
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LEFT LUNG   2 LOBES UPPER, LOWER  
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ALVEOLI   GAS EXCHANGE IN THE LUNGS  
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NEURAL REGULATORS (CNS)   SENDS SIGNALS TO THE CHEST WALL MUSCULATURE TO CONTROL VENTILATION RATE, DEPTH, & RHYTHM  
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CHEMICAL REGULATION   INVOLVES THE INFLUENCE OF CHEMICALS SUCH AS CARBON DIOXIDE & HYDROGEN IONS, WHICH AFFECT THE RATE & DEPTH OF VENTILATION  
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OXYGENATION   AMOUNT OF OXYGEN ENTERING THE LUNGS FROM THE ATMOSPHERE; FUNCTION OF THE CARDIOPULMONARY SYSTEM  
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HEMOGLOBIN   TRANSPORTS MOST OXYGEN & SERVES AS A CARRIER FOR BOTH OXYGEN & CARBON DIOXIDE  
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OXYHEMOGLOBIN   HEMOGLOBIN MOLECULE COMBINED WITH OXYGEN  
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OXYGEN TRANSPORT   -O2 ENTERING LUNGS -BLOOD FLOW TO THE LUNGS -O2 CARRING CAPACITY OF BLOOD WHICH IS INFLUENCED BY: - AMOUNT OF O2  
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HYPOXIA   INADEQUETE TISSUE OXYGENATION WITH A DEFICIENCY IN OXYGEN DELIVERY OR OXYGEN UTILIZATION AT THE CELLULAR LEVEL; LIFE THREATENING  
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HYPOVOLEMIA   DEHYDRATION/SHOCK  
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SIGNS & SYMPTOMS OF HYPOXIA   -TACHYCARDIA -PERIPHERAL VASOCONTRICTION -DIZZINESS -MENTAL CONFUSION  
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TREATMENT OF HYPOXIA   -CARDIAC & RESPIRATORY STIMULANT DRUGS -OXYGEN THERAPY -MECHANICAL VENTILATION -FREQUENT ANALYSIS OF BLOOD GASES  
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HYPOXEMIA   AN ABNORMAL DEFICIENCY IN THE CONCENTRATION OF OXYGEN IN ARTERIAL BLOOD; A LOW PARTIAL PRESSURE OF OXYGEN (PaO2)  
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VALVES OF THE HEART   TRICUSPID - PULMONARY - MITRAL - AORTIC TOILET - PAPER - MY - ASS  
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HYPOVENTILATION   WHEN VENTILATION IS INADEQUATE TO MEET THE OXYGEN DEMANDS OF THE BODY TO ELIMINATE CARBON DIOXIDE  
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AS VENTILATION DECREASES   PaCO2 INCREASES  
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HYPERCAPNIA   HIGH CARBON DIOXIDE LEVELS  
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HYPERVENTILATION   AN INCREASE IN RESPIRATORY RATE, RESULTING IN EXCESS AMOUNTS OF CARBON DIOXIDE ELIMINATION  
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TETANY   CARPOPEDAL SPASM (MUSCLE SPASM OF HANDS/FEET)  
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SYSTOLE   CONTRACTION PHASE; BLOOD IS EXPELLED FROM THE VENTRICLES  
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DIASTOLE   RELAXATION PHASE; BLOOD FILLS THE VENTRICLES  
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MYOCARDIUM   HEART MUSCLE  
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MOTTLING   REDDISH/PURPLISH BLOTCHY SKIN; DECREASED PERFUSION  
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AFTERLOAD   RESISTANCE TO THE EJECTION OF BLOOD FROM THE LEFT VENTRICLE  
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LEFT VENTRICLE/AORTIC PRESSURE FACT   LEFT VENTRICULAR PRESSURE MUSCLE BE GREATER THAN THE AORTIC PRESSURE TO EJECT BLOOD FROM THE HEART  
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PRELOAD   AMOUNT OF BLOOD AT THE END OF VENTRICULAR DIASTOLE (END-DIASTOLIC PRESSURE)  
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CONTRACTION & RELAXATION   1 CARDIAC CYCLE  
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CARDIAC OUTPUT (CO)   AMOUNT OF BLOOD EJECTED FROM THE LEFT VENTRICLE EACH MINUTES (adults = 4-6 L/min)  
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STROKE VOLUME (SV) x HEART RATE (HR) =   CARDIAC OUTPUT  
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STROKE VOLUME (SV)   AMOUNT OF BLOOD EJECTED FROM THE VENTRICLE W/ EACH CONTRACTION (adult = 50-75 mL/contraction)  
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HEART RATE (HR)   BEATS PER MINUTE; REGULATED BY THE SYMPATHETIC & PARASYMPATHETIC SYSTEMS (60-100 beats/min)  
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MYOCARDIAL CONTRACTILITY   ABILITY OF THE HEART TO SQUEEZE BLOOD FROM THE VENTRICLES & PREPARE FOR THE NEXT CONTRACTION  
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CARDIAC INDEX   A MEASURE OF ADEQUACY OF THE CARDIAC OUTPUT  
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CONDUCTION SYSTEM   GENERATES IMPULSES THAT INITIATE THE ELECTRICAL MECHANICAL CHAIN OF EVENTS FOR A NORMAL HEARTBEAT  
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SINOATRIAL (SA) NODE   -"PACEMAKER" OF THE HEART -IN RIGHT ATRIUM NEXT TO SUPERIOR VENA CAVA -IMPULSES BEGIN AT RATE OF 60-100 beats/min  
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ATRIOVENTRICULAR (AV) NODE   -MEDIATES IMPULSE TRANSMISSION BETWEEN THE ATRIA & VENTRICLES -TRANSMITS THROUGH BUNDLE OF HIS & PURKINJE NETWORK FOR ATRIAL EMPTYING  
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ELECTROCARDIOGRAM (ECG)   RECORDS ELECTRICAL ACTIVITY OF THE CONDUCTION SYSTEM AS WAVES & COMPLEXES  
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NORMAL SINUS RHYTHM (NSR)   NORMAL SEQUENCE OF ELECTRICAL IMPULSES ON AN ECG  
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NORMAL ECG WAVEFORM CONSISTS OF:   -P WAVE (atrial depolarization) -QRS COMPLEX (ventricular depolarization) -T WAVE (ventricular repolarization)  
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HEART FAILURE   FAILURE OF THE MYOCARDIUM TO EJECT SUFFICIENT BLOOD VOLUME TO THE SYSTEMIC & PULMONARY CIRCULATIONS  
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MYOCARDIAL ISCHEMIA   WHEN CORONARY ARTERY DOES NOT SUPPLY SUFFICIENT BLOOD TO THE HEART MUSCLE  
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DECREASED PERFUSION =   CHEST PAIN  
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ANGINA PECTORIS   DECREASED BLOOD FLOW TO THE MYOCARDIUM DUE TO SPASMS OR TEMPORARY CONSTRICTION  
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MYOCARDIAL INFARCTION   NECROSIS OF A PORTION OF CARDIAC MUSCLE CAUSED BY OBSTRUCTION IN A CORONARY ARTERY  
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VALVULAR HEART DISEASE   AN ACQUIRED/CONGENITAL DISORDER OF A CARDIAC VALVE CHARACTERIZED BY STENOSIS = OBSTRUCTED BLOOD FLOW = BACKFLOW OF BLOOD RESULTS  
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LEFT-SIDED HEART FAILURE   -IMPAIRED FUNCTIONING OF THE LEFT VENTRICLE -INCREASED PRELOAD OR AFTERLOAD -DECREASED BLOOD EJECTED FROM THE LEFT VENTRICLE -DECREASED CARDIAC OUTPUT -CRACKLES ON AUSCULTATION  
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ORTHOPNEA   DIFFICULTY BREATHING WHILE LYING DOWN  
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RIGHT-SIDED HEART FAILURE   -IMPAIRED FUNCTION OF THE RIGHT VENTRICLE CAUSED BY PULMONARY DISEASE/HYPERTENSION -INCREASE IN PULMONARY SYSTEM -INCREASED RESISTANCE IN THE RIGHT VENTRICLE -RIGHT VENTRICLE FAILS AS A RESULT OF DISTENDED JUGULAR VEINS -PERIPHERAL EDEMA  
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DYSRHYTHMIA   DISTURBANCE IN THE ELECTRICAL IMPULSE OF THE HEART RHYTHM  
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TACHYCARDIA   >100 BEATS/MIN  
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BRADYCARDIA   <60 BEATS/MIN  
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ATRIAL FIBRILLATION (A-FIB)   IRREGULAR ATRIAL ACTIVITY RESULTING IN AN IRREGULAR VENTRICAL RESPONSE W/ RESULTANT IRREGULAR CARDIAC RATE/RHYTHM; NO IDENTIFYABLE P-WAVE  
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VENTRICULAR TACHYCARDIA   RHYTHM SLIGHTLY IRREGULAR; RATE 100-200 BEATS/MIN; P WAVE ABSENT; QRS COMPLEX WIDE >0.12 SECONDS  
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VENTRICULAR FIBRILLATION   -IRREGULAR & CHAOTIC RHYTHM W/ NO DISCERNIBLE WAVES/RATE  
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ASYSTOLE   ABSENCE OF ELECTRICAL ACTIVITY; NO DISCERNIBLE RATE/RHYTHM; PULSELESS/APNEIC  
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ANEMIA   A DEFICIENCY OF RED BLOOD CELLS OR OF HEMOGLOBIN IN THE BLOOD, RESULTING IN PALLOR/WEARINESS  
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97% OF OXYGEN IS CARRIED ON THE HEMOGLOBIN MOLECULE   TRUE STORY :)  
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CARBON MONOXIDE   MOST COMMON TOXIC INHALANT THAT DECREASES OXYGEN-CARRYING CAPACITY IN BLOOD CREATING HYPOXIA  
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DECREASED CONCENTRATED OF INSPIRED OXYGEN =   DECREASED OXYGEN CARRYING CAPACITY OF BLOOD  
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INSPIRED OXYGEN CONCENTRATION   FiO2  
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INCREASED METABOLIC   INCREASED OXYGEN DEMAND  
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FEVER INCREASED NEED OF THE TISSUES FOR OXYGEN =   INCREASED CARBON DIOXIDE PRODUCTION  
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DIAPHRAGMATIC MOVEMENT   ABILITY TO EXPAND & CONTRACT THE CHEST  
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ATELECTASIS   COLLAPSE OF ALVEOLI, PREVENTING NORMAL RESPIRATORY EXCHANGE OF OXYGEN & CARBON MONOXIDE  
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DISEASE/TRAUMA INVOLVING THE MEDULLA OBLONGATA & SPINAL CORD OF THE CNS W/OR HAS THE ABILITY TO IMPAIR RESPIRATION   MMM HMMMM YES IT DOES....PLEASE BE CAREFUL!  
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FLAIL CHEST   FRACTURES CAUSE INSTABILITY IN PART OF THE CHEST WALL; LIFE-THREATENING  
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PARADOXICAL BREATHING   LUNG UNDERLYING THE INJURED AREA CONTRACTS ON INSPIRATION & EXPANDS ON EXPIRATION  
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SURFACTANT   A CHEMICAL IN THE LUNG THAT MAINTAINS THE INTEGRITY OF THE ALVEOLI, KEEPING THE ALVEOLI DRY & PREVENTING ALVEOLAR COLLAPSE; DEVELOPS @ 7 MONTHS GESTATION  
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AHA   AMERICAN HEART ASSOCIATION  
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ALA   AMERICAN LUNG ASSOCIATION  
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ATS   AMERICAN THORACIC SOCIETY  
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ACS   AMERICAN CANCER SOCIETY  
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AHRQ   AGENCY FOR HEALTHCARE RESEARCH & QUALITY  
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FATIGUE   SUBJECTIVE SENSATION REPORTED AS A LOSS OF ENDURANCE  
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PAROXYSMAL NOCTURNAL DYSPNEA (PND)   DYSPNEA THAT OCCURS DURING SLEEP  
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ORTHOPNEA   ABNORMAL CONDITION WHILE LYING DOWN HAS DIFFICULTY BREATHING  
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WHEEZING   HIGH-PITCHED MUSICAL SOUND CAUSED BY HIGH-VELOCITY MOVEMENT OF AIR THROUGH A NARROWED AIRWAY; ASTHMA, ACUTE BRONCHITIS, PNEUMONIA  
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COUGH   A SUDDEN, AUDIBLE EXPULSION OF AIR FROM THE LUNGS  
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HEMOPTYSIS   BLOODY SPUTUM  
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CASCADE COUGH   HOLD BREATHE FOR 2 SECONDS AND THEN COUGH  
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HUFF COUGH   SAYS HUFF WHILE COUGHING  
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QUAD COUGH   PT COUGH WHILE HOLDING ABDOMEN  
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PRODUCTIVE COUGH   RESULTS IN SPUTUM PRODUCTION THAT IS SWALLOWED OR EXPECTORATED  
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INFLUENZA   VIRAL INFECTION THAT CAN CAUSE SERIOUS COMPLICATIONS  
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OXYGEN THERAPY   -TO PREVENT/RELIEVE HYPOXIA -OXYGEN IS A DRUG -7 RIGHTS APPLY TO OXYGEN ADMINISTRATION  
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NASAL CANNULA   SIMPLE/COMFORTABLE (1-6 L/MIN)  
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TRANSTRACHAEAL CANNULA   IV SIZE CATHETER INTO TRACHEA (1/4-4 L/MIN)  
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OXYGEN MASKS   ADMIN O2; HUMIDITY/HEAT  
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SIMPLE FACE MASKS   SHORT-TERM O2 (5-8 L/MIN)  
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PARTIAL NON-REBREATHER   HIGH CONCENTRATION O2 (6-10 L/MIN)  
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VENTURI MASK   HIGH-FLOW O2 (4-12 L/MIN)  
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HUMIDIFICATION   -BUBBLING OXYGEN THROUGH WATER -MORE THAN 4 L/MIN SOURCE OF HAI  
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NEBULIZER   AEROSOL PRINCIPLE TO SUSPEND A MAXIMUM NUMBER OF WATER DROPS/PARTICLES OF THE DESIRED SIZE IN INSPIRED AIR;MEDICATED  
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ARTIFICIAL AIRWAY   PATIENT W/ DECREASED LEVEL OF CONSCIOUSNESS; AIRWAY OBSTRUCTION, MECHANICAL VENTILATION, OR REMOVE TRACHEOBRONCHIAL SECRETIONS  
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ORAL AIRWAY   PREVENTS OBSTRUCTION OF THE TRACHEA BY DISPLACEMENT OF THE TONGUE INTO THE OROPHARYNX; MEASURED JAW TO EAR LOBE  
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TRACHEAL AIRWAY   ENDOTRACHEAL, NASOTRACHEAL, TRACHEAL; DEEP TRACHEAL SUCTIONING  
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45 DEG. LYING ANGLE SEMI-FOWLER   -CARDIOPULMONARY DISEASES -HEALTH LUNG SIDE DOWN IF PNEUMOTHORAX OR ATELECTASIS -AFFECTED LUNG SIDE DOWN IF PULMONARY ABSCESS OR HEMORRHAGE  
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INCENTIVE SPIROMETRY   METHOD OF ENCOURAGING VOLUNTARY DEEP BREATHING BY PROVIDING VISUAL FEEDBACK TO PTS. ABOUT INSPIRATORY VOLUME (BLOW INTO TUBE TO RAISE THE COLORED BALLS AND HOLD AS LONG AS YOU CAN)  
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CHEST PHYSIOTHERAPY (CPT)   MOBILIZES PULMONARY SECRETIONS INCLUDES: POSTURAL DRAINAGE, CHEST PERCUSSION & VIBRATIONS FOLLOWED BY COUGHING/SUCTIONING; >30 mL OF SPUTUM/DAY  
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POSTURAL DRAINAGE   USE OF POSITIONING TECHNIQUES TO DRAIN SECRETIONS FROM  
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CHEST PERCUSSION   STRIKING THE CHEST WALL OVER THE AREA W/ A CUPPED HAND; ALTERNATE HAND MOTION; CHANGES CONSISTENCY/LOCATION OF SPUTUM  
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VIBRATION   FINE, SHAKING PRESSURE APPLIED TO THE CHEST WALL ONLY DURING EXHALATION; SHAKES MUCOUS LOOSE & INDUCES COUGH; PERFORM ON INFANTS & YOUNG CHILDREN  
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CHEST TUBE   CATHETER INSERTED THROUGH THE RIB CAGE INTO THE PHEURAL SPACE TO REMOVE AIR OR FLUIDS  
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PNEUMOTHORAX   COLLECTION OF AIR OR OTHER GAS IN THE PHEURAL SPACE; COLLAPSED LUNG  
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HEMOTHORAX   AN ACCUMULATION OF BLOOD IN THE PHEURAL CAVITY BETWEEN THE PARIETAL & VISCERAL PLEURAE; USUALLY FROM TRAUMA; PREVENTS FULL EXPANSION  
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NON-INVASIVE VENTILATION (NIV)   MAINTAINS POSITIVE AIRWAY PRESSURE & IMPROVES ALVEOLAR VENTILATION W/O ARTIFICIAL AIRWAY; CPAP/BIPAP  
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CARDIAC ARREST   SUDDEN CESSATION OF CARDIAC OUTPUT & CIRCULATION; HEART, BRAIN & TISSUE DAMAGE OCCURS WITHIN 4-6 MIN.; LACK OF PULSE/RESPIRATION  
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CARDIOPULMONARY RESUSCITATION (CPR)   BASIC EMERGENCY PROCEDURE OF ARTIFICIAL RESPIRATION & MANUAL EXTERNAL CARDIAC MASSAGE  
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C-A-B   CHEST COMPRESSION/AIRWAY/BREATHING  
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DEFIBRILLATION RECOMMENDATIONS   5 MINUTES OUT OF HOSPITAL SETTING 3 MINUTES IN HOSPITAL SETTING  
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CHEST COMPRESSIONS   100 COMPRESSIONS/MIN  
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1 OR 2 RESCUER ON AGE 8 AND OLDER   30 CHEST COMPRESSIONS THEN 2 BREATHS  
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2 RESCUER ON AGE 7 AND UNDER   15 COMPRESSIONS THEN 2 BREATHS  
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CARDIOPULMONARY REHABILITATION   ACTIVELY HELPING A PT. ACHIEVE & MAINTAIN AN OPTIMAL LEVEL OF HEALTH THROUGH CONTROLLD PHYSICAL EXERCISE, NUTRITION COUNSELING, RELAXATION & STRESS MANAGEMENT, PRESCRIBED MEDICATIONS, & OXYGEN ADMINISTRATION  
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PURSED-LIP BREATHING   DEEP INSPIRATION & PROLONGED EXPIRATION THROUGH PURSED-LIPS TO PREVENT ALVEOLAR COLLAPSE; SIT UP, DEEP BREATH IN & BLOW OUT THROUGH PURSED LIPS  
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DIAPHRAGMATIC BREATHING   PT. TO RELAX THE INTERCOSTAL & ACCESSORY RESPIRATORY MUSCLES WHILE TAKING DEEP INSPIRATIONS; 2 HANDS W/ 1 BELOW THE BREASTBONE & OTHER 2-3 CM. BELOW MOVING ONLY LOWER HAND ON INSPIRATION  
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THE DIFFERENCE BETWEEN HYPOXIA AND HYPOXEMIA IS:   HYPOXIA IS INADEQUATE OXYGEN AT THE CELLULAR LEVEL  
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A NURSE IS CARING FOR A PATIENT IN RESPIRATORY DISTRESS. WHICH OF THE FOLLOWING IS INCLUDED IN THE ASSESSMENT? (select all that apply)   - USE OF ACCESSORY MUSCLES OF RESPIRATION - RESPIRATORY RATE - PULSE OXIMETRY - ABILITY TO CONVERSE  
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THE NORMAL ELECTRICAL PATHWAY OF THE HEART IS:   SA NODE-ATRIA-AV NODE BUNDLE OF HIS-PURKINJE FIBERS-VENTRICLES  
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THE MOST EFFECTIVE BREATHING TECHNIQUES FOR A PATIENT WITH COPD INCLUDE:   PURSED LIP BREATHING  
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AFTERLOAD IS DEFINED AS:   THE RESISTANCE OF THE EJECTION OF BLOOD FROM THE LEFT VENTRICLES  
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WHICH OF THE FOLLOWING CHARACTERISTICS ARE ASSOCIATED WITH VENTILATOR-ASSOCIATED PNEUMONIA (VAP)?   FEVER AND PROGRESSIVE INFILTRATES ON THE CHEST RADIOGRAPH 3 DAYS AFTER INITIATION OF MECHANICAL VENTILATION  
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WHICH OF THE FOLLOWING FINDINGS ARE CONSISTENT WITH AN INCREASED RISK OF DEVELOPING VENTILATOR-ASSOCIATES PNEUMONIA (VAP)?   POOLING OF SECRETIONS IN THE OROPHARYNX  
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VENTILATOR-ASSOICATED PNEUMONIA (VAP) IS ASSOCIATED WITH COLONIZATION OF THE:   RESPIRATORY AND DIGESTIVE SYSTEMS  
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THE CURRENT COMPRESSION RATE FOR CARDIOPULMONARY RESUSCITATION (CPR) IS AT LEAST:   100 BEATS/MIN  
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PRELOAD IS:   THE AMOUNT OF BLOOD AT THE END OF VENTRICULAR DIASTOLE, OR MEASURED AS END-DIASTOLIC PRESSURE  
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NEURAL REGULATION   NEURAL REGULATION MAINTAINS RHYTHYM AND DEPTH OF RESPIRATION AND THE BALANCE BETWEEN INSPIRATION AND EXPIRATION  
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CEREBRAL CORTEX   VOLUNTARY CONTROL OF RESPIRATION DELIVERS IMPULSES TO THE RESPIRATORY MOTOR NEURONS BY WAY OF THE SPINAL CORD. ACCOMODATES: SPEAKING, EATING AND SWIMMING  
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MEDULLA OBLONGATA   AUTOMATIC CONTROL OF RESPIRATION OCCURS CONTINUOUSLY  
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KUSSMAUL'S   is a deep and labored breathing pattern often associated with severe metabolic acidosis, particularly diabetic ketoacidosis (DKA) but also kidney failure (>35 breaths/min)  
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CHEYNE-STOKES   ABNORMAL PATTERN OF BREATHING, VARYING BETWEEN APNEA AND TACHYPNEA  
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AED   DEVICE USED TO ADMINISTER AN ELECTRICAL SHOCK THROUGH THE CHEST WALL TO THE HEART  
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BREATHING EXERCISES IMPROVE:   VENTILATION OXYGENATION SENSATIONS OF DYSPNEA  
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OXYGEN THERAPY IMRPROVES:   LEVELS OF TISSUE OXYGENATION BY NASAL CANNULA, NASAL CATHETER, OR OXYGEN MASK  
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CHEST PHYSIOTHERAPY INCLUDES:   POSTURAL DRAINAGE PERCUSSION VIBRATION  
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DECREASED HEMOGLOBIN LEVELS ALTER A PATIENT'S ABILITY TO TRANSPORT OXYGEN   YES IT DOES.......WE NEED TO TAKE CARE OF OURSELVES!  
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CARDIAC DYSRHYTHMIAS ARE CLASSIFIED BY:   CARDIAC ACTIVITY AND SITE OF IMPULSE ORIGIN  
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