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disease, inflam.

inflammatory disease processes

QuestionAnswer
clinical presentation of GERD dyspepsia, hypersalivation, eructation, flatulence, dysphagia,odynophagia, chronic cough, asthma, atypical chest pain,bloat ,N, V
Why does GERD happen? incompetent lower esophageal sphincter
risk for GERD obesity, genetics, NG tube, meds, certain foods, pregnancy, large meals, acidic food, bending over, eating late
meds for GERD antacids, histamine receptor antagonists, PPIs, prokinetic drugs
peptic ulcer disease (PUD) etiology? acid, pepsin, H. pylori all play a role in causing PUD. Also chronic high dose of NSAIDs has been implicated. stress is a factor as well.
clinical signs of PUD? burning, gnawing, sharp epigastric pain.
complications of PUD? peritonitis if ulcer eats thru stomach wall into abdomen.
complications of ulcers: hemorrhage/perforation/pyloric obstruction/intractable disease
H. pylori treatment: two choices are Metronidazole and tetracycline. or clarithromycin and amoxicillin. the 2 things are not interchangeable.
PPIs: end in "zole". they reduce gastric acid secretion.
H2receptor antagonists: block histamine stimulated gastric secretion. can be used for indigestion and heartburn. block action of H2 receptor of the parietal cells thus inhibiting HCl secretion. end in "dine".
prostaglandin analogues: reduce gastric secretion and enhance gastric mucosal resistance to injury. can be used to protect stomach lining. ex. Cytotec helps stop NSAID induced ulcers. do not use in pregnant patn.
Created by: wilsoj7
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