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neurologic system

ch 25

QuestionAnswer
risk factors for CVA smoking, diabetic, coronary artery disease, transient ischemic attack, atrial fibrillation, high serum cholesterol, obesity, alcohol, cocaine, age, gender, family history, race, history of CVA, hypertension
gender risk for CVA men at greater risk than women, pregnant women have a greater risk than non preg, bc pills increase risk
race risk for CVA blacks more at risk bc of uncontrolled hypertension
diabetic risk for CVA plaque in A, hight cholesterol, vasoconstriction
coronary artery disease risk for CVA plaque in A, high cholesterol, vasoconstriction
transient ischemic attack risk for CVA temporary loss of consciousness, warning sign of CVA
atrial fibrillation risk for CVA microclots can form adn can travel to brain
high serum cholesterol risk for CVA plaque
obesity risk for CVA high cholesterol
alcohol risk for CVA chronic, hypertension
cocaine risk for CVA vasoconstriction
tonic stage of seizure exxtremities held rigidly
clonic stage of seizure arms move btwn flexion and extension rhythmically
postictal stage of seizure pt difficult to arouse, extremely drowzy
absent seizures no recollection, pt doesnt know its happening
test CN II peripheral vision, snellen chart, confrontation test
test CN I properly identify aromatic substances
test CN III, IV, VI observe eyes for extra ocular movt
PERRLA observe eyes for pupillary size, shape, equality, constriction, accomodation
constriction pupils get smaller when light shined in eye
accomodation when both eyes move together
test balance observe walking, romberg test, eyes closed stand on one foot, tandem walking, hop on one foot then other, knee bends, walk on toes/heels
test coordination rapid pronation/supination on thighs, alternately touch nose with index fingers (eyes closed), touch finger to thumb rapidly, move index finger btwn nose and examiner finger, heel to shin of opp leg
romberg test pt standing, feet together, arms at side, eyes open/closed
graphesthesia identification of number/letter drawn on hand, back, other area
streognosis identification of familiar object in hand
DTR (deep tendon reflexes) muscle contraction response to direct/indirect percussion of tendon
procedure for DTRs pt relaxed, lying or sitting down, patellar, plantar and ankle clonus reflexes, 4+ scoring system
patellar reflex contaction of quadriceps muscles, extension of lower leg
plantar reflex plantar flexion of toes
ankle clonus if hyperactive reflexes, sharply dorsiflex foot, maintain no movt
change in level of consciousness is most sensitive indicator of alterations in cerebral function
can determine if pt is alert/oriented by way that questions are answered during the history
awareness higher level function, controlled by reticular activating system
wakefulness controlled by brainstem
recall tell series of numbers then 5 min later ask them again
awareness is assesed by orientation, memory, attention, calculation, recall, language, judgement, insight, abstraction
first orientation to disappear time/date
2nd orientation to disappear place
last orientation to disapper person
orientation returns in which order opp in which lost
arousal modified assesment for unconsious, assume they can hear, pupillary rxn (hold eye open and shine light), smell breath (ketones, alcohol)
CN III pupillary response, originates in brainstem
small reactive pupils bilateral cerebral dysfunction
bilaterally dilated pupils overdose of hallucinogens or CNS stimulants, pressure in brainstem compressing CN III bilaterally
glascow coma scale assess for best response to eye opening, motor response and verbal response
test for meningeal irritation used when meningitis is suspected, kernigs sign
kernigs sign flexing one leg at hip and knee then extending knee, no pain means negative, pain along vertebral column when leg is extended is a positive sign of inflamm of meninges
brudzinskis sign tests for meningeal irritation
procedure for brudzinskis sign test client supine, neck flexed
+ brudzinskis sign client passively flexes knee in response to head flexion, reports pain along verterbral column
meningitis inflamm of meninges that surround brain spinal chord, invasion of bacteria, viruses, fungi, parasites, or other toxins
most common meningitis bacterial
viral meningitis self limiting infection with full recovery
symptoms of meningitis severe headache, fever, generalized malaise
signs that show meningitis stiff neck, + kernigs sign, + brudzinskis sign
result of menigitis level of consciousness may decrease, may progress to stupor or coma, confusion, agitiation, irritability can occur
2nd most common neurodegenerative disease after alzheimers parkinsons
clinical findings of parkinsons resting tremor, bradykinesia (slow movt), rigidity, masklike fcies, trunk forward flexion, muscle weakness, shuffling gait, finger pill rolling tremor, cogwell rigidity (when pull arm up it shakes down)
parkinsons develops slowly, brains dopamine producing neurons in substantia nigra of basal ganglia degenerate
how a CVA happens cerebral blood vessels occluded by thrombus/embolus or intracranial hemorrhage occurs, brain tissues ischemic
occurence of CVA by what 80% by blood vessels, 20% by hemorrhage
hemorrhage hypertension or cerebral aneurysm (weakened area of artery that ballons from high pressure)
symptoms of CVA directly related to areas of brain involved/extent of ischema , sudden unilateral numbness/weakness of face, arm, leg, trouble walking, dizziness, loss of balance, sudden severe headaches, aphasia, sudden confusion, dysphagia, part loss of vision
guillian barre syndrome widespread demyelinization of nerves of PNS
Gullian barre syndrome believed to be caused by cell mediated immune response to viral infection, respiratory/GI viral infection weeks before onset
recovery of Gullian barre 80-90% have few or no residual deficits, could die if respiratory depression develops rapidly
clinical findings of gullian barre ascending paralysis, descending variation, deep tendon reflex absent, paralysis of thorax (respiratory depression)
ascending paralysis begins with weakness/paresthesia in lower extremities, ascends to upper extremities/face
descending variation facial, glossopharyngeal, vagus, hypoglossal CNs. downward to hand, cant reach feet
in romberg test if client sways with eyes closed but not open problem is proprioceptive
in romberg if client sways with eyes open and closed prob cerebellar disorder
pupils should appear equal, round and reactive to light and accomodation (documented as PERRLA)
what can cause diminished to absent pupillary constriction, ptosis of eye, altered superior and inferior movt of eyeball increased intracranial pressure or trauma to midbrain, causes pressure on CNIII
glascow coma scale is used to asses level of consciousness using a 15pt scale
glascow coma scale asseses best response to eye opening, motor response, verbal response
Created by: ashley0683
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