Random heme associations
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| show | Pb toxicity or myelodysplastic syndrome with 6 years to live
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| anemia in a person who's worked with batteries or a person who lived in an old building | show 🗑
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| show | in B thalassemia major, it's a precipitation of alpha chains
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| walking like a soldier | show 🗑
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| show | A FOLIC DROP. Alcohol, Folate antag like MTX and pyrimethamine, OCPs, Low in diet, Infection, Celiac, Dilantin/phenytoin, Relative folate deficiency, Old, Pregnant
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| see hypersegmented neutrophils on smear vs hypo segmented neutrophils | show 🗑
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| burr cells on peripheral smear | show 🗑
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| spur cells | show 🗑
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| mechanical valve on warfarin in the aortic position and without additional risk factors - what is the pre-procedural anticoagulation recommendation? | show 🗑
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| show | include afib, >1 mechanical valve, ejection fraction <30%, a hypercoagulable state, and previous thromboembolic event, including stroke or transient ischemic attack
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| what is the recommendation for pre-procedural anticoagulation for patients with a mechanical valve and an increased risk of a thromboembolic event | show 🗑
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| patients with a mechanical heart valve and therapeutic INR who require emergent surgery - what to do with the anticoagulation? | show 🗑
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| show | activated protein C resistance, factor V Leiden, the prothrombin gene mutation, antiphospholipid antibodies, a lupus inhibitor, antithrombin deficiency, protein C deficiency, and protein S deficiency
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| show | MGUS <10% plasma cells, <3g M protein
smoldering >10% plasma cells, >3g M protein with NO organ damage
multiple myeloma >10% plasma cells, >3g M protein WITH organ damage
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| MCV in Hereditary spherocytosis | show 🗑
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| When to do FISH to find diagnosis | show 🗑
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| show | Flow cytometry of the peripheral blood would be useful if a hematologic malignancy characterized by a homogeneous population of cells (acute lymphoblastic leukemia, chronic lymphocytic leukemia, non-Hodgkin lymphoma, or acute myeloid leukemia
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| show | multiple myeloma
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| acute thrombosis, warfarin therapy, and pregnancy can cause transient declines or increases in functional protein S levels | show 🗑
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| show | hypercoaguability. activated protein C and S block conversion of F8 and F5 to their active forms. Therefore, increased C+S means more bleeding. decreased C+S means less bleeding, more coagulability.
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| show | AL amyloidosis
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| how to dx AL amyloidosis | show 🗑
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| show | Hereditary spherocytosis
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| Pregnant women with a history of idiopathic venous thromboembolism should receive what for ppx? | show 🗑
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| show | Prophylaxis with both low-dose aspirin and low- or moderate-dose unfractionated heparin or LMWH
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| inheritance pattern for von Willebrand disease | show 🗑
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| show | Corticosteroids and intravenous immune globuli
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| thrombocytopenia, microangiopathic hemolytic anemia, neurologic deficits, kidney impairment, and fever | show 🗑
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| 25% lymphoblasts or more on bone marrow examination | show 🗑
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| show | marker for lymphoid cells
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| CD10 and CD20 | show 🗑
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| show | imatinib used in CML or Philadelphia chromosome-positive ALL
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| how to treat B-cell acute lymphoblastic leukemia | show 🗑
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| when to use Erythrocyte transfusion and exchange transfusion in sickle cells patients | show 🗑
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| show | delayed hemolytic transfusion reaction
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| show | essential thrombocythemia
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| show | essential thrombocythemia
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| presence of (1) microangiopathic hemolytic anemia, characterized by schistocytes on the peripheral smear and increased lactate dehydrogenase, and (2) thrombocytopenia. | show 🗑
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| thrombocytopenia, microangiopathic hemolytic anemia, neurologic deficits, kidney impairment, and fever | show 🗑
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| show | thrombotic thrombocytopenic purpura
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| show | plasma exchange, which should be instituted emergently at diagnosis because 10% of patients die of this disease despite therapy, usually within the first 24 hours
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| show | Mild congenital asymptomatic neutropenia, common in certain ethnic populations, including blacks, Yemenite Jews, and Jordanian Arabs. This condition is not associated with increased infections and requires no therapy.
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| show | evaluation of autoimmune neutropenia if the patient's neutropenia were to worsen
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| show | mutated in patients with severe congenital neutropenia, which is characterized by onset early in life and life-threatening infections
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| show | iron chelation with Deferasirox
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| show | agranulocytosis and kidney failure
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| unprovoked venous thrombosis at an unusual location, hemolytic anemia, and mild to moderate pancytopenia | show 🗑
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| show | diagnosis of PNH is made by flow cytometry, which can identify a subpopulation of erythrocytes or leukocytes lacking specific glycosylphosphatidylinositol-anchored surface proteins, such as CD55 or CD59
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| show | Waldenström macroglobulinemia
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| show | Waldenström macroglobulinemia
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| how to treat hyperviscoscity in Waldenström macroglobulinemia | show 🗑
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| what can be helpful in estimating the prognosis of thrombotic thrombocytopenic purpura (TTP) | show 🗑
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| show | presence of schistocytes on the blood smear makes warm or cold autoimmune hemolytic anemia unlikely
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| show | Epstein-Barr virus and cytomegalovirus infection can cause aplastic anemia, also ParvoB19
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| how to treat HIT | show 🗑
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| hypoxia, and dyspnea, as well as fever and hypotension, occurring during or within hours of a transfusion and resembles noncardiac pulmonary edema | show 🗑
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| show | acute hemolytic transfusion reaction (AHTR) is most commonly caused by a clerical error leading to ABO incompatibilit
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| high Hct and high Epo vs high Hct and Low Epo | show 🗑
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| macroovalocytes and hypersegmented polymorphonuclear cells on the peripheral blood smear and hemolysis. | show 🗑
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| glossitis, weight loss, and pale yellow skin, loss of position or vibratory sense that can progress to spastic ataxia | show 🗑
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| Hereditary spherocytosis associated with what types of stones | show 🗑
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christinapham
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