Busy. Please wait.

show password
Forgot Password?

Don't have an account?  Sign up 

Username is available taken
show password


Make sure to remember your password. If you forget it there is no way for StudyStack to send you a reset link. You would need to create a new account.
We do not share your email address with others. It is only used to allow you to reset your password. For details read our Privacy Policy and Terms of Service.

Already a StudyStack user? Log In

Reset Password
Enter the associated with your account, and we'll email you a link to reset your password.

Remove ads
Don't know
remaining cards
To flip the current card, click it or press the Spacebar key.  To move the current card to one of the three colored boxes, click on the box.  You may also press the UP ARROW key to move the card to the "Know" box, the DOWN ARROW key to move the card to the "Don't know" box, or the RIGHT ARROW key to move the card to the Remaining box.  You may also click on the card displayed in any of the three boxes to bring that card back to the center.

Pass complete!

"Know" box contains:
Time elapsed:
restart all cards

Embed Code - If you would like this activity on your web page, copy the script below and paste it into your web page.

  Normal Size     Small Size show me how

disease, inflam.

inflammatory disease processes

clinical presentation of GERD dyspepsia, hypersalivation, eructation, flatulence, dysphagia,odynophagia, chronic cough, asthma, atypical chest pain,bloat ,N, V
Why does GERD happen? incompetent lower esophageal sphincter
risk for GERD obesity, genetics, NG tube, meds, certain foods, pregnancy, large meals, acidic food, bending over, eating late
meds for GERD antacids, histamine receptor antagonists, PPIs, prokinetic drugs
peptic ulcer disease (PUD) etiology? acid, pepsin, H. pylori all play a role in causing PUD. Also chronic high dose of NSAIDs has been implicated. stress is a factor as well.
clinical signs of PUD? burning, gnawing, sharp epigastric pain.
complications of PUD? peritonitis if ulcer eats thru stomach wall into abdomen.
complications of ulcers: hemorrhage/perforation/pyloric obstruction/intractable disease
H. pylori treatment: two choices are Metronidazole and tetracycline. or clarithromycin and amoxicillin. the 2 things are not interchangeable.
PPIs: end in "zole". they reduce gastric acid secretion.
H2receptor antagonists: block histamine stimulated gastric secretion. can be used for indigestion and heartburn. block action of H2 receptor of the parietal cells thus inhibiting HCl secretion. end in "dine".
prostaglandin analogues: reduce gastric secretion and enhance gastric mucosal resistance to injury. can be used to protect stomach lining. ex. Cytotec helps stop NSAID induced ulcers. do not use in pregnant patn.
Created by: wilsoj7