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Heart/CAD

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Question
Answer
arteriosclerosis   "hardening of the arteries", forming of plaque buildup, medium or large arteries  
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atherosclerosis   "hardening of the arteries", forming of plaque buildup, any artery  
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ischemia   lack of O2 to a tissue/organ, can be brought back with recirculation  
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LDL   takes cholesterol & deposits it into the artery walls, "bad cholesterol", less than 130mg/dl  
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HDL   takes cholesterol thats removed from artery walls & takes to liver to be excreted, "good cholesterol", greater thatn 35 mg/dl  
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total cholesterol   should be less than 200 mg/dl  
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triglycerides   should be 200-300 mg/dl  
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prevention strategies for CAD   nutrition, physical fitness, adequate sleep, modify risk factors, avoid substance abuse, anxiety, stress management & CISM  
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Modifiable risk factors   elevated blood lipids, obesity, smoking, sedentary lifestyle, diabetes mellitus, sypertension & elevated hormone levels(BC)  
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not modifiable risk factors   age, gender, heredity  
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systole   each contraction of the heart  
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diastole   each relaxation period after contraction of the heart  
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cardiac output   blood pumped in 1 min, = stroke vol x heart rate  
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stroke volume   blood pumped in 1 stroke/contraction  
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preload   passive stretching force exerted on the ventricular mesucle @ end of diastole  
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afterload   pressure the ventricular muscle must generate to overcome the increasing pressure of the aorta  
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contractility   force @ which muscles shorten/contract  
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starling's law   increased blood to the heart leads to increased cardiac contrility because the centricles stretch  
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ejection fraction (EF)   % of blood ejected during ventricular contraction  
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cardiac index   measure of cardiac output  
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pulse deficit   pulse is @ different rate than the ventricle is contracting (poor peripheral perfusion)  
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ASHD   arteriosclerotic heart disease  
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angioplasty   insert catheter, pump up balloon, put in stent (mesh tube)  
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digitalis   cardiac med, used for centuries, derived from foxglove plant, +inotropic(force), -chronotropic(rate), -dromotropic(speed), increased cardiac output, CHF, tachyarrhythmias  
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calcium channel blockers   cardiac med, -inotropic(force), -chronotropic(rate), decreases AV conduction, dilates coronary arteries & peripheral vascular resistance, inhibits vascular spasm, for angina, SVT, hypertension, peripheral vascular disease & vascular headaches(migraines)  
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beta adrenergic blockers   cardiac med, -chronotropic(rate), -inotropic(force), -dromotropic(speed), decreases myocardial oxygen consumption & peripheral vascular resistance, for angina, hypertension, dsysrhythmias & vascular headaches(migraines), usually end in "olol"  
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nitrates, nitrites   cardiac meds, vasodilator(arteries & veins), decreases preload & myocardial oxygen demand, oral, sublingual & transdermal  
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angina symptoms   pain lasts 3-5 mins, chest pressure, squeezing or heaviness, radiation patterns, anxiety, dyspnea, nausea &/or vomiting, diaphoresis  
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anaerobic metabolism   cell work with no O2, uses sugars & other things available, not good  
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prinzmetal's angina   varient type, caused by spasms in coronary muscles by arteries, don't know why, happens @ rest in cycles  
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unstable angina/preinfarction   everything is longer & worse, much harder to get to go away, occurs @ rest, new onset  
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stable angina   starts with activity & goes away when @ rest, predictable, often relieved w/ O2, nitro & rest, acute coronary syndrome  
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MONA   treatment for cardiac chest pain, M-morphine, O-oxygen, N-nitro, A-aspirin (not in that order)  
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causes of angina pectoris   CAD & spasms in the muscle of the arterial walls(rare)  
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causes of AMI(acute miocardial infarction)   ASHD, prolonged spasm, embolism, hypoxia, shock, L ventricle most common place to occur, once dead cannot be brought back  
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signs & symptoms of AMI   70%-90% chest pain, could be "silent MI" though, radiation pattern, constant, no relief, Hx unstable angina common, ECG changes, dyspnea, anxiety, nausea &/or vomiting, diaphoreis, cyanosis, palpitations, dysrhythmias  
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collateral circulation   effort of vessels around affected area to try to take over & oxygenate, only lasts short time  
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pleuritic chest pain   often varies w/ inspiration & expiration, chest movement & cough  
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occlusion of right coronary artery   supplies SA node, posterior & inferior walls, anterior R ventricle wall  
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occlusion of left circumflex coronary artery   lateral wall, sometimes inferior & posterior wall  
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occlusion of left coronary artery   massive anterolateral MI, anterior wall of L ventricle, lateral L ventricle wall, superior L ventricle  
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occlusion of left anterior descending coronary artery   anteroseptal MI, anterior wall  
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AMI complications   arrhythmias, sudden death, congestive heart failure, cardiogenic shock & rupture  
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thrombolytics (TBA)   drugs used to dissolve blood clots, has to be done in under 6 hrs from onset of symptoms & under the age of 75  
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CABG, coronary artery bypass graft   take artery from leg & re-route blood, only lasts about 5 yrs  
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pleural effusion   abnormal collection of fluid in the pleural cavity  
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-blood supply to AV node variable, 60% of people it rises from R side   -low O2 venous blood from heart drains into 5 different coronary veins & empties into R atrium via coronary sinus  
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