CPG Pathophysiology
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show | LVF or myocardial dysfunction = ↓ in SV & ejection fraction Leads to ↑ in LVEDP & L atrial pressure ∴ pulm capillary pressure ↑ due to hydrostatic pressure overcoming oncotic pressure=fluid forced into alveolus Impaired Pulmonary compliance Surfactant |
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Anaphylaxis | show | Sebsequent exposure>Reaction with Mast cells>degranulation>mediator release
Airways = oedema and plugging
Skin and CV = vasodilation and ↑ in capillary permeability
GIT = N/V/cramping
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Allergic Reaction | Similar to Anaphylaxis, but NO systemic involvement. Hypersensitivity reaction to a previously encountered antigen, resulting in a non-systemic, non-life threatening localised reaction | show 🗑
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Abdominal Aortic Aneurysm | show |
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Autonomic Dysreflexia | show |
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Pulmonary Embolus | Occlusion of the Pulmonary vasculature by: thrombus, tissue fragment, fat or air. Impact depends on the extent of pulmonary blood flow obstructed, | show 🗑
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COPD | show |
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Croup | Laryngotracheobronchitis, causing acute inflammation and oedema at the narrow glottic and subglottic region of the upper airway. Leading to airway obstruction, associated respiratory distress and hypoxia. | show 🗑
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show | Rapid progressive, life-threatening systemic inflammatory response to a bacterial infection as it enters the blood stream. Marked by fly-like symptoms, red petechial rash, resulting in multiple organ failure and CV collapse |
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Asthma | A reversible hyperinflammatory disease of the small airways mediated by multiple trigger factors cause mast cell degranulation and release of inflammatory mediators. This is marked by mucosal oedema, bronchospasm and mucosal plugging. | show 🗑
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